Spinal Injuries Learning Flashcards

1
Q

What is tetraplegia/quadriplegia?

A

It is a spinal cord injury above the first thoracic vertebra resulting in partial or complete loss of movement and sensation in all four limbs, along with weakened breathing and coughing.

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2
Q

What distinguishes paraplegia from tetraplegia?

A

Paraplegia occurs below the first thoracic spinal nerve, affecting the trunk and lower limbs but preserving arm function.

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3
Q

Define a complete vs. an incomplete spinal cord injury.

A

A complete injury results in total loss of sensory and motor function below the injury level, while an incomplete injury allows some sensory and motor function.

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4
Q

What are the characteristics of upper motor neuron (UMN) lesions?

A

They involve hyper-reflexia, spasms, and increased muscle tone due to intact reflex arcs.

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5
Q

How do lower motor neuron (LMN) lesions present?

A

They involve flaccid paralysis, no spasms, and absent reflexes due to disrupted reflex arcs.

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6
Q

What is anterior cord syndrome?

A

It is damage to the anterior spinal cord affecting motor function, pain, and temperature sensation, sparing proprioception and vibration.

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7
Q

What are the effects of central cord syndrome?

A

It predominantly affects the hands, arms, and trunk due to central damage to the spinal cord, sparing the legs.

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8
Q

What is autonomic dysreflexia, and what level of injury can lead to it?

A

It is a dangerous condition of excessive sympathetic response (high BP), occurring in spinal injuries at or above T6.

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9
Q

Name three symptoms of autonomic dysreflexia.

Name three symptoms of autonomic dysreflexia.

A

Pounding headache, sweating above the injury, and slow pulse (<60 bpm).

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10
Q

How does spasticity in SCI patients present, and what are its benefits?

A

It manifests as increased muscle tone and spasms but helps maintain muscle size, circulation, and can aid functional activities like transfers.

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11
Q

What are the risks and management strategies for pressure sores in SCI patients?

A

Risks include reduced sensation, muscle wasting, and incontinence. Management involves pressure-relieving cushions, regular repositioning, and proper seating.

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12
Q

What is the primary concern with heterotopic ossification in SCI?

What is the primary concern with heterotopic ossification in SCI?

A

The formation of bone outside the skeleton can lead to joint stiffening and fusion.

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13
Q

What neurological assessment tool is standard for spinal cord injuries?

A

The American Spinal Injury Association (ASIA) Impairment Scale (AIS).

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14
Q

What is Brown-Séquard Syndrome?

A

A spinal cord lesion affecting one side, causing:

Same-side: loss of motor function, proprioception, vibration, and light touch.
Opposite-side: loss of pain, temperature, and crude touch sensation.

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15
Q

What are the key features of Cauda Equina Syndrome?

A

Loss of movement and sensation in lower limbs.
Flaccid paralysis.
Absent reflexes.
Bladder, bowel, and erectile dysfunction.

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16
Q

What are expected functional outcomes for a C6 spinal cord injury?

A

Tenodesis grip.
Potential for independence in washing, dressing, and personal care with assistive devices.
Ability to self-propel on flat surfaces indoors.
Can drive with vehicle modifications.

17
Q

What are the symptoms of autonomic dysreflexia?

A

Hypertension (>200/100 mmHg).
Pounding headache.
Sweating and flushing above the injury level.
Slow pulse (<60 bpm).
Nasal congestion and anxiety.

18
Q

What are the primary risks of osteoporosis in SCI patients?

A

Weak and brittle bones due to reduced weight-bearing and muscle activity.
Increased fracture risk.
Prolonged healing time for fractures.

19
Q

How does postural hypotension present in SCI patients?

A

Dizziness and light-headedness upon sitting or standing.
Blood pooling in the lower extremities.
Can lead to fainting if untreated.

20
Q

How does spasticity develop in SCI patients?

How does spasticity develop in SCI patients?

A

Hyperactive stretch reflexes due to lost corticospinal tract inhibition.
Triggered by infections, skin breakdown, or tight muscles.
Peaks in the first year post-injury.

21
Q

What are common causes of respiratory complications in SCI?

A

Paralysis of intercostal and accessory muscles.
Poor cough ability.
Immobility leading to infections.

22
Q

What are the benefits of spasticity in SCI?

A

Maintains muscle size and circulation.
Can aid in transfers.
Acts as a warning system for issues like infections or pressure sores.

23
Q

What are the primary risks of developing pressure sores in SCI patients?

A

Reduced sensation, leading to unnoticed soreness.
Muscle wasting causing prominent bony areas.
Incontinence issues.
Increased tone causing poor posture.
Aids: Pressure-relieving cushions, regular turning, and proper seating.

24
Q

What factors contribute to respiratory complications in SCI patients?

A

Paralysis of intercostal and abdominal muscles.
Reduced diaphragm function.
Prolonged bed rest.
Associated injuries like rib fractures.

25
Q

What causes heterotopic ossification in SCI, and how is it managed?

A

Caused by abnormal bone growth outside the skeleton, typically near large joints.
Managed with physical therapy and, in severe cases, surgical release.

26
Q

What are the common signs and symptoms of a urinary tract infection (UTI) in SCI patients?
A:

A

Frequent urination.
Burning sensation during voiding.
Fever and chills.
Cloudy or strong-smelling urine.

27
Q

What is syringomyelia, and how can it affect an SCI patient?

A

It is the enlargement of the spinal cord’s central canal.
Can cause numbness and weakness above the original injury level.

28
Q

How is autonomic dysreflexia treated in SCI patients?

A

Sit the patient upright to lower blood pressure.
Check and remove noxious stimuli (e.g., catheter blockage).
Monitor blood pressure and seek medical attention if elevated.

29
Q
A