Acute coronary symptoms Flashcards
complete obstruction MI =
STEMI
major differing factor along acute coronary syndrome continuum
ST elevation (STEMI) vs not (NSTEMI, UA)
presentation unstable angina
acceleration of ischemic symptoms (frequency, intensity, duration)
angina at rest
no relief by usual dose of Nitro
sypathetic effect of MI
parasymph
symph: cold clammy skin diaphoresis
parasympth: nausea, vomting,
cardiac findings of MI
S4 (+S3 is systolic dysfxn) gallop
dyskinetic bulge (ant wall MI)
systolic murmur (if mitral regurg. or VSD)
ECG abnormalities UA/NSTEMI
ST depression and or T wave inversion
tranient with chest pain(UA)
persist (NSTEMI)
STEMI ECG progression
ST elevation
Hours: ST elevation, reduced R, Qwave
Day1-2 Twave inversion
Days: Normalizes ST, T inversion
Weeks: Q wave persists
(S STarteRs Take Normal Questions
serum markers of infarction in NSTEMI and STEMI
cardiac troponins
Creatine Kinase (CK-MB)
beginning, peak for Serum MI markers
Cardiac troponins
CK-MB
cTn rise 3-4h, peak 18-36hr
**CK-MB **rise 3-8 hours, peak 24hr
serum biomarkers for UA
none
treatment difference STEMI vs non
STEMI have total occlusion and benefit from immediate reperfusion therapies
treatment high/low risk STEMI
low - fibrinolytic therapy (If PCI not available)
high - Primary PCI
treatment NSTEMI low/high risk
Low - conservative, cath lab only if reccurent angina or stress test positive
high - cath lab > PCI or CABG
most important factor in post MI recovery+outcome
extend of LV dysfunction
standard post MI therapy
aspirin
beta block
HMG-CoA inhibitor
ACE inhibitor (if LV dysfunction)