Urinary - Bladder Flashcards

1
Q

Which ligament (patent urachus) are we likely to cut during cystotomy that is firmly attached to bladder wall at the level of the umbilicus?

A

Ventral ligament *avoid lateral ligaments as they contain ureters*

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2
Q

Name the region/separation between the ureteral orifices and the urethra….

A

Trigone

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3
Q

What is the most common congenital abnormality of the bladder?

A

Urachal abnormalities

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4
Q

What is the holding layer of the bladder? What does this mean?

A

Submucosa! So whenever we suture the bladder, we must make sure we engage the submucosa

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5
Q

How long does it take for the bladder to heal just about to full strength?

A

~14 days! (Much better than most other abdominal organs)

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6
Q

What is the most common TYPE of urachal abnormality?

A

Vesicouracheal diverticulum

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7
Q

What is a persistent urachus?

A

A (tube) connection from the bladder all the way to the umbilicus; This causes dribbling of urine at the level of the umbilicus —> will cause urine scalding/omphalitis

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8
Q

How do we surgically treat a persistent urachus?

A

Remove the patent urachus (which should be the ventral ligament of the bladder): - ligate at the level of the bladder, dissect out, and ligate at the level of the umbilicus and remove that patent tissue, thru a ventral midline incision

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9
Q

What is a vesicouracheal diverticulum?

A

Diverticulum* at the level of the bladder wall, but it doesn’t continue all the way to the umbilicus - it becomes the ventral ligament of the bladder *Allows accumulation of urine pooling in that area - predisposing to UTIs, urolithiasis, urge incontinence

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10
Q

How do we surgically treat vesicouracheal diverticulum?

A

Partial cystectomy (cut out part of the bladder wall associated with the diverticulum) And Diverticulectomy (removing the remnant attached to the bladder wall)

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11
Q

What are the clinical signs of a ruptured bladder?

A

Sometimes none (asymptomatic) Many cases will show hematuria, anuria, abdominal pain In trauma cases, just assume you have a ruptured bladder until you can rule it out

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12
Q

What is the progression of signs associated with a bladder rupture?

A

Over a period of time, metabolic abnormalities start (12-24 hours)… Dehydration, acidosis, azotemia, hyperkalemia *death in 47-90 hours*

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13
Q

How do we rule out ruptured bladder?

A

Plain radiographs: -can see free abdominal fluid (distension) -absence of bladder -decreased serosol detail of the intestinal silhouette US: - fluid accumulation - US-guided FNA - visualize and evaluate bladder wall

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14
Q

What is the best and most accurate diagnostic modality for bladder rupture?

A

Positive Contrast Urethrocystogram: Taking urethral catheter, placing it in the bladder, and inject radio-opaque material into the bladder —> take rads and see if contrast media is leaking out of bladder (pathopneumonic)

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15
Q

How do we evaluate the fluid from abdominocentesis when diagnosing a ruptured bladder?

A

Urea: (small molecule; will diffuse across peritoneum) compare to serum BUN —> should be about equal [urea in peritoneal fluid and serum urea] Crea: (larger molecule; does not readily diffuse across peritoneum) Crea in peritoneal fluid should be > serum Crea

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16
Q

How is it that we can use the presence of hyperkalemia as a factor in diagnosing bladder ruptures?

A

Development of acidosis —> displacement of H+ with K+ —> K+ becomes extracellular —> hyperkalemia

17
Q

What is serosal patching?

A

Take a piece of serosa (of a mobile intestine - so the antimesenteric border) and you suture it to the level of incision of your ruptured bladder closure *helps to seal, even if incision breaks down, and will re-mucosalize*

18
Q

Although the dorsal approach to cystotomy was once advocated, the ventral approach is preferred because …?

A

Better visualization of the trigone (where stones hide) And you can see the ureteral orifice

19
Q

What type of suture has been known to predispose a patient to urolith formation?

A

Monofilament nonabsorbable *it’s ok to use this, but if you do, don’t expose it to the lumen! —> serosal—>muscular—>submucosal layer, but no bladder mucosa if we use this

20
Q

What suture do we choose to close the bladder?

A

Absorbable or non-absorbable, it depends the patient and presentation: PDS, monocryl, vicryl, dexon, prolene, nylon, etc

21
Q

What layer of the bladder does polypoid cystitis affect? What can it be confused with?

A

Mucosal layer; can be confused with TCC due to resemblance —> biopsy confirms

22
Q

Most common tumor of the urinary tract in the canine?

A

TCC

23
Q

Obesity, Insecticide exposure, Herbicide, And cyclophosphamide Are predisposing factors for…?

A

TCC

24
Q

What peculiar clinical signs may a patient with TCC present with?

A

Those similar to LUTD; Metastatic disease
—>lameness/coughing

25
Q

If we suspect TCC, what type of biopsy should we attempt for confirmation?

A

Transurethral biopsy
place urethral catheter (palpate the neoplasia rectally),
push it up against tumor, & inject a little saline; aspirate cells

*AVOID FNA*{could reseed!}

26
Q

How do we treat TCC?

A

Partial cystectomy with >1cm borders

Often involves trigone so may require salvage procedure:
-ureterocolonic anastomosis -ureterouterine anastomosis

Chemotherapy: - Piroxicam, Cisplatin, Mitoxantrone
*still even with Tx, MST: 4-6 mo :(

could do cystostomy tubes to buy time for the chemotherapeutics to work

27
Q

When would you use a non-absorbable suture in closing the bladder?

A

In immune-compromised patients;
but cannot pass thru the lumen of the bladder!!
—> predisposes to urolith formation

28
Q

Cushing goes thru the ____, ____, & ____ layer… and the Lambert just goes in the _____ layer.

A

Serosa, muscular, & submucosal layer… Seromuscular