TOPIC 11 - neuro part 2 Flashcards

1
Q

types of headaches

A

primary
tension
migraine
cluster
secondary

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2
Q

primary vs secondary headaches

A

primary are not caused by disease or another medical condition (ex: tension, migraine, cluster)

secondary are caused by another condition or disorder (ex: sinus infection, neck injury, brain tumor)

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3
Q

pain location of tension headaches

A

Tension headache is often described as a feeling of a weight in or on the head and/or a band squeezing the head.

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4
Q

pain location of migraine headaches

A

Migraine headache is described as an intense, throbbing or pounding pain that involves one temple. The pain usually is unilateral (on one side of the head), although it can be bilateral.

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5
Q

pain location of cluster headaches

A

Cluster headache pain is focused in and around one eye and is often described as sharp, penetrating, or burning.

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6
Q

tension headaches

A

most common
bilateral location : pressing/tightening quality
mild or moderate
episodic or chronic
bilateral frontal-occipital : contant, dull, bandlike

NO warning symptoms, NO nausea or vomiting, NO trouble with physical activity
SENSITIVITY to light or sound

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7
Q

migraine headaches

A

unilateral throbbing pain, premonitory symptoms or triggers, onset between 20-30, affects more females than males, state of neuronal hyper-excitability in occipital cortex of the cerebral cortex, may or may not have known precipitating factors

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8
Q

what medical issues are migraine headaches associated with

A

seizure disorders, ischemic stroke, asthma, depression, anxiety, myocardial infarction, Raynaud’s syndrome, and irritable bowel syndrome.

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9
Q

precipitating factors for migraine headaches

A

include foods, menstruation, head trauma, physical exertion, fatigue, stress, missed meals, weather, and drugs. Food triggers include chocolate, cheese, oranges, tomatoes, onions, monosodium glutamate, aspartame, and alcohol (particularly red wine).

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10
Q

risk factors for migraine headaches

A

Family history
Low level of education
Low socioeconomic status
High workload
Frequent tension-type headaches

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11
Q

clinical manifestations of migraine headaches

A

neurologic, psychologic, or other premonitory manifestations
aura
steady, throbbing
synchronous with pulse
may last 4-72 hrs
vary in severity

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12
Q

cluster headaches

A

generally occur at same time of day or night
onset between 20-45
men more affected

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13
Q

what is the most common type of headache

A

tension

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14
Q

what is the most severe primary headache

A

cluster

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15
Q

triggers for cluster headaches

A

alcohol
strong odor
weather changes

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16
Q

what part of the brain is affected in cluster headaches

A

ophthalmic branch of trigeminal nerve
hypothalmus
irregularities with melatonin and cortisol

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17
Q

manifestations of cluster headaches

A

Sharp, stabbing, intense pain lasts minutes to 3 hours
May occur every other day and as often as 8x/day
Can occur in cycles with remission periods in between
Pain is generally located around the eye, radiating to the temple, forehead, cheek, nose, or gums
Swelling around the eye, lacrimation (tearing), facial flushing or pallor, nasal congestion, and miosis (constriction of the pupil).
often agitated and restless, unable to sit still or relax
aura similar to migraine may occur in 14% of patients up to 60 minutes before an attack.
Cluster headaches can occur every other day and as often as eight times a day.
Because cluster periods often occur seasonally, headaches may be mistaken for symptoms of allergies.

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18
Q

health history for assessment of headaches

A

Seizures, cancer, stroke, trauma, asthma or allergies, mental illness, stress, menstruation, exercise, food, bright lights, noxious stimuli
Medications
Surgery and other treatments

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19
Q

objective data for assessment of headaches

A

Anxiety or apprehension
Diaphoresis, pallor, unilateral flushing with cheek edema, conjunctivitis

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20
Q

details about the headache in assessment

A

Location, Type of pain
Onset, Frequency, Duration, time of day
Relation to outside events

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21
Q

if no systemic underlying disease is the cause of the headache, what guides therapy

A

the type of headaches

types of therapies: Drugs, medications, yoga, biofeedback, cognitive-behavioral therapy, and relaxation training

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22
Q

symptomatic drug therapy for tension headaches

A

Mild-moderate headache treated with aspirin, acetaminophen, or an NSAID alone or in combination with a sedative, muscle relaxant, or tranquilizer

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23
Q

preventative drug therapy for tension headaches

A

Tricyclic antidepressants
Antiseizure medications

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24
Q

preventative drug therapy for migraine headaches

A

Antiseizure drugs - GABA, topiramate
Botox
SSRIs

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25
Q

symptomatic drug therapy for migraine headachces

A

Mild to moderate headache can obtain relief with NSAID, aspirin, or caffeine-containing combination analgesics

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26
Q

skin patch for migraine headaches

A

zecuity

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27
Q

first line therapy for moderate to severe headaches

A

triptans
- vasoconstrict : caution to pt with heart disease or stroke

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28
Q

preventative drug therapy for cluster headaches

A

High-dose verapamil

Varied other options : lithium, ergotamine, antiseizure drugs (e.g., topiramate), and melatonin.

