Unit 2 and 3 Chapter 32 Cardiomyopathy and Pulmonary Edema Flashcards

1
Q

What is Pulmonary Edema?

A

*Fluid in and around the alveoli
*Interferes with gas exchange
*Increases work of breathing

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is present during x-ray with pulmonary edema?

A

Infiltration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

S/s of Pulmonary edema

A
  • Crackles
  • Dyspnea at rest
  • Disorientation or acute confusion (especially in older adults as early symptom)
  • Tachycardia
  • Hypertension or hypotension
  • Reduced urinary output
  • Cough with frothy, pink-tinged sputum
  • Premature ventricular contractions and other dysrhythmias * Anxiety
  • Restlessness
  • Lethargy
    *cold, clammy, or cyanotic
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Is Pulmonary Edema life-threatening?

A

A. yes

CPE leads to progressive deterioration of alveolar gas exchange and respiratory
failure. Without prompt recognition and treatment, a patient’s condition can
deteriorate rapidly – like 15 minutes!!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What is the priority for patients with pulomary edema?
A. administration of antihypertensive medications
B. 100% oxygenation via oxygenation device
C. administration of furosemide
D. Administration of desmopressin

A

B. 100% oxygenation via oxygenation device

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Treatment plan for patients dx with Pulmonary Edema

A
  • he priority nursing action is to administer oxygen therapy at 5 to 12 L/min by simple facemask or at 6 to 10 L/min by nonrebreathing mask with reservoir (which may deliver up to 100% oxygen) to promote gas exchange and perfusion
  • pply a pulse oximeter and titrate the oxygen flow to keep the patient’s oxygen saturation above 90%.
  • If the patient’s systolic blood pressure is above 100, administer sublingual nitroglycerin (NTG) as prescribed to decrease afterload and preload every 5 minutes for three doses while establishing IV access for additional drug therapy.
  • The health care provider prescribes rapid-acting diuretics, such as furosemide or bumetanide. Give furosemide IV push (IVP) over 1 to 2 minutes to avoid ototoxicity
  • . Monitor vital signs frequently, at least every 30 to 60 minutes.
  • Bumetanide may be administered IVP over 1 to 2 minutes to avoid ototoxicity or as a continuous infusion to provide consistent fluid removal over 24 hours.
  • If the patient’s blood pressure is adequate, IV morphine sulfate may be prescribed to reduce venous return (preload), decrease anxiety, and reduce the work of breathing.
  • Ultrafiltration
    *
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

When is Ultrafiltration used?

A

In severe cases of fluid overload and renal dysfunction or diuretic resistance, ultrafiltration may be used.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What are the benefits of Ultra filtration?

A
  • Decrease in cardiac filling pressures
  • Decrease in pulmonary arterial pressure
  • Increase in cardiac index
  • Reduction in norepinephrine, renin, and aldosterone
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is Cardiomyopathy?

A

Cardiomyopathy is a subacute or chronic disease of cardiac muscle, and the cause may be unknown.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What are the 4 types of Cardiacmyopathies?

A
  • -Dilated cardiomyopathy,
  • -Hypertrophic cardiomyopathy,
  • -Restrictive cardiomyopathy,
  • Arrhythmogenic right ventricular cardiomyopathy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is Dilated Cardiomyopathy

A

-Dilated cardiomyopathy (DCM) is the structural abnormality most commonly seen.
-Ventricular wall thickness is normal, but both ventricles are dilated and weakened (left ventricle is usually worse) and systolic function is impaired.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What population is most affected from Dilated cardiomyopathy?

A. 72 eldery women n
B. a 12 year old child
C. 40- year with past history of smoking
D. 68 year old with benign prosthetic hypertrophy

A

C. 40- year with past history of smoking

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the cause of Dilated Cardiomyopathy

A

Causes may include…
* alcohol abuse,
* chemotherapy,
* infection,
* inflammation,
* poor nutrition

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

S/s of Dilated Cardiomyopathy

A

-fatigue and weakness
-Left sides heart failure
-dysrythmias
-heart block
-systemic or pulmonary emboli*
-S3 and S4 gallops
-Moderate to severe cardiomegaly
-Decreased CO
-dyspnea on exertion (DOE),
-decreased exercise capacity,
-fatigue,
-palpitations
-low cardiac output
-hypertention

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is the life expectancy for a patient with Dilated Cardiomyopathy?
A. 10 years
B. 20 years
C. 5 years
D. 30 years

A

C. 5 years

will need consulting and family therapy due to poor prognosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Which of the following is the most common type of cardiomyopathy

A. Dilated cardiomyopathy,
B. Hypertrophic cardiomyopathy,
C. Restrictive cardiomyopathy,
D. Arrhythmogenic right ventricular cardiomyopathy

A

A. Dilated cardiomyopathy,

17
Q

What would you see on a ECG for a patient with Dilated cardiomyopathy

A

heart block

18
Q

What is the typical tx for Dilated cardiomyopathy

A

-heart failure tx
-heart transplant

19
Q

Life expectancy for Dilated cardiomyopathy without heart transplant

A

Most die within 5 years after the onset of manifestations.

20
Q

Heart transplant

A

Heart transplantation (surgical replacement with a donor heart) is the treatment of choice for patients with severe DCM cardiomyopathy.

The procedure may also be done for end-stage heart disease caused by coronary artery disease, valvular disease, or congenital heart disease.

21
Q

Candidate requirements for heart transplant

A
  • Life expectancy less than 1 year
  • Age generally younger than 65 years
  • New York Heart Association (NYHA) Class III or IV
  • Normal or only slightly increased pulmonary vascular resistance
  • Absence of active infection
  • Stable psychosocial status
  • No evidence of current drug or alcohol misuse
22
Q

S/s of Heart transplant rejection

A
  • Shortness of breath
  • Fatigue
  • Fluid gain (edema, increased weight)
  • Abdominal bloating
  • New bradycardia
  • Hypotension
  • Atrial fibrillation or flu er
  • Decreased activity tolerance
  • Decreased ejection fraction (late sign)
23
Q

What is a common complication of post Heart transplant

A

Be very careful about handwashing and aseptic technique because patients are immunosuppressed from drug therapy.

Infection is the major cause of death and usually develops in the immediate post-transplant period or during treatment for acute rejection.

24
Q

What type of medication should a patient be on with a heart transplant for life?

A

IMMUNOSUPPRESSANT

-Cyclosporine