TL block 7 Flashcards
CAM-ICU questions
- acute change in mental status or fluctuating?
- pt inattentive or easily distracted?
- RASS other than zero?
- disorganized thinking?
trigger point myofascial pain
-limited ROM
-muscle spasm upon palpation
-radiation of pain to somewhere else w/ palpation
-palpation can cause autonomic symp
Concerns for ACLS in pregnant patients
if >20 weeks
-L displacement of uterus w/ compressions
-Same energy charge for defib
-delivery of fetus in 4 minutes if no ROSC
-if Mg running, stop and give calcium
-have LMAs available incase ETT is too difficult
Common SE w/ interscalene blocks
-ipsilateral Hornor syndrome (stellate ganglion blocked) -> ptosis, miosis, anhidrosis
-ipsilateral phrenic n blocked
Asthma DLCO
Increased
-inc lung volumes
exercise DLCO
increase! b/c cardiac output is increased -> more flow through pulm vessels -> more Hg in lungs -> Inc DLCO
L to R cardiac shunt DLCO
increased
-more blood going to lungs
What determines DLCO?
-blood flow (cardiac output)
-Hg conc
-lung parychema (fibrosis)
Zenker’s diverticulum
CI to TEE
Absolute contraindications to TEE
-Zenker diverticulum
-Active GI bleed
-esophageal tumor
-recent esophageal surgery
-Mallory Weiss tear
-Scleroderma
-perforated esophagus
-esophageal rings/strictures/webs
-esophageal trauma
-recent variceal bleeding
-esophagectomy
RF for MR following acute MI
-adv age
-inferior/posterior MI
-extending infarct
-hx of prior MI
-multiple vessel CAD
-recurrent ischemia
Setting of power failure, what works and what doesn’t?
Works: O2 delivery, manual PPV, if vaporizers variable-bypass they will work
doesn’t: monitoring, all electrical, or if cassette vaporizers
Anion Gap Equation
Na - (Cl + bicarb)
Causes of non-anion gap metabolic acidosis
- Giving Cl -> excessive NS, TPN
- GI/renal losses of bicarb: renal tubular acidosis, acetazolamide, diarrhea, high ostomy output
- Dec acid secretion -> hyperaldo, renal tubular acidosis
surgical blood loss replacement in neonates
1:1 colloid (blood, albumin)
1:1.5 isotonic crystalloid
when to restart subq 5k BID heparin after catheter?
immediately
how long to hold heparin 5k BID before catheter removal?
4-6 hours
how long to restart heparin 5k BID after catheter removal?
immediately
heparin 7.5k-10k BID how long to hold before epidural?
12 hours AND normal coag status
heparin 7.5k-10k BID when to restart after neuraxial?
avoid if catheter in place
heparin 7.5k-10k BID when to restart after catheter removal?
immediately
Therapeutic subq heparin > 20k per day: when to hold before neuraxial?
24 hours AND normal coags
Therapeutic subq heparin > 20k per day: when to restart once epidural placed?
avoid if catheter in place
Therapeutic subq heparin > 20k per day: when to restart when catheter removed?
immediately
IV UFH: when to hold prior to neuraxial?
4-6 hours AND normal coags
IV UFH: when to restart once catheter in place?
1 hour
IV UFH: how long to hold before catheter removal?
4-6 hours AND normal coags
IV UFH: how long after catheter removal can we restart?
1 hour
LMWH ppx daily how long to hold before neuraxial?
12 hours
LMWH ppx daily how long to wait after catheter placed to restart?
12 hours
LMWH ppx daily how long to hold before catheter removal?
12 hours
LMWH ppx daily, how long to wait to restart after catheter removal?
4 hours
and NO no earlier than 12 hours after catheter placement
LMWH ppx BID dosing:how long to hold before neuraxial?
12 hours
LMWH ppx BID dosing: how long to wait to restart after catheter in place?
Avoid while catheter in place
LMWH ppx BID dosing: how long after catheter removed to wait until restarting?
4 hours AND no earlier than 12 hours after initial catheter placement
Therapeutic LMWH dosing: how long to hold before catheter placement?
24 hours
Therapeutic LMWH dosing: how long to wait to restart after catheter placed?
avoid if catheter in place
Therapeutic LMWH dosing: how long to wait to restart after catheter removed?
4 hours AND no earlier than 12 hours after intial placement
Complications of refeeding syndrome
-weakness (incl resp weakness)
-myocardial depression
-rhabdo
-hemolytic anemia
-arrhythmias
-neuro disturbances
-impaired O2 delivery
-immunosuppression
Situations that worsen refeeding syndrome:
-hyperventilation: low CO2 causes intracellular shift of phosphate
-inc renal elimination of phos: hyperPTH, loop diuretics
-dec phos absorption: Vit D def
Normotensive pt, most effective way to reduce inc ICP
propofol bolus
post-craniotomy 3d ago, now new surgery nitrous during case, post op not following commands or waking up, dx? next steps?
Dx: tension pneumocephalus
next step: CT scan
tx: neurosurg air loculi aspiration
if HIPPA breached, next steps?
