wk 3 respiratory ppt/2 Flashcards

1
Q

ARDS - proning is typically reserved for

A

refractory hypoxemia not responding to other therapies

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2
Q

rupture of overdistended alveoli during mechanical ventilation

A

barotrauma

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3
Q

ARDS early s/s

5

A
  1. dyspnea
  2. tachypnea
  3. cough
  4. restlessness
  5. chest auscultation normal or fine, scattered crackles
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4
Q

ARDS etiology

A

develops from a variety of direct and indirect lung injuries; sepsis is most common cause

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5
Q

occurs when large tidal volumes used to ventilate noncompliant lungs

A

volutrauma

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6
Q

ARDS early chest x ray

A

may be normal or may show minimal scattered interstitial infiltrates

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7
Q

ARDS mgmt - mechanical ventilation

3

A
  1. PEEP opens collapsed alveoli
  2. higher levels of PEEP are often needed to maintain PaO2 >60
  3. high levels of PEEP can compromise venous return
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8
Q

how to prevent VAP

A

oral hygiene

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9
Q

high pressure alarm - do what?

2

A

suction or they could be fighting the vent (increased sedation)

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10
Q

ARDS late chest x ray

A

diffuse and extensive bilateral interstitial and alveolar infiltrates “white out”

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11
Q

can a PICC be used for TPN

A

yes

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12
Q

barotrauma

A

rupture of overdistended alveoli during mechanical ventilation

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13
Q

if on oxygen 60% for > 24 hours

3

A
  1. increase in pulmonary capillary pressure
  2. fibrotic changes in alveoli
  3. decreased surfactant
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14
Q

ARDS early ABGs

A

mild hypoxemia and respiratory alkalosis caused by hyperventilation

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15
Q

RF drug therapy

5

A
  1. bronchodilators
  2. steroids - inflammation
  3. diuretics - reduce pulmonary congestion
  4. antibiotics - pulmonary infection
  5. benzos, narcotics - reduce anxiety and pain
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16
Q

ARDS oxygen therapy

2

A
  1. give lowest concentration that results in PaO2 >60

2. common to intubate with mechanical ventilation because PaO2 cannot be maintained

17
Q

volutrauma

A

occurs when large tidal volumes used to ventilate noncompliant lungs

18
Q

what are two s/s that a patient needs to be intubated soon

A
  1. retractions

2. elevated lactic acid levels

19
Q

ARDS late - s/s worsen with

A

progression of fluid accumulation and decreased lung compliance

20
Q

patho changes of ARDS thought to be due to

A

stimulation of inflammatory and immune systems

21
Q

sudden progressive form of acute RF

A

ARDS

22
Q

ARDS late s/s

6

A
  1. suprasternal retractions
  2. tachycardia
  3. diaphoresis
  4. changes in sensorium with decreased mentation
  5. cyanosis
  6. pallor
23
Q

septic pts are at high risk for developing what

A

ARDS

24
Q

ARDS - complications of treatment

5

A
  1. ventilator associated pneumonia
  2. barotrauma
  3. volutrauma
  4. high risk for stress ulcers
  5. renal failure
25
Q

ARDS results in

5

A
  1. severe dyspnea
  2. hypoxia
  3. decreased lung compliance
  4. diffuse pulmonary infiltrates
  5. loss of recoil from scarring
26
Q

ARDS late ABGs

A

hypoxemia and a PaO2/FiO2 ratio < 200 despite increased FiO2

27
Q

ARDS - alveoli

A

alveolar capillary membrane becomes damaged and more permeable to intravascular fluid; alveoli fill with fluid

28
Q

drug for pulmonary congestion if HF is present

A

nitrates

29
Q

ARDS late - pulmonary function tests reveal

A

decreased compliance and lung volume

30
Q
ARDS positioning strategies
4
- turn
-mediastinal
-fluid 
-don't
A
  1. turn from supine to prone position - may be sufficient to reduce inspired oxygen or PEEP
  2. mediastinal and heart contents place more pressure on lungs with supine
  3. fluid pools in dependent regions of lungs
  4. don’t lay them on their good side