Anderson Pulmonary Flashcards

1
Q

lobes of lungs

A

3 on right
2 on left
started out with 3 on both sides but 2 on left due to heart

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2
Q

remnant of 3rd left lobe

A

lingular lobe

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3
Q

location of apex of lungs at max inhalation

base

A

4cm above 1st rib

base to bottom of rib cage

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4
Q

hilar surface

A

area where vessels go in and out

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5
Q

lung pleura

A

visceral: next to lung
parietal: adheres to throacic cage
fluid in between pleura

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6
Q

pneumothorax

A

loss of vacuum between pleura

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7
Q

blood from aorta to lung via ____

blood from lung tissue to azygos and pulmonary veins ___

A

bronchial artery

bronchial vein

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8
Q

only artery in body that caries deoxygenated blood

A

pulmonary artery

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9
Q

what nerve constricts bronchioles, and is sensory to lungs

A

parasympathetic CNX

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10
Q

area defined by the heart

A

mediastinum (heart and pericardium) is middle mediastinum

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11
Q

area behind heart with esophagus, descending aorta, azygos veins, thoracic duct, sympathetic trunk

A

posterior mediastinum

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12
Q

what is in the anterior medastinum?

A

thymus gland

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13
Q

surface landmarks of resp system

  • at mcl, where is lung?
  • at axillary line
  • costal angle
A

pleura = inhalation

  • 6th rib, pleura to 8th
  • 8th rib, pleura to 10th
  • 10th rib, pleura to 12th
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14
Q

what is order of draining in sinuses?

A

sphenoid ethmoid
ethmoid and frontal to maxillary
maxillary to nasal cavity

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15
Q

tidal volume

A

inspiration and expiration that is just enough so you don’t pass out. Normal breathing

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16
Q

what is vital capacity

A

inspiratory capacity, tidal volume, expiratory capacity

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17
Q

inspiratory capacity

A

sum of tidal volume and IRV

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18
Q

FEV1 is what % of forced vital capacity?

A

80% and based on first second

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19
Q

Ventral Resp Group (VRG)

A

active respiration in Medulla - expiration

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20
Q

What stimulates inspiration?

what inhibits inspiration?

A

apneustic center in lower pons

pneumotaxic center in upper pons

21
Q

during metabolic acidosis (decreased HCO3)

A

respond by decreasing CO2

- always respond with other chemical

22
Q

how would body respond to respiratory acidosis (increased CO2)

A

by increasing HCO3

23
Q

how would body respond to respiratory alkalosis (decreased CO2)?

A

by decreasing HCO3

24
Q

at a PO2 of 100mm Hg what is hemoglobin saturation?

at 40 mm Hg

at 25 mm Hg

A

100% saturated
O2 bound to all four heme groups on all hemoglobin molecules

75% saturation (3 of 4 heme groups on each hemoglobin molecule have O2 bound)

50% saturation (2 of 4)

25
where does % saturation max out with carbon monoxide poisoning
50% (need more than that to stay alive) | - either die or have headaches
26
what does HCO3 leave RBCs in exchange for?
Cl- (chloride shift)
27
what inhibits smooth muscle in lungs?
sympathetic beta 2 autonomic nerve fibers
28
epiglottitis
epiglotis swells and then airways swell - after H influenza or beta hemolytic strep - steeple sign - head bent over and drooling (only way air can still move through) - stridor
29
what causes pulmonary edema?
left sided heart failure | - fluid overload/renal failure, decreased albumin, lymph obstruction
30
where do pulmonary emboli originate from?
originate in deep veins of legs and pelvis | sudden onset cyanosis and dyspnea
31
pulmonary HTN due to
left sided heart failure, mitral stenosis, increased pulmonary vascular resistance, emboli, scleroderma
32
Atelectasis
collapse or incomplete expansion of acini | tumors, FB, mucus blockage, surfactant deficiency
33
causes of pulmonary edema
left heart disease | pneumonia, toxic gas inhalation
34
decreased compliance due to
fibrosis lack of surfactant high pulmonary venous pressure
35
increased compliance due to
emphysema
36
obstructive lung disease
inability to move air out of lungs
37
interstitial/restrictive lung disease
scarring and tightening of lungs so they are unable to expand
38
chronic obstructive disease: emphysema
``` "pink puffers" normal pCO2 because compensating high RBCs/high hematocrit permanent dilation barrel chest ```
39
chronic bronchitis
``` blue bloaters increased pCO2 can cause cor pulmonale can become cyanotic persistant cough with sputum ```
40
lobar pneumonia
strep pneumonia gm + rusty brown sputum starts at bottom and works its way up (klebsiella gm - in alcoholics = red hepatization)
41
broncho pnemonia/lobular
patchy opportunistic infection
42
atypical pneumonia
see diffuse patchy feathery infiltrates | walking pneumonia
43
TB
delayed hypersensitivity type IV caseating granulomas apices of lungs/high O2
44
asthma
IgE type I hypersensitivity | cardiac asthma from left sided heart failure
45
pneumoconiosis
inhalation of dust upper lobes silicosis, asbestosis type I hypersensitivity IgE
46
Goodpasture's syndrome
antibodies against basement membrane of lung and kidneys | type II immune injury
47
hypersensitivity pneumonitis
farmers lung pigeon breeders lung non-caseating
48
pneumothorax
air/gas in pleural cavity
49
small/oat cell undifferentiated carcinoma
pancoast tumors, rapid death | ability to produce hormones