6.16 - Electrocardiography Analysis Flashcards
1
Q
What is the basic passage of electrical conduction through the heart?
A
- SAN - spontaneously active cells, conducts current down atria and internodal pathways –> P-wave of ECG
- AVN - current slows down between atria and ventricles = atrial muscle contraction to expel blood into ventricles
- Septum and His-Purkinje system - current travels through septum –> His-Purkinje system. Rapid contraction through left and right bundles to Purkinje fibres leading to ventricular contraction –> QRS complex of ECG
- cardiac muscle relaxes and membrane potential recovers/repolarises –> T-wave of ECG
2
Q
How many electrodes are placed and how many leads are generated?
A
- 10 electrodes –> 12 lead recording
- limb leads (I, II, III aVR, aVL, aVF) - coronal view
- chest leads (V1 to V6) - axial view
3
Q
What are chest (precordial) leads?
A
- V1 - 4th intercostal space (ICS), right margin of sternum
- V2 - 4th ICS along the left margin of the sternum
- V3 - midway between V2 and V4
- V4 - 5th ICS, mid-clavicular line
- V5 - 5th ICS, anterior axillary line
- V6 - 5th ICS, mid-axillary line
4
Q
What is axis deviation?
A
- change in heart position (seen in taller people)
- OR change in position of electrical conduction
- e.g. if right bundle branch gets damaged then left branch becomes more prominent, changing the conduction –> aVF graph will show greatest change in electrical conduction
- right and left axis deviation (RAD & LAD)
5
Q
Which leads suggest which types of axis deviation?
A
- 0 to -90 degrees –> LAD
- 90 to +/- 180 degrees –> RAD
- 0 = I
- 60 = II
- 90 = aVF
- 120 = III
- -150 = aVR
- -30 = aVL
6
Q
Where does lead II go to and from?
A
- from top right to bottom left and shows the biggest QRS wave of all the leads
- if we tilt the heart slightly clockwise, the heart configuration is different and the nodes are no longer parallel to the lead II = less deflection of the QRS wave
7
Q
What would changes in atrial conduction (with examples) do to the ECG?
A
- change the P-wave
- atrial fibrillation - loss of P-wave
- atrial flutter - sawtooth pattern
- this aberrant activity is caused by other cells in heart forming pacemaker potentials
- sometimes these little fluttery currents may not be big enough to pass through AV node to cause ventricular contraction
8
Q
What would happen to the ECG if there was a barrier/problem in conduction between atria to ventricles/at AVN (e.g. cell destruction)?
A
- impacts P-R interval - longer = bigger gap between P-wave and QRS complex initiation
- first degree heart block - delay in conduction (caused by a kind of ‘traffic jam’ at AVN)
- second/third degree heart block - loss of conduction caused by a proper blockage at AVN
- we get missing QRS complexes after P-waves and no T-wave either since that is associated with ventricular repolarisation
9
Q
What do the squares represent in an ECG?
A
- 25mm/sec
- small square = 40ms (0.04s)
- big square = 200ms (0.2s)
- 5 large squares = 1 second
10
Q
How do we calculate heart rate?
A
- bpm –> 60s = 300 large squares
- heart rate = 300/large squares (in one heart beat)
- e.g. 300/3.3 = 91bpm
11
Q
What can we find out about the rhythm from an ECG?
A
- P-R interval: small squares x 40 (e.g. 5 x 40 = 200ms)
- QRS duration: small squares x 40 (e.g. 2.5 x 40 = 100ms)
- ST segment
12
Q
How do we figure out the axis?
A
- using limb leads
- e.g. here lead I is biggest suggesting it is going along lead I
- but lead III upside down = going away from lead III
- between 0 and -30 degrees –> LAD?
- look at lead I and AVF (perpendicular to each other) - if peaks same direction then normal, if lead I up and AVF down (Leaving each other) = left axis deviation, if lead I down and AVF up (towards each other) = right axis deviation.