Dentine hypersensitivity and gingival recession Flashcards

1
Q

what is gingival recession?

A
  • gingival margin positioned apical to the cement enamel junction with exposure of the root surface
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2
Q

what is the pocket depth,attachemnt level and gingival recession lengths in gingival recession

A
  • base of pocket to gingival margin (4.5mm)
  • attachement level; CEJ to base of pocket (7.5mm)
    Gingival recession: CEJ to gingival margin (3mm)
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3
Q

what is the prevalence of gingival recession

A

Recession of 1mm or more in 58% of adults age 30+
increase prevalence and extent with age
- 37.8% and extent of 8.6% of teeth in 30-39 year olds
- 90.4% and extent of 56.3% in 80-90yrold

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4
Q

what is the distribution of gingival recession

A
  • maxillary 1st molars and mandibular central incisors
    -upper and lower canine,1st premolar and incisor teeth- define hypersensitivity
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5
Q

where is greater gingival recession common in

A
  • left side of jaw
  • Males V females
  • Afrocarribeans V white Caucasian
  • good oral hygiene associated with buck surfaces (overbrushing)
  • poor oral hygiene associated with lingual surfaces of lower anterior teeth
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6
Q

what is the aetiology of recession

A
  • normal sulcus and undiseases interdental crystal bone
  • anatomical position of tooth
    extent of cortical bone
    tooth position in arch
  • Orthodontic tooth movement
    creation of dehiscnece
    volume of soft tissues
    greater risk of recession with excessive proclamation of lower incisors and arch expansion
    Periodontal disease
    Trauma - foreign bodies- lower lip piercing
    finger nail picking
    toothbrushing- hard toothbrush
  • partial dentures, gingival stripping
    topical cocaine
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7
Q

In recession and keratinised tissue what is the width and thickness

A
  • certain epic-coronal width of keratinised tissue required
  • no minimum width
  • thickness and texture of pre attached ginger
  • thin, fragile tissue predisposed to recession in presence of plaque induced inflammation or trauma
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8
Q

is it thin or thick gingivae where recession is more likely to occur

A
  • thin gingivae
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9
Q

what are the local plaque retention factors

A
  • calculus
  • subgignval margins increase plaque retention
  • more pronounced inflammation see in thin ginger
  • does increase in thickness decrease risk of recession
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10
Q

what are consequences of recession

A
  • fear of tooth loss
  • plaque accumulation and bleeding gignva
    -aesthetics
    -root caries
  • abrasion
  • pain from dentine hypersensitivity
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11
Q

what is dentine hypersensitivity

A
  • characterised by short, sharp pain arising from exposed dentine in response to certain stimuli which cannot be explained as arising form any other dental defect or disease
  • It may go on to manifest as a dull ache beyond the duration of the stimulus, possibly as an altered plural response
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12
Q

explain the epidemiology of dentine hypersensitivity

A
  • The prevalence distribution and appearance of the disease have been reported differently in different studies. These differences are due to the differences in populations, habits,diets and methods of investigation
  • peak incidence is 20-40 years
  • perceived by patient/self reported 8-30%
  • GENDER BIAS TO SNESITVITY f>m
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13
Q

In which order are the most often affected teeth in dentine hypersensitivity

A

buccal/labial and cervical areas

  • first premolars
  • canines
  • incisors
  • second premolars
  • molars
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14
Q

Describe the aetiology of dentine hypersensivtity

A

dentine exposure (lesion localisation )

  • Tubules made patent (lesion ignition)

+ stimulus

but pulp must be vital

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15
Q

describe the brannstrom hdyrodynamic hypothesis

A

Dentien hypersensitivity caused by movement of dentinal tubule contents
- Increased outward fluid flow causes a pressure change across the dentine
- Distortion of A-delta fibre causes pain

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16
Q

Name other possible mechanisms of sensitivity condition

A
  • odontoblasts as receptors - inconclusive
  • nerves in dentine ( only seen in 1% of tubules in the cervical margin, however electrical current and cold stimulate nerves directly
  • Hydrodynamic mechanism
17
Q

describe the hydrodynamic mechanism of pulp nerve activation

A
  • stimulus affects dentinal tubules
  • odontoblasts with odntobalst process extending partway to tubules and nerves
  • A delta/beta fibre conduct impulse form pain experienced for pulp
    -fluid flow changes also result in electrical discharge which may be able to stimulate nerves electrically

rate of fluid flow depends on 4th power of the radius
if a tubule is twice the width fluid flow is 16 X greater

