Week 3: Bone Diseases Flashcards

1
Q

Joint effusion

A

Accumulation of fluid in the joint associated with an underlying
condition
 There are typical
appearances of effusions in the ankle, knee, elbow and hip
that aid in the diagnosis of subtle
pathologies

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2
Q

Sail Sign

A

in the elbow. Anterior fat pad elevation looks like a
sail and is a sign of joint effusion and
potential fracture

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3
Q

Legge-Calve-Perthe’s Disease

A

Flattening of the femoral head due to
vascular interruption
 Avascular necrosis of the capital
femoral epiphysis of the femoral head
 Most common in Caucasian males 4-8 years old

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4
Q

Osgood schlatter’s

A

Incomplete separation or
avulsion of the tibial tuberosity
 A chronic fatigue injury due to
repeated microtrauma at the
patellar ligament insertion onto
the tibial tuberosity, usually
affecting boys between 10-15
years

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5
Q

Osteopetrosis

A

Rare, hereditary bone dysplasia causes “marble bones”, severity varies
 Normal resorptive calcium mechanism is absent, interferes with normal bone replacement
 Prevents bone marrow from forming, patients can be anemic
 Bones become brittle and stress fracture often
 Technical factors (mAs, kV) must be increased to account for increased bone formation

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6
Q

Osteogenesis imperfecta

A

brittle bone disease):
an inherited generalized disorder of connective tissue
 Characterized by fragile bones, bowed legs and an
unusual blue color of the normally white sclera of the
eye
 Patients suffer repeated fractures
 Healing process can cause bizarre deformities due to
bizarre callus formation
 Skull ossification is slow, producing Wormian bones
(wide sutures and intra-sutural bone development)
 Extreme care must be taken when positioning these
patients
 Lower kV to compensate for the loss of bone quality
 Radiographically: severe osteoporosis, thin defective
cortices, Wormian bones in skull

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7
Q

Achondroplasia

A

Hereditary disorder
 The most common form of
dwarfism
 Growth plates of the long bones
don’t proliferate new growth
properly, leading to a normal
trunk length but abnormally short
limbs, and large head

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8
Q

Acromegaly

A

Excessive growth hormone AFTER bone growth has stopped
 Often caused by a pituitary tumor
 Sometimes follows gigantism (excessive
growth hormone BEFORE bone growth has stopped) if hypersecretion of growth hormone doesn’t cease at maturity

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9
Q

Congenital (Developmental) Hip Dysplasia

A

Results from incomplete acetabulum formation caused by physiologic & mechanical factors
 Femoral head pops out of joint during flexion and abduction
 May use x-ray or US for diagnosis
 Imaging appearance:
 AP pelvis & bilateral frog-leg (Cleaves)
views are required to make a diagnosis
 AP - larger joint space
 Cleaves – hip dislocation posteriorly and superiorly

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10
Q

Rheumatoid Arthritis

A

Chronic systemic disease of unknown
cause
 Appears primarily as a non-infectious
inflammatory arthritis of the small
joints of the hands and feet
Begins as an inflammation of the
synovial membrane (synovitis) that
lines the joints

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11
Q

Bursitis

A

Inflammation of the bursae
(small fluid filled sac within joint
space to reduce friction)
 Causes pain, frozen joints
(immobility)
 Not seen radiographically except
by calcification deposits in
adjacent tendons (calcific
tendonitis)
 Needs US diagnosis

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11
Q

Osteoarthritis

A

Degenerative condition
 Loss of joint cartilage and reactive new bone formation
 Usually affects the weight-bearing
joints (spine, hip, knee and ankle) and
the interphalangeal joints of the
fingers

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12
Q

Osteomyelitis

A

An inflammation of the bone and
bone marrow
 Caused by infectious organism
 A soft tissue infection may
spread from a skin abscess or
decubitus ulcer eg. foot of a
diabetic patient, to cause
cellulitis and eventually
osteomyelitis in adjacent bones

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13
Q

Osteomyelitis Image Appearance

A

Imaging Appearance:
* Localized, deep soft tissue
swelling adjacent to the
metaphysis
* Metaphyseal lucency which
progresses to bone destruction
(ragged, moth-eaten
appearance)

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14
Q

Osteoporosis

A

Generalized or localized
deficiency of bone density
– bone mass is decreased
 Lack of calcium and
vitamin D
 Compression fractures
common in spines;
fractured wrists and hips
in elderly

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15
Q

Osteopenia

A

ecreased mineral bone density
 Can be from disease or disuse
 Osteopenia can be risk factor for osteoporosis

16
Q

Osteomalacia

A

Loss of bone density due to nonmineralized
osteoid
 Caused by vitamin D deficiency
 Makes bones soft

17
Q

Gout

A

A disorder in the metabolism of purine (a component of nucleic acids) in which an increase in the blood level of uric acid leads to the deposition of uric acid
crystals in the joints, cartilage, and
kidney. Urate crystals can be present in
the joint spaces.
 Common in the first metatarsophalangeal
joint but may attack any joint
 Extremely painful, patient may not
tolerate blankets on foot
 Imaging appearance:
 Joint inflammation (effusion) and
destruction

17
Q

Rickets

A

Pediatric disease of osteomalacia
 Vitamin D deficiency
Imaging appearance
 Cupped and frayed metaphyseal ends of the bone
 Bowing of weight bearing bones

18
Q

Paget’s Disease
(osteitis
deformans)

A

A chronic metabolic bone
disease; destruction of bone followed by a reparative process results
in weakened, deformed, and thickened bone structures that tend to
fracture easily
 Commonly affects the
skull, long bones, pelvis
and vertebra

19
Q

Ischemic Bone Necrosis

A

A condition that occurs when there is loss of blood to the bone;
eventually without adequate blood supply, the bone dies.
 Also known as avascular
necrosis, osteonecrosis,
and aseptic necrosis
 Most common site is the
femoral

20
Q

Osteochondritis Dissecans

A

The end result of an aseptic separation of bony or cartilaginous fragment
 The piece can become a “loose body” in the joint
 Thought to be from repetitive stress in
young athletes
often in condyles of knees

21
Q

SNAT

A

suspected non-accidental trauma
Battered child syndrome