Swine Diseases Flashcards
PRRS and Swine Influenza
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
APP& glasser’s disease
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
E. coli and salmonella
Enteric disease Nose to nose or nose to feces contact Diarrhea Dehydration Vomiting Lethargic Decrease water consumption Off feed
TGE and Swine Dysentery and coccidiosis
Enteric disease Nose to nose or nose to feces contact Diarrhea Dehydration Vomiting Lethargic Decrease water consumption Off feed
Strep. Suis& erysipelas & PMWS
Systematic disease Nose to nose contact or through air Poor condition Pale skin Swollen joints Convulsions Increases LN Decrease appetite
Swine pox&greasy pig disease&mange&lice
Skin diseases Poor condition Pale skin Dark color areas skin Flaking Decrease appetite
Transmissible gastroenteritis
Highly contagious VIRAL disease affecting pigs on all ages
Newborns less than 10
Caused by corona virus
Spread by aerosol, direct fecal contact or fomite
Incubation
Transmissible gastroenteritis:host risk factors
Parity
Herd immunity levels
Environmental/management factors (all in all out)
Transmissible gastroenteritis: pathogen risk factors
Persists in environment for a few weeks
Survives freezing
Sensitive to light and disinfectants
Transmissible gastroenteritis: immune response
Strong antibody response ( IgA in intestinal tract protection)
SIgA (excreted in sows milk and passive immunity for piglets)
Best passive immunity from natural GI infection of the sow
Transmissible gastroenteritis: pathogenesis
Nasal secretions and milk-replicates respiratory intestinal tracts
Destroys epithelial cells jejunum and Illium
Transmissible gastroenteritis: clinical signs
Vomiting, intense thirst, dehydration
Diarrhea
Pigs less than a year old die
Abortion
Transmissible gastroenteritis: diagnosis
Clinical signs
Necropsy
Antibodies of intestinal contents
Transmissible gastroenteritis: treatment and control
No specific treatment Increase farrowing room temp Vax Feed ground up dead to sows Sanitation
RECOVERED PIGS ARE IMMUNE FOR ONE YEAR
Atrophic Rhinitis & mycoplasmal pneumonia
Respiratory disease Nose- nose contact Cough, fever Labored breathing, nasal discharge Snout dis formation Dehydration Decrease feed and water consumption
E. Coli, salmonella, TGE, swine dysentery, coccidiosis
Enteric diseases(spread nose to nose to forced contact) Diarrhea Dehydration Vomiting Off feed Weird water amounts Lethargic
Strep. Suis, erysipelas, PMWS
Systematic diseases(nose to nose contact or through air) Poor condition, pale skin, dark areas on skin, swollen joints,
Skin diseases
Swine pox
Greasy pig disease
Mange
Lice
Pale skin
Flaking
Transmissible gastroenteritis (TGE)
HIGHLY contagious a VIRAL disease
Caused by coronavirus
Spread through aerosol, direct fecal oral contact, or fomites
Epidemic: explosive in naive herds–lasts 3-5 weeks
Endemic: partial immunity, contagious introduction of susceptible pigs
Transmissible gastroenteritis (TGE): HOST risk factors
Parity
Herd immunity levels
Environmental/management factors (continuous flow)
Transmissible gastroenteritis (TGE):PATHOGEN risk factors
Persists in environments for a few weeks
Survives freezing
Sensitive to light aNd disinfectants
Immune response
Pigs develop strong antibody response (IgA)
sIgA: excreted in Sows milk_passive immunity
BEST PASSIVE IMMUNITY FROM NATURAL GI INFECTION OF SOW
Transmissible gastroenteritis (TGE): clinical signs
Vomiting, thirst,
Diarrhea(yellow with bad odor)
Abortion
Subsequent infections not as severe
Transmissible gastroenteritis (TGE): diagnosis
Clinical signs
Necropsy
Fluorescent antibodies of intestinal contents
Transmissible gastroenteritis (TGE): treatment and control
NO TREATMENT
Recovering pigs immune for a year
Increased farrowing room temp Vax Feed grand up dead to sows Sanitation and disinfection Enhanced immunity from MLV (IgG) reduces disease severity
Porcine Epidemic Diarrhea (PED)
Important disease in Europe and Asia No cross protection between TGE and PED Maternal antibodies protect piglets Immunity is short lived and reinfection can occur (chronic) Diagnosis: PCR Prevention:bio security and vax
Porcine Reproductive and Respiratory Syndrome (PRRS)
Important disease in NA and Europe
VIRAL: caused by artervirus
Spread by close contact and in uterus
Porcine Reproductive and Respiratory Syndrome (PRRS): risk factors
Herd level of immunity and general herd health Virus strain Overcrowding Continuous barn flow Pregnancy state at time of infection
Porcine Reproductive and Respiratory Syndrome (PRRS): signs of repro form
Increased stillborns, mummies, abortions and weak piglets
Sows feed decreased which increases pre weaning
Will run through herd in four months
Porcine Reproductive and Respiratory Syndrome (PRRS): signs of respiratory form
Chronic pneumonia
Decreased weight gains
Blue eared disease
Increased death loss
Porcine Reproductive and Respiratory Syndrome (PRRS): diagnosis
Vague Clinical signs Blood tests Virus insulation Necropsy No treatment available
Porcine Reproductive and Respiratory Syndrome (PRRS): control
All in all out segregation
Buy known replacements with retesting during isolation
Disinfecting
Quarantine for 60 days
Vaccinate (MLV when not pregnant) killed & limited cross protection
Pseudorabies
Mad itch, aujeszkys disease
VIRAL
Pigs are host
