SVT Flashcards
What is the most common type of paroxysmal supraventricular tachycardia (SVT)?
AV nodal reentrant tachycardia (AVNRT).
What is the primary mechanism of AVNRT?
A reentrant circuit within the AV node due to dual conduction pathways.
What is the most common risk factor for AVNRT?
Idiopathic, with onset typically in young adulthood.
What are the common clinical symptoms of AVNRT?
Palpitations, dizziness, dyspnea, and sometimes syncope.
What does the ECG show in AVNRT?
Narrow-complex tachycardia without visible P waves or with retrograde P waves.
What is the first step in evaluating a patient with tachyarrhythmia?
Assess airway, breathing, circulation (ABCs), secure the airway if necessary, and check hemodynamic stability.
What is the stepwise approach for treating narrow QRS tachycardias?
If stable, use vagal maneuvers, then adenosine, then beta-blockers or calcium channel blockers. If unstable, perform synchronized cardioversion.
What is the first-line treatment for hemodynamically unstable SVT?
Synchronized cardioversion, also called direct countershock (DC cardioversion).
What are the indications for synchronized cardioversion in SVT?
Severe hypotension, ischemic chest pain, altered mental status, or signs of shock (<90 mmHg SBP).
What is the initial management for hemodynamically stable SVT?
Vagal maneuvers such as Valsalva, carotid massage, or ice pack application (in children).
What is the next step if vagal maneuvers fail in stable SVT?
Intravenous adenosine.
What is the dosing protocol for adenosine in SVT?
Initial dose of 6 mg IV push, followed by 12 mg if the first dose is ineffective.
What is the effect of adenosine on the AV node?
Temporarily blocks conduction, terminating AVNRT and unmasking atrial activity.
What are contraindications to adenosine use?
Asthma due to risk of bronchospasm, and severe hypotension.
What are common side effects of adenosine?
Facial flushing, hypotension, shortness of breath, and transient bradycardia.