40. Diseases of the Nose Flashcards
bony nose on the nasal ridge, the rest is cartilage, some fatty tissue
relevant anatomy
inferior turbinate which is what you can see, middle turbinate, superior turbinate
internal structures of the nose
internal carotid > opthalmic artery > anterior ethmoidal artery and posterior ethmoidal artery
blood supply to the lateral wall
external carotid > maxillary artery > greater palatine artery, lesser palatine artery, and sphenopalatine artery > branches of sphenopalatine artery
blood supply to the lateral wall
facial artery > branches of the facial artery
blood supply to the lateral wall
very similar to lateral wall supply, kiesselbach’s plexus bracnes of the internal and external carotid
blood supply to the septum
upper part of nose/face drains via angular vein intracranially to the cavernous sinus, also drainage via fiacial to jugular vein
venous drainage of the nose
upper lip, philtrum, and nose, contains cavernous sinus, oculomotor nerve, trochlear nerve, opthalmic nerve, abducent nerve, maxillary nerve, internal carotid artery, sella turcia, sphenoid sinus, sphenoid bone, nasopharynx, pituitRY GLAND, optic chiasm
danger triangle
primarily respiratory epithelium, specialized olfactory area, some squamous epithelium at the nares
nasal mucosa
increased secretions, nasal obstruction, bleeding, foul odor, pain, altered sense of smell
symptoms of nasal pathology
nasal speculum opens up and down, or nasal endoscopy but needs topical anesthetic, can visualize nasopharynx
examination of the nose
palpate all the facial bones so you know it is isolated, no imaging if tender only at the nasal bridge, the nose is aligned, the nareas are patent, and there is no septal hematoma, management of most nasal fraftures is deferred until swelling resolves, for extensive facial trauma CT is the preferred imaging modality
isolated nasal trauma
can compress the septum and cause necrosis, can become infected, drainage followed by packing, with ENT follow up, remember image
septal hematoma
common complaint, usually not a sign of systemic disease, common causes include trauma, nosepicking, dry mucosa due to dry winters, allergies, less common causes include coagulopathy, thrombocytopenia, and cancer
epistaxis
not common 1:150,000 people is about 50/year in the US, primarily adolescent males, can present with massive epistaxis or mass effect, benign but grows aggressively
juvenile nasopharyngeal angiofibroma
personal history of bleeding issues, blood in the stool or urine, gum bleeding, anticoagulant medications, family history of bleeding disorders, prior episodes on the same side with what triggers them, if treatment was attempted
epistaxis history
management is done stepwise, apply pressure, add topical vasoconstrictor oxymetazoline, cautery only if you can see the source and only do one side of the septum, then tamponade, then surgical management
epistaxis history
pressure, topical vasoconstrictor oxymetazoline, cautery if you can see the source, only do one side of the septum, tamponade, surgical management
epistaxis management
if unable to control pressure +/- topical vasoconstri ction +/- cautery, proceed with packing, can be vaseline gauze or commercial products, packing remains for 24-48 hours, packing remains for 24-48 hours, oral antibiotics are not uniformly recommended but consider for prevention of sinusitis and toxic shock syndrome, may pack both sides if first doesn’t stop it, if still bleeding suspect posterior bleed
anterior expistaxis
when efforts to stop epistasis as already described don’t work you may have a posterior bleed, posterior pack and also anterior pack, will often need inpatient care
posterior epistaxis
uncontrolled bleeding or bleeding that recurs when pack is removed may need a surgical or interventional radiology approach
if all else fails
don’t touch your nose, don’t put anything in your nose like finger, tissue, or gauze, use a humidifier when sleeping, use saline spray, ENT referral if the patient needed more than anterior packing, assess for causes/sources
epistaxis after care
most common in children, can cause purulent drainage, foul odor, bleeding, and pain, acutely parents can see the child put it there, but subacutely can cause unilateral purulent drainage with bad smell, can be removed with parent’s kiss, balloon catheter, forceps, caveats with button batteries, concern for aspiration, check for other orifices, consider sinus infection
nasal foreign body
make sure there is only one foreign body, if subacute presentation with purulent discharger, treat with oral antibiotics for presumed sinus infection, consult ENT if injury
nasal foreign body aftercare
itchy nose, stuffiness, watery nasal discharge, sneezing, itchy etes, seasonal with pollen or perennial, pale boggy mucosa with swollen turbinates and watery nasal discharge, treat with oral medications like antihistamines, and leukotriene modifiers, topical meds intranasally like antihistamines and steroids, or immunotherapy, avoid allergens
allergic rhinitis
nasal polyps
generally bilateral in older patients, related to chronid inflammation or infection, red flags include unilateral polyps which may be malignancy, children or young adults with polyps consider underlying disorders like cystic fibrosis, obstructive complains as well as allergy, fullness, decreased sense of smell, treat with topical steroids, oral steroids if very symptomatic, surgical resetction
nasal polyps
overuse of oxymetazoline and or phenylephrine nasal spray, is real
rhinitis medicamentosa
deviated septum, perforated septum
other causes of nasal obstruction symptoms
acute painful inflammation of the entrance to the nose, usually no complaint of obstruction, erythema may have pustule and or crusting, tendnerness, usually cause by s aureus, treat with topical muciprocin or antistaph antibiotic, venous drainage is relevant
nasal vestibulitis
common cold caused by a variety of viruses, accompanying systemic disease and other respiratory symptoms, course is 7-10 days with congestion thick and sometimes discolored toward the middle of the course, treatment is symptomatic, no antibiotics unless there is a sinus infection
acute rhinitis
failry common especially as people get older, can lead to loss of enjoyment of food and ultimately malnutrition and depression, obstructive diseases 30% like sinusitis, allergies, polyps, post infetious like 15-30%, with SARS cov-2 most patients who have lost smell recover it spontaneously within a few weeks, trauma 10-30% direct mucosal injury to the CNS anywhere on the pathway, medications, drugs of abuse, aging, CNS disease like alzheimer’s, parkinson’s, multiple sclerosis, stroke, pseudotumor, cerebri, idiopathic, workup is directed by history, may include CT scan to evaluate sinus disease or MRI to evaluate CNS, treatment depends on cause, often no treatment with patience required
olfactory dysfunction
olfactory neurons > bulb > nerves > nuclei, cortex, and amygdala
olfactory pathway