Surg - Viva - Abdominal Compartment Syndrome Flashcards
Normal intra abdominal pressure
5-7mmHg
What is intra abdominal hypertension and compartment syndrome defined
1) Abdominal Perfusion Pressure = MAP - Intra-abdominal pressure
Intra abd hypertension is IAP >12 mmHg
Grade 1 - 12-15
2 - 16-20
3 21-25
4 >26 mmHg
Comparrment syndrome - Sustained of > 20mmHg that is associated with new organ dysfunction
(Occurs with or without an APP of < 600mmHg
Risk factors of IAH and ACS
Low abdominal wall compliance
Surgery with tight closure
Trauma, burns, obesity
Proning
Increased abdominal content
Intralumin - ileusm gastroparesis, psuedo obstruction
Extraluminal - ascites, haemoperitoneum, pneumoperitonieum
Capilary leak Sepsis Pancreatitis Burns Hypothermia, acidosis, transufion Positive fluid balance, fluid resus
Mechanical vent
PEEP?>10
Increased head of bed angle
Shock/hypotension
How do you measure intra abdominal pressure
Direct - needle into abdomen - laparoscopy
Indirect - Transduce in a viscous, such as bladder, stomach, colon, uterus
Intra-vesical pressure commonly used via bladder catheter
25ml sterile saline put into bladder and drainage bag reconneted and cross clamped
16G needle connected to pressure transducer introduced via the culture port site of catheter.
Resp effects of IAH
Atectasis and collapse, diaphragm splinting
Reduced chest wall and lung compliance
VQ mismatch, hypoxaemia and hypercapnia
PEEP worsens venous return and CO
Cardiac effects of IAH
Raised IAP directly pressures the vasculature
Reduced cardiac output as reduced venous return
increased afterload by arterial compression
Reducing cardiac output reduced the perfusion pressure
Neuro effects of IAH
Raised ICP secondary to impaired venous return do to raised intra thoracic pressure
Hypoxaemia and hyperapnoea causes cerebral vasodilation, worsening ICP
Renal effects of IAH
Pressure on renal vasculature
Reduced CO –> reduced renal blood flow
Pressure on renal outflow tract, increased pressure in tubules,and reduces filtration gradient
Activation of RAA system, worsens the insults
Gastric/hepatic IAH
Hypoperfusion worsened by venous hypertension, –L bowel wall oedema
Bowel ischaemia and translocation, risk of sepsis
Reduced hepatic flow
Biliary stasis
Principles of management
ABCDE approach and treat abnormalities
Optimise phsyiology and metabolic derangement
First - spot it - monitor at risk patients
Medical management - adequate sedation, analgesia and muscle relaxation, avoid coughing straining
Avoid prone position
Fluid - avoid excess admin, consider RRT for fluid removal
Evaculate the GI tract - gastric decompression, prokintetics, laxatives
Drain collections, paracentesis
Organ support - APP>60 with vasopressors
Optimise the vent, adequate ventilation with recruitment
Surgical - laparotomy and decompression is definateive
Negative pressure wound therapy for an open abdo (laporostomy)
Complications
Death - untreated, mortality near to 100%
MOF
Why do an open abdomen
Used to be a last resort
Now preferred strategy in compartment syndrome in both trauma/non trauma patients
Open abdomen approach enables initial control of haemorrhage and/or contamination, allow peritoneal packing, faciliative resus to normal physiology and subsequent re-exploration
Specific indications for open abdo
Severe necrotising pancreatitis
Abdominal sepsis
Damage control post trauma
Emergnecy vasculr surgery
Issues with open abdo’s
Nursing - skin care around wound moist and could be damaged
Turning and positioning
Pain
Sig. fluid loss, post op losses high, litres per day
Unreliable fluid balance
Protein and nitrogen loss - malnutrtion
Infection - wound care makes sterility difficult
Visceral injury - adhesions
Ileus
Abdo wall hernias,
Maybe never closes
Enterocutaneous fistula
ITU management of open abdomen
Pain relief
Nutrition
Nursing
Resus to restore physiology
Correct hypothermia/coag/acidosis
Lung protective vent stratgey
Sedation/analgesia/blockade
Aim RASS -4 in acute phase
Enteral feeding is safe in intact GI tract
Supplemental protein, 2g Nitrogen per litre of fluid content
Abx if needed
Monitor for ACS
Source control - ?bedside washout