Spinal cord injury Flashcards
Ascending tracts
spinothalamic- pain and temp- crosses in SC, dorsal columns- cutaneous and gracilis- touch, sense, vibration- crosses in medulla
spinocerebellar- uncrossed
descending tracts
corticospinal- lateral crosses in medulla, anterior crosses at spinal- lateral motor info to limb, anterior- motor info to axial muscle
vestibulospinal (uncrossed)- integration of head and neck and trunk with extremities
reticulospinal- lateral facilitates flexion and inhibits extension (medial opposite)
rubrospinal (crosses at origin)- control fine movement
tectospinal (crosses at origin)- control muscles in response to visual stimulus
why is the SC vital
conveying sensory info to brain and motor info to and from the periphery, SC lesions may impair motor, sensory and autonomic function
traumatic lesions- 84%
non traumatic SC lesion
degenerative disc disease and SC stenosis, spinal infract, tumour, inflammation of SC, viral infection, developmental/ congenital abnormalities
SC injury
80% of cases occur in males, male aged 15-25, 10-80 cases per million per year
vulnerable areas of the vertebral columns
C5-7- 55% of all, thoracolumbar0 T12, mid thoracic- T4-7
autonomic NS
damage to SC can impact this, parasympathetic- cervical and radial damage and sacral- calms everything down, sympathetic- thoracic and lumbar- responsible for increase BP, HR, Respiratory rate- fight or flight
pathophysiology of SC lesion
need to have normal oxygenation, perfusion, and acid/base balance to aid management of injury
vasogenic oedema and altered blood flow account for clinical deterioration
3 different mechanisms of SC injury
destruction from direct trauma, compression by bone fragment, hamatoma or disc material, schema from damage or impingement on the spinal arteries
what is tetraplegia or quadriplegia
impairment or loss of motor control and/ or sensory function in cervical segment of the cord
it affects all 4 limbs
what is paraplegia
impairment or loss of motor and/or sensory function in thoracic, lumbar and sacral segments
complete SC lesion
complete loss of function below point of injury, outcome more predictable, impairment of motor function
damage to descending pathways- UMN, damage to anterior motor neurone and LMN
spinal shock
at level of lesion- complete destruction of nerve cells= flaccidity, gradually anterior horn cells below level of lesion have ability to recover but have no control from higher centre= spasticity and spasm
incomplete SC lesion
some sparing of neural activity below the level of lesion, more common- 55-65%, outcome less predictable,
incomplete SC lesion- anterior cord syndrome
motor paralysis below lesion, loss of pain and temp, retain proprioception and vibration, cause- disc herination
incomplete SC lesion- central cord syndrome
central cord syndrome- associated with whiplash, motor dysfunction in upper limbs, bladder dysfunction, corticospinal and spinothalamic tract