Thoracolumbar Spine V- Persistent IDD Flashcards

1
Q

Persistent IDD is aka as….?

A

Degenerative Disc Disease (DDD)

Age-related disc changes although NOT always due to age

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2
Q

With chronic or persistent IDD, disc changes are due to numerous ________ and allow herniations (_______ _______) to GRADUALLY develop over time

A

variables; nuclear migration

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3
Q

Persistent IDD is most common in what region?

A

Lumbar

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4
Q

With persistent IDD, only ___-____% are symptomatic

A

1-3

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5
Q

Persistent IDD is the source of pain in < ___% of LBP

A

5

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6
Q

Persistent IDD is most common in ___-____ yr. olds

A

30-50

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7
Q

95% of persistent IDD cases occurs at which vertebral levels?

A

L4-S1

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8
Q

Persistent IDD ____ Acute IDD

A. >
B. <

A

A.

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9
Q

Is persistent IDD or acute IDD more prevalent?

A

Persistent IDD

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10
Q

Etiology of persistent IDD:

Acute ____

Mixed findings with ____

______ strength

_______ lifestyle

A

IDD
age
Lower
Sedentary

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11
Q

With persistent IDD, why could lower strength be the cause?

A

Bc the local muscles become weak and then the global muscles have to take over and become overworked from “picking up the slack”

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12
Q

With persistent IDD, why could a sedentary lifestyle be the cause?

A

“You don’t use it, you lose it” They are not moving enough and too little stress on the spine, is just as bad as doing nothing

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13
Q

What are two additional etiologies of persistent IDD?

A

Heavier occupational lifting

Smoking

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14
Q

With the etiology of persistent IDD, genetics typically are associated with:

_____ IDD and with ____-______ _______changes in the cervical region

A

Lumbar; age-related disc

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15
Q

With the etiology of persistent IDD involving genetics, ___-____% of inheritance can be modified by diet and lifestyle

A

65-85

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16
Q

With persistent IDD, it is NOT caused by….

_______ loading/______ activities

________ driving

A

routine/ physical

prolonged

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17
Q

The pathogenesis of persistent IDD is ________ and has what kind of inflammation?

A

gradual; persistent

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18
Q

With persistent inflammation, there is an in-growth of _______ fibers from _____IDD healing and can lead to what kind of pain?

A

nociceptive
ACUTE
nociplastic

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19
Q

With persistent inflammation, it brings excessive and destructive proteins ( _______ ) and what likely enters the disc?

A

amyloidosis; low-grade infection

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20
Q

Persistent IDD pathogenesis (gradual):

____ GAGs and MORE ______ and dehydrated nucleus (more or less/ synovial or fibrotic)

MORE _____ disc that kills disc cells and limits ______ (base or acidic/inflammation or proliferation)

_______ disorganization (annular or endplate)

_______ and loss of cartilage at ____ _____ (thickening or thinning/ nucleus pulposus or end plates)

________ inflammation and fatty deposits in vertebra ( ______ )- (decreased or increased/ fibrotic or modic)

NOW _______ herniations and nuclear migration GRADUALLY develop (per Miller)
(acute or persistent)

A

Less; fibrotic

acidic; proliferation

annular

Thinning; end plates

Increased; modic

persistent

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21
Q

Protrusion is also called a _____

22
Q

With a protrusion (bulge), the nucleus ______ but remains contained in the ____

A

migrates; anulus

23
Q

What is the MOST common herniation?

A

Protrusion (bulge)

24
Q

With _______ the nucleus migrates through the outer annulus

A. protrusion
B. extrusion
C. free sequestration

25
_____ ______ is when the nucleus migrates and breaks away from the annulus
Free Sequestration
26
With persistent IDD, what likely develops where the nucleus migrates into the vertebral body?
Schmorl's Nodes
27
With persistent IDD, there can be ________ and cause initial changes related to loss of disc height and integrity
narrowing
28
With persistent IDD, _______ may develop and lead to ??
instability, hypermobility
29
With joint hypermobility relating to persistent IDD, it is noted during _____ and _____ planes but not _____ plane
sgaittal; frontal; transverse
30
With persistent IDD and joint narrowing.... it contributes to initial _______ has a _____ load bearing on facets A.instability; greater B. hypomobility; greater
instability; greater
31
If the neural foramen starts to narrow, what can develop?
stenosis
32
With persistent IDD, there are greater ______ It can lessen prior instability due to associated ______ of a joint
ARJC stiffening
33
With persistent IDD S&S _____ change allows tissues to adapt w/o symptoms for some time indicated by lack of symptoms with image changes in ___/____ of individuals
Slow; 2/3
34
With persistent IDD, symptoms may present as..?
acute
35
With persistent IDD, PT Rx: Mechanical Diagnosis and Therapy _____-_____ benefit Better for P! and NOT _______ NOT Superior to: Education ______ therapy and motion _____ _______ exercises
Short-term function Manual; exercises Stabilization
36
With persistent IDD, PT Rx: You need to consider ________ driver of symptoms from the development of other conditions even if imaging shows _____ _______
primary; disc changes
37
With acute and persistent internal disc derangement, there is a negative outcome predictor with ________
peripheralization
38
Peripheralization is the opposite of _______
centralization
39
With acute and persistent IDD, peripheralization has a significant association with: Mental ______/depression ____ behaviors _______ - coversion of anxiety into bodily symptoms fear of ______ ___-______ signs
distress pain Somastication work non-organic
40
With acute and persistent IDD, if symptoms present > six months prior to any treatment this could lead to what kind of outcome?
worse
41
With acute and persistent IDD MD Rx: There are conflicting and unclear benefits with these medications: ______, ________ ______, AND _________
NSAIDS, muscle relaxants, acetaminophen
42
With acute and persistent IDD MD Rx: patients may be prescribed a ______ dose pack for a large inflammatory response
steroid
43
With acute and persistent IDD MD Rx: A _______ injection is short-term but NOT long-term relief or functional changes
epidural
44
With acute and persistent IDD MD Rx: _______ treatment benefits the potential infection source
Antibiotic
45
With acute and persistent IDD MD Rx involving surgery: Waiting an avg. of _____ months of sx does NOT minimize the benefits of sx
4 1/2
46
With acute and persistent IDD MD Rx involving surgery: Some studies demonstrated _____ and improved benefit with sx vs. PT; particularly with severe _____ IDD
earlier; acute
47
With acute and persistent IDD MD Rx involving surgery: It is a _____ process and has the same overall outcomes without sx after 2 years
slower
48
With acute and persistent IDD MD Rx: What is an example of a spinal decompression surgery?
laminectomy or partial discetomy
49
With acute and persistent IDD MD Rx: What are some indications for surgery?
persistent and or worsening radiculopathy when symptoms are unresponsive to non-surgical treatments
50
With persistent IDD involving sx for hypermobility/instability present: ______ fusion has... NO difference vs. PT in long-term outcomes with pain, health status, satisfaction, or disability NOT additive to laminectomy or discectomy May lead to adjacent joint hypermobility/instability
Lumbar
51
With persistent IDD involving sx and TDR: Better load distribution across _______ Safe and effective treatment MORE than ____ years post-op At ____ years follow up, no differences compared to PT alone without radiculopathy for in return to work, life satisfaction, fear avoidance behavior, drug use, back performance
segments 5 2
52