18
Q

what does a sensitive dentine show

A
  • A disrupted smear layer
  • may more dentinal tubules at the surface
  • tubules not occluded by deposit
  • tubule diameter wider
19
Q

what is root sensivtity

A
  • term proposed for dentine hypersensitivity form gingival recession due to periodontal disease and treatment
  • potentially microorganisms invading root dental tubules
20
Q

which factors allow dentine to become exposed

A
  • resulting form loss of enamel
  • removal of enamel by restorative procedures
    -attrition
    -abrasion
    -erosion
  • combined erosion and abrasion
21
Q

how does erosion occur

A
  • intrinsic or extrinsic source of acids
  • enable and dentine loss and surface softening
  • increase tooth surface loss by toothpaste abrasion in intra oral environment acid
    -pH of the acid is not the only factor in erosion
  • Type,chemcial strength,temeprartue,exposrue time are other variable s
  • Think of fruits other than citrus,health supplements, mouthwashes,fruit teas,alcopops
22
Q

how does the dentine become exposed

A
  • gingival recession occurs exposing root surface covered with cementum
  • cementum a relatively weak structure in thin layer
  • cementum not designed to be exposed in the real environment
  • wear and tear including toothbrushing readily remove cementum and expose the dentine beneath
23
Q

how does toothbrushing contribute to dentine hypersensitivity/gingval recession

A
  • toothbrush design/bristles may contirbtue
  • abrasive particles may remove the smear layer and open retinal tubuels
  • detergents might help to remove the smear layer
  • tubules could be occluded with particulate matter form the paste
24
Q

how is hypersneitvity managed

A

Tubule occlusion
- promotes formation of new tissue e.g smear layer, intratubualr dentine,tertiary dentine in response to stimulus or trauma
- application of an artificial barrier.g varnish,dentine ,bonding agents,composite resins, GICs and toothpastes

Blocking plural nerve repsonse
- potassium ions diffuse along tubules and raise extracellualr K+ conc, reducing nerf excitailtity
-un proven in human
- clinically unlikely that ions will diffuse into tubules against flow of dentinal

25
Q

Ideal quality software barrier materials

A
  • retentive
  • insoluble
  • penetrate tubules
  • form oecnaila tags into tubules
    -seal the end of tubules
26
Q

managing hypersensitivity ; home use products

A
  • toothpaste,gels and mouthwashes
  • contain potassium, strontium, oxalates ns fluoride salts
  • Novamin - releases calcium and phosphate ions from saliva to give a HAP like layer
27
Q

describe the Evaluation of efficacy

A
  • inherent problems of clinical trial s
  • pain perception is subject,qualitative and open to individual interpretation
  • psychological medical, gender and cultural issues abound
  • stimulus variable or fixed
  • response is often complex
  • unable to examine effects on subjects tissue
28
Q

Describe the placebo effect

A
  • all products achieved a modest reduction in hypersensitviety
  • irrespective of presence of active ingredients
    placebo effect of 40%
29
Q

describe how to manage hypersensitivity in surgery

A
  • varnish duraphat
    clinpro
  • 1-3 layers of adhesive resin bonding systems / seal and protect, optioned solo.scothc bond
  • desensitising polishing paste
  • reinforced GIC where theirs abrasion cavity progression
  • watch for overhangs at the gingival margin
30
Q

describe rpevenatiave patient advice

A
  • smoking cessation
  • reduce risk factors- diet history, limit acidic drinks,nightime splints OHI
  • orthodontic therapy
    -partical dentures and restorations
31
Q

How is root caries treated

A
  • radigraphas to detect root caires interporximally
  • prevention; diet OHi
    -prevntion; flruodie,mouthrinses, gels, custom amde trays , topical professional application
  • recontuirng shallow lesions
    -conventional restoration
32
Q

how is periodontal disease treated?

A

initial therapy
correctiev therapy
supportive therapy

33
Q

how are aesthetics restores from gingival recession

A
  • reassure re further progression
  • removable gingival veneer silicone or acrylic
    -mask to cover the black traingles of interdental spaces
  • crowns and veneers
  • root coverage
34
Q
A