Invasive nerve blocks, deep brain stimulation, and ablative neurosurgical procedures have been used for refractory cluster headaches

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29
Q

symptomatic drug therapy for cluster headaches

A

triptans (dont give to pt with vascular risk factors)
100% oxygen at 6-8 L/min for 10 min non rebreather (repeat after 5 min rest)

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30
Q

headache nursing implementation

A

Teach patient about preventive treatment
Dietary counseling for food triggers
Avoid smoking and other environmental triggers
An inability to cope with daily stresses can cause headaches
Daily exercise, relaxation periods, and socializing help reduce recurrence and should be encouraged
Suggest alternative pain management such as relaxation, meditation, yoga, and self-hypnosis
Encourage a quiet, dim environment
Massage and heat packs can help with tension-type
Patient should make a written note of medications to prevent accidental overdose

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31
Q

headache provoking foods

A

chocolate, cheese, oranges, tomatoes, onions, monosodium glutamate, aspartame, alcohol, excessive caffeine, fermented or marinated foods

32
Q

what med is prescribed to be taken before planes take off to decrease likelihood of attacks that occur at high altitudes

A

ergotamine

33
Q

dementia causes dysfunction of loss of :

A

Memory
Orientation
Attention
Language
Judgment
Reasoning
Behavior or personality

34
Q

what factors does dementia interrupt

A

Work
Social responsibilities
Family responsibilities
Ability to perform ADLs

35
Q

what are the most common causes of dementia

A

neurodegenerative conditions (alzheimers)
vascular conditions

36
Q

vascular dementia results from what

A

ischemic or hemorrhagic brain lesions caused by cardiovascular disease

37
Q

mixed dementia

A

2 or more types of dementia at the same time
usually alzheimers with vascular dementia

38
Q

lewy bodies dementia

A

neurodegenerative dementia
presence of lewy bodies in brainstem and cortex (intraneural cytoplasmic inclusions)
includes features of parkinsons

39
Q

normal pressure hydrocephalus

A

uncommon
caused by obstruction of CSF flow : meningitis, encephalitis, head injury (manifestations = dementia, urinary incontinence, difficulty walking)
treatable if diagnosed early

40
Q

what ethnicity is alzhemiers most common in

A

african americans and hispanics
lower socioeconomic status / poor access to healthcare

41
Q

alzheimers disease

A

chronic, progressive, neurodegenerative brain disease
cannot be prevented, cured, or slowed

42
Q

patho of alzheimers

A

Abnormal amounts of β-amyloid are cleaved from the amyloid precursor protein (APP) and released into the circulation. The β-amyloid fragments come together in clumps to form plaques that attach to the neuron. Microglia react to the plaque, and an inflammatory response results.

43
Q

early warning signs of alzheimers

A

Memory loss that affects job skills
Difficulty performing familiar tasks
Problems with language
Disorientation to time and place
Poor or ↓ judgment
Problems with abstract thinking
Misplacing things
Changes in mood or behavior
Changes in personality
Loss of initiative

44
Q

diagnostics of alzheimers

A

patient evaluation :
Complete health history
Physical examination
Neurologic assessment
Mental status assessment
labs
imaging : CT, MRI, PET

45
Q

examples of neuropsychologic testing for alzheimers

A

Mini-Cog
Mini-Mental State Examination (MMSE)
– Used to determine a baseline from which to evaluate change over time

46
Q

mini mental state vs mini cog

A

mini cog : draw a clock
MMSE : oriented to time, registration, naming, reading

47
Q

confusion assessment method

A

BEST PRACTICE FOR ASSESSING CONFUSION IN OLDER ADULTS
Assess onset, attention level, type of thinking, LOC, disorientation, memory impairment, perceptual disturbance, psychomotor agitation, psychomotor retardation, and the sleep-wake cyle

48
Q

subjective and objective data for alzheimers assessment

A

subjective =
Past health history
Medications
Health perception–health management
Nutritional-metabolic
Elimination (incontinence)
Activity-exercise
Sleep–rest pattern
Cognitive-perceptual

objective =
Disheveled appearance
Neurologic
Early, middle, late changes

49
Q

gradual vs abrupt onset of dementia

A

gradual and progressive = neurologic degeneration

abrupt = vascular dementia

50
Q

what can you do to treat and prevent vascular dementia

A

treat risk factors :
Hypertension, diabetes, smoking, hypercholesterolemia, cardiac dysrhythmias

51
Q

care for alzheimers

A

NO CURE
care is aimed at :
Delaying onset of symptoms
Controlling undesirable behavioral manifestations
Providing support for family caregiver

