Systemic risk factors part 2 Flashcards

1
Q

what does the features of chronic periodontitis include in the old 1999 classification

A

most prevalent in adults but can occur in children and teenagers
- slow to moderate progression + exacerbations
- plaque aetiology
- destruction consistent with local factors
-subgingival calculus frequent finding
-modifying factors include : local and systemic factors, smoking , stress, poorly controlled diabetes
-treat in usual 3 phases - no systemic antibiotics

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2
Q

what common features are present In aggressive periodontitis

A
  • rapid attachment loss and bone destruction
  • familial aggregation
  • localised form
  • generalised form
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3
Q

what are the clinical and radiographic features found in aggressive periodontitis

A
  • localised first molar/ incisor presentation
  • CAL - clinical attachment loss
  • deep pockets in association with CAL
  • alveolar bone loss
  • angular defects incisors
  • arc shaped bone loss first molars
  • often symmetrical distribution right/left
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4
Q

what are the secondary features of aggressive periodontitis

A
  • amounts of microbial deposits inconsistent with severity of periodontal tissue destruction
  • elevated proportions of aggregatibacter actinmycetmcomitons
  • phagocyte abnormalities
  • hyper responsive macrophage phenotype, including elevated levels of PGE and IL-1Beta
  • progression of attachment loss and bone loss may be self arresting
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5
Q

what are features of localised aggressive periodontitis

A
  • specific features
  • cicumpubertal onset
  • robust serum antibody response to infecting agent
  • localised first molar/ incisor presentation
  • interporximal attachment loss CAL on at least 2 permanent teeth , one of which is a first molar and involving no more than two teeth other than first molars/ incisors
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6
Q

what is the difference between the 1999 and the 2018 class-action of periodontal disease

A
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7
Q

which terms do not exist in the 2018 classification pf periodontal disease

A
  • chronic periodontitis
  • aggressive periodontitis
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8
Q

define periodontitis following the EFP S3 clinical guidelines

A

Periodontitis is defined by the loss of periodontal tissue support, which is commonly;y assessed by radiographic bone loss or interproximal loss of clinical attachment measured by probing

Other meaningful description of periodontitis include the number and proportions of teeth with probing pocket depth over certain thresholds , the number of teeth lost due to periodontitis, the. number of teeth with intrabony lesions and the number of teeth with furcation lesions

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9
Q

what 3 dimensions should a periodontitis diagnosis encompass

A
  • definition of a periodontitis case based on detectable CAL loss at tow non-adjacent teeth
  • Identification of the form of periodontitis- necrotising periodontitis, periodontitis as a manifestation systemic disease or periodontitis
  • description of the presentation and aggressiveness of the disease by stage and grade
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10
Q

what are the notes on diagnosis of localised aggresive periodontitis

A
  • history and exam - correct diagnosis
  • BPE for initial screening should flag warning
  • Per indices with periodontal probe - PCP10
  • radiographs where indicated/ justified
  • early detection important
  • start apropriotate therapy, improve prognosis
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11
Q

how do we detect aggressive periodontitis for under 18s

A
  • simplified BPE
  • index teeth
  • BPE codes 0,1,2 ages 7-11 years
  • full range BPE codes 0,1,2,3,4*
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12
Q

what are specific feature of generalised aggressive periodontitis

A
  • usually affects age <30 yrs, may be older
  • poor serum antibody response to infecting agents
  • pronounced episodic nature of extraction of attachment and alveolar bone
  • Generalised interproximal attachment loss CAL affecting at least 3 teeth other than first molars and incisors
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13
Q

what are the principles of management of aggressive periodontitis

A
  • early diagnosis is essential- screening
  • establish correct diagnosis - affects therapy
  • is there an underlying modifying factor or systemic factor that needs managing
  • poorly controlled diabetes mellitus
  • refferal to a specialist should be considered by the GDP
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13
Q

what are the principles of managemtns of aggressive periodontitis.

A

management is directed to
- suppression of infecting organisms
- providing environment conducive to long term maintenance
Therapy in usual 3 phases
- initial cause related
- corrective
-supportive

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14
Q

what are the differences in managements of chronic and aggressive periodontitis

A
  • Initial cause related therapy (GDP can do both)
  • Corrective therapy ( consider specialist for aggressive peirodontitis/ stage 3 or 4 Grade C in younger individuals or disproportionate bone loss to the amount of plaque and calculus)
  • Supportive therapy (GDP can do both)
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14
Q

what are the corrective therapy for aggressive periodontitis ; consider use of adjunctive systemic antibtitocs

A
  • microbial aspects
    initial therapy should decrease microbial load
  • corrective therapy, need to disrupts biofilm of root surfaces in conjunction with adjunctive systemic antibiotics
  • workable regime; 2 visits for half mouth RSD one week apart, debate about starting antibiotics, where indicated at first visit or prescribing antibiotics at end of 2nd visit
    -LDI protocol; prescribe adjunctive antibiotics at end of 2nd visit
  • Microbial sampling in theory can be useful beforehand to identify periodontal pathogens but not routinely done
15
Q

what is the prognosis of aggressive periodontitis

A
  • improves with
    early correct diagnosis ; appropriate therapy
    frequent recall/monitoring after therapy
  • elimination of associated microorganism is requirement of success
  • extractions may be indicated
  • burn out MAY occur without therapy
    unpredicable - therefore treat
16
Q

what is periodontitis

A
  • chronic inflmamaotry disease associated with dysbitoic plaque biofilms and characterised by progressive destruction of tooth supporting apparatus
  • primary features; loss of periodontal tissue support manifest through clinical attachment loss; radiographically assessed bone loss ; presence of peeriodntla pocketing; gingival bleeding
17
Q

Why do we use systemic antibiotics in necrotising periodontal disease?

A

Necrotzing ulcerative gingivitis is a mixed bacterial infection caused by a group of anaerobes - spirochetes and fusiform bacteria
- The microrgnasims found in large numbers in the slough and necrotic tissue at the surface of the ulcer and also invades greatest distance in the underlying intact tissue at the base of the ulcer. This is evidences by electron microscopy

17
Q

what is the acute phase treatment of NUG

A
  1. Removal of supra and sub signal deposits - ultrasonic scaling
  2. Systemic antibiotic - metronidazole tablets 200mg, 3 times daily for 3 days
  3. Chlorhexidine mouth rinse
18
Q

how is periodontal abscess managed

A

-Is It vital?
- Can drainage be established
- Are there systemic effects - Yes SYSTEMIC ANTIBIOTCS
- Can the occlusal force be reduced

19
Q

what are NICE recommedndatiosn on antibtioic prophylaxis

A
  • prophylaxis against infective endocarditis is not recommended for people undergoing dental procedures
  • based on systemic review of available evidence
  • risk of IE weighed against risk of antibiotic anaphylaxis
20
Q

What is recommended for periostat

A

sub antimicrobial dose oxycylcine
- low dose doxycycline 20mg, twice daily
- sub- antibmciorbial dose ie NOT antibacterial dose
- suggested for extended use, at least 3 months adjunct to scaling and RSD
- similar prescribing for acne
- claimed to reduce bystander damage
-NOT recommended at present in UK

21
Q

what are the non antibacterial effects of tetracycline

A
  • concentrates in GCF
  • binds to root surface
  • slow release
  • fibroblast stimulation
  • Osseous induction
  • anticollagenase