5 - EKG II: Clinical (Kim) Flashcards

1
Q

****What is a stardard strip for a 12 lead EKG?

What is a rhythm strip?

VERIFY W/DR KIM VIA EMAIL

A

10 second

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is the count off method?

A

Start at peak of 1st QRS complex

Each dark line is the following:

300-150-100-75-60-50

2nd peak falls between those numbers

Would not work very well for an irregular heart beat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How would you measure heart rate using markers on an EKG?

What method is the most accurate to measure a heart rate?

A

Count QRS complexes in the 6-sec, multiply by 10

Markers are often 3-sec intervals

Count the QRS peaks on the strip, multiply by 6

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What should you look for to indicate normal SA Rhythm?

A

Every P Wave is followed by QRS Wave

Every QRS Wave is preceded by a P Wave

The P Wave is upright in Leads: I, II, III

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How do you quickly asses the Mean QRS Axis?

A

Lead I: + or -

Positive: -90 to +90

Lead II: + or -

Positive: -30 tp +150

Overlap gives -30 to +90 normal range

Predominantly UPRIGHT QRS in Leads I and II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Clinical: EKG Presentation of Right Ventricular Hypertrophy?

A

V1 / V2: Large R Wave

Right Axis Deviation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Clinical: EKG Presentation of Left Ventricular Hypertrophy?

A

V5, V6, I, aVL: Taller than normal R Waves

V1, V2: Deep S Waves

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Clinical: Right Bundle Branch Block (RBBB)

A

Normal right ventricle depolarization interrupted

Contraction is spread from Right Ventricle via cell-to-cell, no bundle of His–QRS Widened, Late Current in direction of right ventricle

V1 will have RSR’ Wave (Rabbit Ears), V6 S Wave

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Clinical: Left Bundle Branch Block

A

Widened QRS

Notched R in V6

No R, prominent S in V1

Downward directed QRS in V1

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Fascicular Blocks (hemiblocks)

Left Anterior Fascicle

Left Posterior Fascicle

A

Blockage of divisions after the bundles

LAF - Anterior, Superior, Lateral LV

LPF - Posterior, Inferior, Medial LV

Do not widen QRS

LAFB most common, Left Axis Deviation of Mean QRS Axis

LPFB - Right Axis Deviation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Left Axis Deviation

Right Axis Deviation

A

Left Axis: I (+) , II (-), aVF (-)

Right Axis: I (-), aVF (+)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Clinical: Pathologic Q Waves in Myocardial Infarction?

A

Irreversible necrosis of heart can be marked by formation of pathologic Q waves

More prominant that regular, will form in leads overlying infarcted tissue

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What would be the leads with pathologic Q waves in the following areas?

Inferior

Septal

Anterior

Lateral

Posterior*

A

Inferior: II, III, aVf

Septal: V1, V2

Anterior: V3, V4

Lateral, V5, B6, I, aVL

Posterior: V1, V2 (tall R, not Q)

Posterior due to no leads on back, will NOT be RAD (RVH causes RAD, Post. MI does NOT)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Clinical: Transient Myocardial Ischemia

A

Abnormality of ST Segment and T waves–related to coronary artery disease

ST Segment Depression and T Wave flattening/inversion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Clinical: Acute ST Segment Elevation Myocardial Infarction (Acute STEMI)

A

ST Elevation, Tombstoning of T Waves

T Wave inversion, decreased R wave

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Lead Locations:

Inferior Leads

High Lateral Leads

Low Lateral Leads

Septal Leads

Anterior Leads

A

Inferior Leads: II, III, aVF

High Lateral Leads: I, aVL

Low Lateral Leads: V5, V6

Septal Leads: V1, V2

Anterior Leads: V3, V4

17
Q

What is meant by currents of injury?

A

Possible cause for deviation of the ST segment as result of injur

A voltage gradient exists between normal and ischemic zones, leading to current flow between these regions

18
Q
A
19
Q

Clinical: Acute Non-ST Segment Elevation Myocardial Infarction (non-STEMI)

A

Cause: Usually acute partially occlusive coronary thrombus; result in ST segment depression and/or T Wave inversion (rather than elevation)

20
Q

What condition can cause the following:

  1. ST ‘scooped’ Depression/Mild PR Prolongation
  2. Tall “peaked” T Waves (Castle)
  3. Flattened P, Wide QRS
  4. ST Depression, Flat T, U Wave
  5. Shortened QT Inerval
  6. Prolonged QT Inerval
A
  1. Digoxin Therapy
  2. Hyperkalemia (high potassium)
  3. Severe Hyperkalemeia
  4. Hypokalemia (low potassium)
  5. Hypercalcemia
  6. Hypocalcemia
21
Q
A