Pt must be notified up to 60 days after date of discovery
if breach of pt information affected more than 500 individuals next steps:
-notify individuals
-US Dept of Health & Human services notified
-prominent media outlet in the sate or jurisdiction where breach happened
Allodynia
perception of ordinarily nonnoxious stimulus as painful
(touch of clothes as painful)
Anesthesia dolorosa
pain in an area that lacks sensation
(after trigeminal neurolytic block)
Difference between mixed venous O2 saturation and central venous O2 saturation
mixed venous: taken from pulm artery catheter
central venous: taken from central line
-central venous is 2-5% less than mixed venous b/c high extraction of head and upper extremities
Direct inhibitors of hypoxic pulmonary vasoconstriction
-hypocarbia
-infxn
-vasodilators (NG, nicaridipine, nitroprusside)
-metabolic alkalosis
-volatiles at greater than 1 MAC
Microcirculation and cardiogenic shock
-due to pump failure -> venous congestion
-to compensate we get arterial vasoconstriction
-b/w capillaries and organ interstitium -> favor of fluid movement into intersititum and then capillaries become leaky and also favor fluid movement into interstitium -> but reversible
LWhat is octreotide used for?
Acromegaly: suppresses GH
Where is lumbar sympathetic ganglia located?
L1-L5
What symp n block causes diarrhea?
Celiac plexus (T5-12)
-innervation to intraabd organs
Tetralogy of Fallot
-VSD
-overriding aorta
-RVOT obstruction
-Right ventricular hypertrophy
Tet spells steps!
tet spell: shifted to R->L shunting of blood
1st step: give O2 and bend legs, squat (inc SVR to promote BF to pulm, O2 pulm vasodilation)
2nd step: IVF and narcotic like morphine (inc preload, dec HR, dec PVR, dec RR)
3rd step: beta blocker (dec HR, improve preload, relax RVOT obstruction)
4th step: phenylephrine (inc SVR, promote blood flow)
5th: ECMO, emergency surgical repair
treatment if pt w/ tetralogy of fallot starts to experience heart failure
digoxin and loop diuretics
-goal to maintain SVR
Def of wide complex QRS
> 0.09 seconds
energy for synchronized cardioversion in peds
0.5-1 J/kg
unstable wide complex tachycardia
synchronized cardioversion
sinus tachycardia v SVT in peds
sinus: p waves
-HR < 220 for infants
-<180 for children
-<150 for adults
SVT: no p waves
HR >220 infants, >180 peds
posterior fossa surgery, acute HTN and bradycardia w/ retraction why?
Brainstem compression -> cushings triad
Once pt gets diabetic neuropathy, what’s next?
depression of reflexes -> motor weakness
-autonomic neuropathy -> sluggish pupillary light reflexes, gustatory hidrosis (lots of sweating at head and upper torso after meals)
-resting tachycardia
superior laryngeal nerve
innervated cricothyroid muscle (VC adductor)
-so if RLN damage -> VC adduction
Meds to avoid w/ myotonic dystrophy
-neostigmine
-succinylcholine
-K containing solutions
**shivering will also cause myotonias!
Lab findings primary hyperparathyroidism
-hyperCa
-hypoPhos
-non AG metabolic acidosis (dec bicarb reabsorption)
-normal to high 24hr urinary calcium
post parathyroidectomy, weak voice mild neck discomfort in PACU, next morning voice sounds different and weakens after speaking for a long time
superior laryngeal n injury
Most common n injured in parathyroid/thyroidectomy
superior laryngeal n
SIADH dx criteria
hypoNa w/ urine Na > 20
-inc urine osm while dec serum osm
-euvolemic/hypervolemia
How to tell SIADH apart from cerebral salt wasting
volume status!
SIADH: euvolemic
cerebral salt wasting: hypovolemic
Diabetes insipidus labs
-either ADH not produced or kidneys not responsive
-hyperNa (>145), hyperosm blood, (>305) urine low sodium, urine low osm (<300)
-usually euvolemic, can easily become hypovolemic
normal urine osmolality
500-850
normal urine specific gravity
1.005-1.030
infant blood transfusion, when to start?
Hct <20 if hemostasis achieved
Hct < 25 if additional bleeding still expected
-initial volume of 10-15 cc/kg
maximum allowable blood loss eq
MABL = est blood volume * ([starting Hct - target Hct] / starting Hct)
how much does 1u PRBCs raise Hg or Hct in adults?
Hg inc 1
Hct inc 2-3%
Peds 10-15 cc/kg pRBC raise hg or hct in adults?
hg inc 1
Hct inc 2-3%
How to tell MH apart from thyroid storm
Hypercapnia
inc CK
lactic acidosis
Thyroid storm treatment
propthiouracil and supportive
lab values in Addison’s dx
primary adrenal insuff
-hypoNa, hyperK, metabolic acidosis, hyperCl, hypoglycemia, hyperCa
(hypercalcemia due to dec in GFR 2/2 hypovolemia with an increase in Ca release from the bone)
Prazosin MOA
selective alpha 1 blocker
why inc HR w/ phenoxybenzamine
non-selective alpha blockade -> loss of alpha 2 presynaptic inhibition of norepinephrine release