Vascular Surgery Flashcards

1
Q

Risk factors for Abdominal Aortic Aneurysm (AAA):

A
  • Male, older age, family history
  • Hyperlipidaemia, hypertension
  • Smoking
  • Inflammatory (Takayasu’s aortitis, Behcet’s) or connective tissue (Marfans, Ehlers Danlos)
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2
Q

ABPI of 1.3 or more means what ulcer and what is the pathophysiology? Would you use leg compression?

A

Blood vessel hardening from PVS.
Most likely venous ulcer.
You can use leg compression.

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3
Q

ABPI of less than 0.5 means what ulcer and what is the pathophysiology? Would you use leg compression?

A

Severe arterial damage.
Arterial ulcer.
Do not use leg compression.

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4
Q

What are the characteristics of an arterial ulcer? and what can you find around the affected area?

A

Painful ulcer, usually around the toes or heels.
Might have areas of gangrene around.
Cold to touch. No palpable pulses.
Low ABPI.

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5
Q

What are the characteristics of a venous ulcer? and what can you find around the affected area?

A

Painless ulcer, usually above the ankles.
Shallow and superficial.
Irregular shape

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6
Q

What skin changes can you see with venous insufficiency?

A

Oedema, skin pigmentation (haemosiderin), lipodermatosclerosis, eczema or thrombophlebitis

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7
Q

What is the typical description of an arterial ulcer?

A
Punched out appearance.
Smooth (regular) wound edges.
Usually lateral malleolus.
Lower extremities cool to touch. 
Pale shiny thin skin.
Minimal hair growth.
Minimal drainage from wound.
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8
Q

Risk factors for venous ulcers?

A

DVT.
Incompetent valves.
Varicose veins.

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9
Q

How do you define varicose veins?

A

Tortuous dilated segments of vein due to valvular incompetence.

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10
Q

What is the pathophysiology of varicose veins? Hint: valve

A

Incompetent valves which allows blood from the deep venous system to the superficial venous system at the sapheno-femoral junction and sapheno-popliteal junction.

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11
Q

Risk factors for varicose veins?

A

Pregnancy
Standing for long
Obesity
Family history

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12
Q

Complications of varicose veins?

A

Unsightly, pain, itching, aching, swelling.

Skin changes, ulceration, thrombophlebitis or bleeding.

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13
Q

What is the gold standard investigation for varicose veins?

A

Duplex ultrasound.

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14
Q

What are the invasive (3) and non-invasive management options for varicose veins?

A

Invasive: thermal ablation, foam sclerotherapy, vein ligation/stripping/avulsion.

Non: Compression stocking w patient education.

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15
Q

Causes of acute limb ischaemia?

A

Occlusion of the arterial supply due to:

embolization
thrombus in-situ
trauma (compartment syndrome)

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16
Q

What are the classic symptoms of acute limb ischaemia?

A

Common: Pain, pulselessness, pallor

Paraesthesia, paralysis, perishing cold.

17
Q

Signs of Critical Limb Ischaemia

A
  • 6Ps
  • Gangrene/necrosis
  • Unpalpable pulses
  • Pallor on elevation
  • Delayed cap refill time
  • Ulceration
  • ABPI < 0.5
18
Q

What could absent radial pulses mean?

A
  • Acute limb ischaemia
  • Chronic peripheral vascular disease (atheroma or vasculitis)
  • Graft site for pv CABG
19
Q

What is a Marjolin Ulcer?

A

Aggressive ulcerating form of squamous cell carcinoma.

  • Occurs at sites of chronic inflammation i.e. burns , osteomyelitis
  • Usually lower limb.
20
Q

Management for neuropathic ulcers:

  • Cons (1)
  • Surg (1)
A

Cushioned shoes to reduce callous formation

Amputation

21
Q

What is a painful ulcer related to inflammatory bowl disease or rheumatoid arthritis?

A

Pyoderma gangrenosum.

22
Q

Tearing chest pain radiating to the back
Tachycardia and hypotension
New aortic regurgitation murmur
Signs of end organ ischaemia (6P, reduced urine)

A

Aortic dissection

23
Q

What investigations would you order for ‘‘tearing chest pain radiating to the back”

A

Bedsides: General obs, ECG

Bloods: FBC, U&Es, LFTs, troponin, coag studies, cross match 4u, ABG (lactate for ischaemia)

Imaging: CT angiogram (diagnose, classify and assist w surgery) and Transoesophageal ECHO

24
Q

DaBakey & Stanford classification for Aortic Dissection
I, II, IIIa & IIIb
Type A & Type B

A

1: ascending aorta + aortic arch
2: ascending aorta
3a: distal to subclavian at descending until diaphragm
3b: extends beyond diaphragm until abdominal

A: 1 2 + a bit of descending
B: 3

25
Q

Acute management of Aortic Dissection?

A

High flow oxygen
IV assess via two large bore cannula - Fluids and crossmatch blood. Keep blood pressure below 110mmHg and enough for cerebral perfusion.

26
Q

Management for Type A and Type B (asymptomatic)

A

A: surgical management due to bad prognosis
- removal of ascending aorta and replacement with graft.

B: if asymptomatic, manage medically.

  • BP management via IV Labetalol (BB) or CCB if BB not tolerated.
  • rapidly lower the systolic pressure, pulse pressure, and pulse rate to minimise stress of the dissection and limited further propagation.
27
Q

Complications of Aortic Dissection?

A
  • MI secondary to coronary artery dissection
  • Aortic aneurysm
  • Aortic regurgitation
  • Aortic rupture
  • Cardiac tamponade
  • Stroke or paraplegia (spinal artery)