week 9: language Flashcards

1
Q

elements of language

A
phonemes
morphemes
semantics
syntax
grammar
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what are phonemes?

A

the smallest unit of sound that makes a difference to the meaning (c to car)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what are morphemes?

A

the smallest unit of language that has meaning (ing,un,able)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what is semantics?

A

meanings of words or sentences

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what is aphasia?

A

inability/impaired ability to produce or understand speech and writing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

signs of aphasia

A

paraphasia
neologism
nonfluent speech

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what is paraphasia?

A

the substitution for a word by sound, an incorrect word or an unintended word eg. dat instead of hat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is neologism?

A

an entirely made-up word

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is nonfluent speech?

A

talking with considerable effort

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what other impairments do aphasia patients experience?

A

agraphia/dysgraphia

alexia/dyselxia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is agraphia/dysgraphia?

A

acquired or developed the inability to write/impairment of writing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is alexia/dyslexia?

A

acquired/developed inability to read/impairment of reading

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

brain regions involved in language

A

Temporal lobe

  • Broca’s area
  • Wernicke’s area
  • primary auditory area

primary motor cortex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

patient tan and paul broca

A
Paul Broca  
patient Leborgne (Tan)
- right hemiparesis (right side paralysed) 
- loss of speech 
- comprehension ok
- damage to Broca's area
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Karl wernickes patients

A

2 patients with deficits in comprehension and fluent, incomprehensible speech
damage was found in posterior to primary auditory cortex (wernickes area)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

explain lichtheims house

A

A represents auditory area (wernickes)
M represents motor/spoken word (Broca)
B represents concept area
red lines represent an area where something could go wrong

17
Q

How did the Wernickes-Geschwind model of language develop?

A

combined concept of Wernickes and Lichtheims findings

18
Q

how do we hear words?

A

The auditory cortex transmits information about the sounds to Wernickes area where the sounds are analysed and decoded

19
Q

How do we speak?

A

for a word to be spoken, Wernickes area must transmit this information to Brocas area where a speech plan is activated. Brocas area sends the plan to the motor cortex which controls the muscles needed to speak

20
Q

what is the arcuate fasciculus?

A

the bundle of nerve fibres formed by axons transmitting info from wernickes to brocas area

21
Q

How do we say the name of a seen object or word?

A

Visual information is transferred to the angular gyrus which arouses the auditory pattern in wernickes area. the auditory form is then transmitted via the arcuated fasciculus to brocas area. then the model for the spoken form is activated and transmitted to the motor cortex and the word is spoken

22
Q

types of aphasias

A

brocas
wernickes
conduction

23
Q

how does one get brocas aphasia?

A

damaged to the brocas area which is the posterior part of the inferior frontal convolution in the left hemisphere (left frontal lesion)

24
Q

what area is brocas area anterior to?

A

the motor cortex which controls the muscles involved in speech (tongue, jaw, throat)

25
Q

damage to the brocas area mean?

A

damage to this region destroys memory traces of the movements required to produce speech

26
Q

Broca’s aphasia symptoms

A
  • slow/high effort deliberate speech
  • telegraphic speech
  • difficulty finding/saying correct phoneme/words
  • anomia
  • phonemic paraphasia
  • reduced verbal fluency
  • repetition
  • hemiplegia (paralysis of side of body)
  • insight intact
27
Q

summary of brocas aphasia symptoms

A

categorised by difficulties speaking but being consciously aware of the conversations and their challenges

28
Q

phonemic paraphasias

A

unable to find appropriate phoneme so swaps it for a similar one
eg. pill for spill

29
Q

telegraphic speech

A

simple grammatical structure
focus on nouns singularly
eg. eat dinner

30
Q

anomia

A

inability to come up with desired word

31
Q

how does one get wernickes aphasia?

A

results from damage to the vicinity of Wernicke’s area in the left superior temporal cortex posterior to the primary auditory cortex

32
Q

damage to the wernickes area mean?

A

unable to comprehend
wernickes area is involved in memory for sounds of speech and mediates the linking of the auditory representations of words with their meaning

33
Q

wernickes aphasia symptoms

A
  • inability to comprehend speech
    fluent speech with no distress with the correct speed and pattern
  • jargon makes their speech difficult to interpret
  • semantic paraphasias
  • neologism
  • talks around topic
    -inability to repeat words/phrases
  • no paralysis
  • poor insight into language difficulties
  • understands facial expressions
34
Q

semantic paraphasias

A

errors that have a semantic similarity to the desired word

eg, son for daughter

35
Q

conduction aphasia

A
  • normal speech comprehension and production
  • difficulties in repetition of non-meaningful words and word sequences
  • cannot say words they don’t have a mental image for eg. can say fruit but cannot make up a word