Swine Viruses Flashcards

1
Q

virus family of Hog Cholera

A
  • flavivridae
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2
Q

hog cholera also known as

A
  • classical swine fever
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3
Q

hog cholera affects swine of what age group and what is its morbidity and mortality

A
  • all ages

- high morbidity and mortality

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4
Q

T/F: Hog Cholera occurs in the US

A
  • false - was eradicated
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5
Q

T/F: severe leukopenia is always present with Hog Cholera

A
  • true
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6
Q

swine infected with Hog Cholera how does their skin change

A
  • hyperemia and purpura of abdomen and ears
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7
Q

T/F: young swine may die preacutley but they always show clinical signs before

A
  • false - they show no clinical signs
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8
Q

effect of Hog Cholera on pregnant Sows (4)

A
  • reabsorption
  • abortion
  • fetal mummification
  • still births
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9
Q

what disease of hog cholera is associated with extended or intermittent clinical disease

A
  • subacute and chronic
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10
Q

most common route of entry for Hog Cholera

A
  • ingestion
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11
Q

how does hog cholera create profound leukopenia and thrombocytopenia

A
  • 2nd replication in endothelial cells, lymphoid organs, bone marrow -> hemorrhages
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12
Q

pathognomonic lesion of Hog Cholera

A
  • infarction of spleen
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13
Q

most prominent lesion of Hog Cholera

A
  • general exhaustion of lymphoid system -> white cells are completely gone
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14
Q

what happens to pigs that are live born from a dam infected with Hog Cholera

A
  • are persistently infected
  • immunologically tolerant
  • life-long virus shedders
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15
Q

T/F: Hog Cholera is not notifiable

A
  • false - it should be notified
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16
Q

how is hog cholera transmitted

A
  • direct or indirect contact - avoid fomites
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17
Q

virus family of African Swine Fever (ASF)

A
  • asfaviridae
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18
Q

T/F: ASF needs it envelop to infect

A
  • false
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19
Q

which virus causes paracrystalline arrays in the cytoplasm of infected cells

A
  • African Swine Fever
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20
Q

what is the reservoir and vector of African Swine Fever

A
  • soft ticks
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21
Q

what are the 2 epidemiology of African Swine Fever

A
  • in warthog

- in domestic swine

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22
Q

T/F: severe leukopenia is a clinical sign of African Swine Fever

A
  • true
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23
Q

mortality rate of African Swine Fever

A
  • can be high in naive populations
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24
Q

where are gross lesions of African Swine Fever located

A
  • lymphatic and vascular systems
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25
what clinical sign of African Swine Fever is also seen in Hog Cholera
- enlarged friable spleen | - petecchia in cortex of kidneys
26
T/F: swine that survive African Swine Fever are free of disease
- false - are persistently infected
27
T/F: there is a vaccination for African Swine Fever
- false - hard to make because it does not need its envelope so it avoids producing neutralizing antibodies
28
viral family of pseudorabies
- herpesviridae
29
T/F: pseudorabies does not exist in the wild pig population
- false - it does in the southern states
30
pregnant sow infected with pseudorabies before day 30
- resorption
31
pregnant sow infected with pseudorabies after day 30
- abortion
32
pregnant sow infected with pseudorabies later
- mixture of mummified, macerated, stillborn, weak and normal piglets
33
mortality rates in piglets born to non-immune sows of pseudorabies
- 100%
34
weaned, growing and mature swine clinical signs for 4-8 days with pseudorabies
- sneezing - coughing and fever - constipation and occasional vomiting
35
day 5 of infection with pseudorabies
- incoordination, muscle spasm, circling | - neurologic signs
36
day 6-8 of infection with pseudorabies
- moribund and die within 12 hours
37
secondary host of pseudorabies - cattle clinical signs
- pruritus "mad itch" | - progressive CNS involvement with death
38
secondary host of pseudorabies - dogs clinical signs
- no tendency to attack
39
T/F: pseudorabies has viremia
- false
40
shedding of recovered pigs from pseudorabies
- can shed continuously in nasal secretions
41
diagnosis of pseudorabies
- antibody ELISA
42
T/F: pseudorabies can be shed in feces and urine as well as saliva and nasal discharge
- false - only through saliva and nasal discharge
43
virus family of Postweaning multisystemic wasting syndrome (PMWS)
- circoviridae
44
clinical signs of PMWS
- multi-systemic wasting syndrome of weaned piglets
45
3 forms of PMWS
- reproductive failure (breeding) - respiratory disease in finishing pigs - porcine dermatitis and nephropathy syndrome
46
T/F: PMWS can be shed in feces and urine as well as saliva and nasal discharge
- false - is shed in the feces
47
is there a vaccine for PMWS
- yes - but is strain specific
48
porcine parvovirus viral family
- parvoviridae
49
ages that porcine parvo effects and what dramatic effects can be seen
- domestic swine of all ages | - dramatic effects in naive pregnant pigs (stillbirth, mummified, embryonic death, infertility)
50
porcine parvo infection before 30 days of gestation
- fetal death and resorbtion (EDI)
51
porcine parvo infection between 30-70 days of gestation
- usually stillbirth and mummified
52
porcine parvo infection after 70 days of gestation
- developing lesions
53
porcine parvo effect on boars
- low fertility
54
pathogenesis of porcine parvo
- replicates in tonsils, LN, thymus, spleen, lungs and other organs of fetus - also replicates in peripheral blood lymphocytes (monocytes & macrophages -> leukopenia)
55
how many days does it take after infection for porcine parvo to reach the fetus
- 15 days
56
diagnosis of porcine parvo
- IF of fetal tissues
57
shedding of porcine parvo in boars
- shed in semen
58
vaccination window of porcine parvo
- 7 months
59
transmissible gastroenteritis (TGE) virus family
- coronaviridae
60
4 different diseases of TGE
- transmissible gastroenteritis (TGE) - vomiting and wasting disease - porcine epidemic diarrhea - respiratory disease
61
vomiting and profuse yellow diarrhea clinical signs of what virus
- transmissible gastroenteritis (TGE)
62
in older pigs clinical signs of transmissible gastroenteritis (TGE)
- course is shorter and rarely any vomiting
63
pathogenesis of transmissible gastroenteritis (TGE)
- villous enterocytes of small intestines are destroyed -> shortening of villi
64
why is recovery of transmissible gastroenteritis (TGE) fast
- crypt cells are unaffected
65
diagnosis of transmissible gastroenteritis (TGE)
- gross lesions are limited to GI
66
which antibodies are protective against transmissible gastroenteritis (TGE)
- maternal IgA - protective | - systemic IgG - no protective
67
swine vesicular disease virus family
- picornaviridae
68
swine vesicular disease has similar clinical signs as what other virus
- FMD
69
swine vesicular disease shed in what excretion
- feces
70
vesicular exanthema of swine virus family
- caliciviridae
71
from what disease is indistinguishable clinically from vesicular exanthema of swine
- FMD
72
swine influenza virus family
- orthomyxoviridae
73
entry of swine influenza
- aerosol
74
porcine reproductive and respiratory syndrome (PRRS) virus family
- arteriviridae
75
what virus persists well in environment even at low temperatures
- porcine reproductive and respiratory syndrome (PRRS)
76
transmission of porcine reproductive and respiratory syndrome
- contact - airborne - sexual transmission via semen
77
clinical signs of porcine reproductive and respiratory syndrome (PRRS)
- blue discoloration of ears, snout, vulva
78
T/F: a vaccine is available for reproductive and respiratory syndrome (PRRS)
- true