Achilles Tendinopathy Flashcards
Prevalence
Most frequently with ______/______
overuse/injury
Most common in…
- _______/_______ activities
- _________ > competition
- ___-____ year olds
10-20% of _______
recreational/competitive
training
30; 50
runners
Risk Factors and Etiology
- Limited calf ________
- Calf ________ could lead to under supply/overuse
- Biological _____ and family _____
- Excessive ________
flexibility
weakness
males; hx
pronation
Overuse may occcur with _____ ______ control, which includes ______ _______ deficits and ______ deficits
impaired LQ
hip neuromuscular
balance
Excessive Pronation is a earlier extended and or excessive combination of ____/_____/_____
DF; ABD; EV
Excessive pronation can become excessive if there is ______/_______ present
More commonly:
_______ or _________
Impaired ______ _____- top down influence
Less commonly: ______ or medial ______ hypermobility
hypermobility; instability
tibfib; talocrural
LQ control
subtalar; knee
Excessive pronation may become excessive if ______ ________ hypombility is present
Ex:
Limited ______ _______ may lead to midfoot and forefoot excessively EV and ABD
Limited _____ ______ may lead to excessive ankle DF
adjacent joint
talocrural DF
knee EXT
Risk Factors and Etilology
Abnormal _______ structure and prior _______
______ age (more plastic)
Also consider… _____ errors, _______ factors, and improper _________
________- overuse
Systemic dz with persistent _______ and poor _______ _______
tendon; injury
older
training; environmental; shoes
obesity
inflammation; blood supply
Pathomechanics
Repetitive ______ with _______ from limited ____ and or excessive _______
Collagen fibril ________/_________ and fibroblast death from altered fluid movement leads to _______, ________ nitric acid with persistent ___________
lengthening; compression
DF; EV
thinning; disorganization
overheating; increased; inflammation
Pathomechanics
Thickened but _______ tendon from:
increase of ____-______ matrix
fat _________
________ force transfer
Impaired _______ _________
weaker
non-collagen
deposition
ineffective
motor control
Symptoms
_______ onset that limits ______ activity
Localized _____ and _______
Particurarly after __________
Lessens with _____ bout of activity and increases with _____ to ______ activity
gradual; WB
pain; stiffness
inactivity
mild; moderate; severe
Signs
Observation- Achilles _______
Possibly impaired ____ _____ and or excessive _______
ROM- possible P! and limitation with ______
Resisted/MMT: Possible pain with _____, may be _____
possibly ____/______ weakness
Accessory Motion- possible _______ hypomobility for _______
Palpation- TTP 2-6 cm proximal to ______ (higher seas) area of less _____ ______
More _____ achilles P! indicates plantaris involved
Achilles _______ (high spec)
thickening
LQ control
pronation
DF
PF; weak
hip; knee
talar; DF
insertion; blood supply
medial
crepitus
Special Tests
- _____ ____ (high spec)
- _____ _____ Test (high spec)
- ________ Tests
- _______ _______ _______
- M. length- shortened ______
Arc Sign
Royal London
Stability
Single Heel Raise
Gastrocs
PT Rx
_______ NOT indicated
_______ stress is BEST within appropiate P! levels
______ management
______ wear
Timeline: ____-_____ wks
Prognosis: ______%
POLICED
Rest
Optimal
Weight
Shoe
8; 12
80
PT Rx
Modalities
LASER: ___________
Ionto: dexamethasone helpful for ____ function
Shockwave Therapy: support for P! relief with ADLs when added to ____ weeks of exercise
NO indication on ______ changes or return to sport
contradictory
P!
4
structure