Uworld Mix 9/29 Flashcards
56m has progressive asthma symptoms. Patient has nighttime cough and wheezing. She sometimes needs her albuterol inhaler after meals. She is already on fluticason, albuterol, lisinopril, and aspirin with no change in her meds for YEARS. She fat. Not wheezing now. What is the next step in management?
PPI like esomeprazole - She has asthma exasperations from GERD. notice its post meals these happen.
65 m, COPD, Afib, HTN and T2DM - had SOB for 3 days, as well as runny nose, itchy eyes and sore throat. She is treated later with bronchodilatory, steroids, high flow facial mask, and lorazepam. 30 min later, has a tonic clonic seizure. Why do dis happenz
Carbon Dioxide retention - this is actually O2 induced. After sudden O2 administration cause vasodilation (cant vasoconstriction), decreased CO2 uptake, and decreased RR–> CO2 retention
What does hypercapnia cause cerebrovasculature to do
VASODILATION
LIst the timeline of complications after MI - Hours, Days, Weeks, Months
Reinfarction - hours to 2 days Vent Septal Rutpure - hours to 1 wk Free Wall Rupture - Hours - 2wks Papillary Muscle Rupture - 2 days to 1wk Pericarditis - 1 day to 3 months Left Ventricular Aneurysm - 5d to 3 months
How would a ventricular aneurysm present, and how would you confirm
Progressive decompensated HF from 5 days to 3 months after an MI, confirmed with Echocardiographay, showing thinned, dyskinetic wall.
How would a Ventricular Wall Rupture present?
A large pericardial effusion, causing tamponade, hypotension, elevated JVP, and then pulseless electrical activity
What would be the sign of ACUTE Liver Failure? theres 3 specific qualifiers
Signs for hepatic Encephalopathy, Elevated LFTs in the thousands, and INR >1.5
What differentiates acute liver failure from acute hepatitis?
Hepatic Encephalopathy. Acute Hepatitis has a much better prognosis. ACH could LEAD to ALF.
Would you see hyperbilirubinemia in Acute Liver Failure?
Yes, but it is NOT a requirement.
48 Year old Caucasion Male is SOB, out of breath. HE has a harsh Systolic mumur best heard at R2nd ICS radiating up the carotids. S4 is heard. What is the cause?
a Bicuspid aortic valve - he has CHF from aortic stenosis. a bicuspid aortic valve is the most common cause of aortic stenosis in patients under 70.
66m has SOB. started a week ago. recent hx is of stending for Coronary Artery Disease. He has smoked for 35 years, had a hx fo neumonia 6 mo ago, and is in mild respiratory distress. He has decreased breath sounds at the base. ph 7.46, pO2 73, pCO2 31 - whats dx is doing dis shit
CHF. the key here is the hix of coronary artery disease, the wheezing and the bibasilar crackles. Dont let the APPEARANCE of ABG fool you into COPD exasperation. THey have respiratory alkalosis. COPD has acidosis.
How would aspirine, alcohol, and cocaine cause bloody emesis?
ASA decreases the protective prostoglandin production, and cocaine results in vasoconstriction, the alcohol will cause mucosal erosion. This all causes a hemorrhage
What would you see in bronchoscopic biopsy for Histoplasmosis?
Granulomas with yeast forms
Patient had a bowel resection from crohns disease has been under parentereal nutrition for 2 years. Now they have gallstones causein RUQ pain. How did their hc cause this?
Gallbladder stasis - because of the resection, and parenteral nutrition, there isnt proper stimulation for CCK, which would have triggered gallbladder contraction.
When would you see estrogen induced increased cholesterol secretion is the cause of gallstones?
Pregnancy. Estrogen causes more cholesterol secretion, and progesterone causes reduction in bile acid secretion.
Would INCREASED enterohepatic recycling of bile acids cause gallstones?
NO
How would Crohns disease or ileal resection predispose to cholesterol gallstones
causing DECREASED recycling of bile acids.
34 m sexually active complaining of palpitations and dizziness. He travels frequently for works. LIver span is 8 cm and spleen isnt palpable. No Cervical lymph or skin rash. Only lab adnormality is 80k platelets. Platelet clumping has been ruled out. What does he have, and what would be next tests?
ITP - this is isolated thrombocytopenia. Could be either from decreased production, or increased destruction. Preliminary tests would be HIV, Hep C and EBV testing. However, since there is no LAN, EBV is less likely.
Sexualy active male has fever, sorethrough HA, skin rash. Rash is “spread to his entire body.” Has not been outdoors. There are several raised, grey mucosal patches. Has diffuse LAN.. HIV negative. What is the cause?
Syphilis. yes, you DO see oral lesions when secondary.
How would a patient with 2o Syphilis with EBV?
The rash in EBV is more UNLIKELY.
What is the range for normal calcium?
8.4-10.2