52
Q

drug therapy for alzheimers

A

memantine - protects nerve cells against excess amounts of glutamate
SSRI’s
antidepressants - trazodone may help with problems with sleep
antipsychotic drugs - managing behavioral problems

53
Q

health promotion for alzheimers

A

Avoid harmful substances
Challenge your mind
Exercise regularly
Stay socially active
Avoid trauma to the brain
Take care of mental health
Treat diabetes
Take care of your heart
Get enough sleep
Get the right fuel

54
Q

patient responses to alzheimers diagnosis

A

Depression
Denial
Anxiety and fear
Withdrawal
Feelings of loss

55
Q

when there is an inability to communicate symptoms who is in charge of communicating for the pt

A

caregiver or health care professionals

56
Q

behavioral problems with AD

A

Repetitiveness
Delusions
Hallucinations
Agitation
Aggression

Altered sleep patterns
Wandering
Hoarding
Resisting care

57
Q

interventions of behavioral problems

A

assess physical status before environment
remove stimulus
reassure about safety
rely on mood and behavior rather than verbal communication

58
Q

nursing strategies for behavioral problems

A

redirection
distraction
reassurance

59
Q

sundowning

A

Specific type of agitation
Patient becomes more confused and agitated in late afternoon or evening
May be due to disruption of circadian rhythms

60
Q

nursing interventions for sundowning

A

Create a quiet, calm environment
Maximize exposure to daylight
Evaluate medications
Limit naps and caffeine
Consult health care provider on drug therapy

61
Q

safety risks with alzheimers

A

Injury from falls
Ingesting dangerous substances
Wandering
Injury to others and self with sharps
Burns
Inability to respond to crisis

62
Q

manifestations of infection

A

change in behavior, fever, cough (pneumonia), pain, urination (bladder problems)

63
Q

caregiver support

A

Work with the caregiver to assess stressors and to identify coping strategies to reduce the burden of caregiving.
Determining what the caregiver views as most disruptive or distressful can help to establish priorities for care.

64
Q

expected outcomes for alzheimers disease

A

Functions at highest level of cognitive ability
Experiences no injury
Remains in restricted area during ambulation and activity
Performs basic personal care activities of daily living including

65
Q

delirium

A

state of temporary but acute mental confusion
often preventable or reversible
can be life threatening

66
Q

contributing factors to delirium

A

Impairment of cerebral oxidative metabolism
Cholinergic deficiency
Excess release of dopamine
Change in serotonin

Stress, surgery, sleep deprivation

COGNITIVE
Dementia
Cognitive impairment
Depression
History of delirium
ENVIRONMENT
Admission to ICU
Use of physical restraints
Pain (especially untreated)
Emotional stress
Prolonged sleep deprivation
FUNCTION
Functional dependence
Immobility
History of falls
SENSORY
Sensory deprivation
Sensory overload
Visual or hearing impairment

67
Q

DELIRIUM MNEUMONIC CAUSES

A

Dementia, dehydration
Electrolyte imbalances, emotional stress
Lung, liver, heart, kidney, brain
Infection, ICU
Rx Drugs
Injury, immobility
Untreated pain, unfamiliar environment
Metabolic disorders

68
Q

when does delirium develop

A

over 2-3 day period
can develop within hours

69
Q

early manifestations of delirium

A

Inability to concentrate
Disorganized thinking
Irritability
Insomnia
Loss of appetite
Restlessness
Confusion

70
Q

later manifestations of delirium

A

Agitation
Misperception
Misinterpretation
Hallucinations

71
Q

how long do delirium symptoms last

A

1-7 days
can be years
can never recover

72
Q

distinctions of delirium rather than dementia

A

Sudden cognitive impairment
Disorientation
Clouded sensorium

73
Q

diagnostic studies for delirium

A

Medical history
Psychologic history
Physical examination
Careful attention to medications
Cognitive measures
Confusion Assessment Method (CAM)

74
Q

lab tests to look at for delirium

A

Serum electrolytes
Blood urea nitrogen level
Creatinine level
Complete blood count (CBC)
Drug and alcohol levels

Electrocardiogram (ECG)
Urinalysis
Liver and thyroid function tests
Oxygen saturation level
Lumbar puncture

75
Q

role as nurse for delirium

A

Prevention
Early recognition
Treatment
Focus on eliminating precipitating factors
Protect patient from harm
Encourage family members to stay at bedside
If delirium is secondary to infection, antibiotic therapy is started
Reorientation and behavioral interventions
Create a calm and safe environment
Provide reassurance
Pay attention to environmental stimuli

76
Q

Other risks for people with delirium

A

Immobility
Skin breakdwon

77
Q

Drug therapy for delirium

A

reserved for those with severe agitation
puts patients more at risk for falls and injury

Dexmedetomidine (Precedex) for sedation
Neuroleptics
Haloperidol (Haldol)
Risperidone (Risperdal)
Olanzapine (Zyprexa)
Quetiapine (Seroquel)
Short acting benzos