Ventricular Rythms

Question 1

QRS

Answer 1

> .12 if initiated in the ventricles

a beat >.12 isn’t necessarily initiated in the ventricles

Question 2

PVC

Answer 2

Wide and bizarre >.12 QRS

Question 3

Interpolated PVC

Answer 3

doesn’t affect rythm. falls in the middle of 2 beats

Question 4

Compensatory pause

Answer 4

PVC interrupts normal rhythm

Question 5

sinus brady with bigeminal PVC’s

Answer 5

electrical twice the mechanical

Question 6

Run of PVC’s (or burst)

Answer 6

3 or more PVC’s in a row

Question 7

Couplet of PVC’s in a row

Answer 7

2 PVC’s in a row

Question 8

electrical doesn’t match mechanical pulse

Answer 8

Pulsus deficit

Question 9

Frequent PVC’s are = to

Answer 9

6 or more per minute

Question 10

Ventricular escape beats

Answer 10

A. Ectopic occurs after pause in which supraventricular pacemaker fails to initiate impulse
B. Protective mechanism

Question 11

VTAK

Answer 11

Heart extremely irritable
Ventricular focus could speed up and over ride pacer sites
Polymorphic vs. monomorphic

Question 12

Sustained VTAK

Answer 12

> 30 seconds

Question 13

Nonsustained VTAK

Answer 13

<30 seconds

Question 14

6 characteristics of VTAK

Answer 14

1. Rate is 150-250
2. Rhythm is regular
3. P wave none usually, if there doesn’t conduct
4. PRI—none
5. QRS
   a. wide bizarre
   b. > .12 sec
6. T wave—–not seen

Question 15

> 40 IVR

Answer 15

accelerated idioventricular rhythm

Question 16

5 H’s

Answer 16

Hypovolemia
Hypoxia
Hydrogen-ion(acidosis)
Hypo/hyperkalemia
Hypothermia

Question 17

5 T’S

Answer 17

Tablets (OD)
Tamponade
Tension Pheumo
Thrombosis (coronary)
Thrombosis (emboli)

Question 18

LIDO

Answer 18

protocol

Question 19

NACO3

Answer 19

protocol

Question 20

Amiodarone

Answer 20

protocol

Question 21

S127

Answer 21

protocol

Question 22

etiology of PVC

Answer 22

1. Damage to His-Purkinje system
2. Increased sympathetic tone
3. Hypoxia or Acidosis
4. AMI or myocardial ischemia or CHF
5. Electrolyte imbalances (K, Ca++, Mg)
6. Digitalis toxicity
7. Use of cyclic antidepressants, phenothiazines
8. Stimulate uses

Question 23

Etiology of VTAK

Answer 23

Same causes as PVC's
Ischemia
Hypoxia
Electrolytes
Acid-base problems
Increased sympathetic tone

Question 24

Polymorphic VTAK

Answer 24

Torsades de Pointes

Question 25

Torsades Itiology

Answer 25

Drug induced
Eating disorder
Electrolyte imbalances (magnesium, potassium, calcium)

Question 26

V flutter

Answer 26

often degenerates to vfib

Question 27

V Flutter symptoms

Answer 27

Palpitations Dyspnea
Dizziness Anxiety
Diaphoresis Angina
Seizures

Question 28

V Fib Etiology

Answer 28

1. Usually preceded by ventricular irritability
2. Ventricular damage
3. Hypoxia
4. Electrolyte disturbances
5. Electric shock
6. Malfunctioning pacemaker
7. Drugs

Question 29

Often first rhythm following defibrillation

Answer 29

Idioventricular