lower limb pathologies 1 Flashcards

1
Q

Avulsion injuries

A

most common in adolescence who are involved in sport as the tendons are stronger than apophyses (where tendon attach), bones are not ossified. occur when tendon/ ligament is pulled of the bone and a fragment of cortical bone with it

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2
Q

where are avulsion injuries common

A

glut med/ min- GT, iliopsoas- LT, ASIS- sartorius, ischial tuberosity- hamstrings, AIIS- rec fem

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3
Q

what can delayed treatment to an avulsion injury lead to

A

sub optimal outcome- not always possible to repair tendon if left too long

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4
Q

what is a stress fracture of femoral neck

A

They are a consequence of exceeding repetitive submaximal loads, which creates an imbalance between bone resorption and bone formation, early recognition is necessary to prevent development, can become displaced, losing blood supply to head of femur

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5
Q

how are stress fracture of femoral neck diagnosed

A

they appear white on MRI (high signal zone)= early aspect of stress fractures, the MRI is a test looking for bone marrow oedema
1% occur at femoral neck

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6
Q

risk factors stress fracture of femoral neck

A

common in runners, less in general population,
REDs- relative injury deviancy syndrome- look for female athletes in teens/20’s and exercising a lot- they do not have correct calorie intake for amount burned, reducing bone density

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7
Q

what is avascular necrosis

A

this is a condition where there is a loss of blood supply to the bone. bone is a living tissue, hence loss of blood supply, means bone death. Can lead to bone collapse and arthritis

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8
Q

symptoms of avascular necrosis

A

stiffness of hip, night pain, limp, pain in groin, buttocks, front of the thigh

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9
Q

femoral neck avascular necrosis

A

the femoral head receives its blood supply through the neck of femur. fractures across this zone may cause a loss of this supply leading to tissue death

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10
Q

what is hip dysplasia

A

where the hip socket doesn’t cover the ball portion of thigh bone, it may lead to hip joint being dislocated, the joint doesn’t form around the ball leading to excessive movement

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11
Q

hip dysplasia- centre edge angle

A

normal centre edge between 25-40°, <25-30 borderline dysplasia, 20° dysplasia, <16° will develop arthritis

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12
Q

labrum tears

A

this involves rim of collagen called labrum, also associated with condole injuries- injury to articular cartilage. end range of motion position of hyper-abduction, hyperextension, hyper-flexion and ER contributes to tears

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13
Q

risk factors labrum tears

A

football, ice hockey, soccer, the majority are not caused by specific event, standard MRI only has 35% sensitivity and accuracy in detecting labrum pathology, accuracy can be improved by up to 90% using contrast

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14
Q

2 mechanisms of impingement

A

CAM type- FAI- femoral side of join, pincer type FAI- acetabulum side of joint, pathological hip condition characterised by abnormal contact of femoral head/ neck junction with acetabulum, can occur in normal ROM as a result of bony abnormality

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15
Q

what does FAI stand for

A

femoral acetabular impingement

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16
Q

hip impingement CAM FAI

A

this is caused extra bony growth/ tuberance on the proximal femoral neck of head junction. can become symptomatic in physical young males- growth plates

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17
Q

how are hip impingement CAM FAI diagnosed

A

radiography- alpha angle- X ray hip at 90° flexion and 20° abduction

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18
Q

Hip impingement pincer

A

result of excess acetabular coverage of femoral head-pressing on labrum, can be global (coxa profunda)- deep hip socket, or focal anteriorly (acetabular retroversion)- altered orientation rim pressing upon labrum

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19
Q

how can Hip impingement pincer be located

A

, can be located on radiographic imaging by looking at lateral centre head angle- line from head of femur, second line going to rim of acetabulum- <40°=positive

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20
Q

does hip impingement lead to

A

it can lead to osteoarthritis but not always- increased risk of impingement in athletes competing in sport in early adult year, less risk compared to general population,

21
Q

how can Hip impingement pincer occur

A

can occur in extreme ROM such as ballie and gymnastics, bone deformity from birth, may be caused by repetitive abutments of articular cartilage

22
Q

greater trochanteric pain syndrome

A

GT[S common cause of lateral hip pain, attribute to tendinopathy of glut max/min, or bursal pathology, compressive forces cause impingement of these structures onto the GT by the ITB- puts pressure on tendons and structures on bursa and glut med/min tendons

23
Q

risk factors greater trochanteric pain syndrome

A

females 40-60, post-menopausal, lower femoral neck shaft angle, increase BMI

24
Q

snapping hip syndrome/ coxa saltans

A

characterised by audible snapping of hip joint during movement, can be charcterised into internal and external

25
Q

internal snapping hip syndrome

A

caused by iliopsoas over iliopectineal eminence, paralabral cysts

26
Q

external snapping hip syndrome

A

cause by ITB snapping over GT, or proximal hamstring tendon rolling over ischial tuberosity, or TFL or glut max over GT

27
Q

what does a chondral refer to

A

a chondral defect refers to a focal area of damage to the articular cartilage
osteo- bones, chondral- cartilage

28
Q

what is an osteochondral defect

A

refers to a focal area of damage that involves both the cartilage and piece of underlying bone, can occur acutely or develop as a result of several chronic condition

29
Q

what is osteochondral defect caused by- osteochondral fragment

A

can be due to seperation of the osteochondral fragment caused by an acute traumatic injury or as the end result of an unstable fragment in oesochondritis dissecans- where a small segment of bone separates due to a lack of blood support

30
Q

what is osteochondral defect caused by- osteochondral ion

A

could occur due to acute osteochondral impact ion of bone with resultant contour deformity

31
Q

what is osteochondral defect caused by- collapsed subchondral bone

A

could occur due to a collapse of the subchrondral bone in a subchondral insufficiency fracture (where underlying bone loses bone density leading to insufficiency fracture) or avascular necrosis or a bone collapse uncovering a large subchondral cyst

32
Q

subchondral cyst- synovial fluid interfusion theory

A

which proposes that articular surface defects and increase intra-articular pressure allow intrusion of synovial fluid into the bone, leading to formation at cavities

33
Q

subchondral cyst- bone contusion theory

A

according to which non- communication cysts arise from subchondral foci of bone. necrosis that are the result of opposing articular surface coming in contact with each other

34
Q

groin injury

A

3 points of the traiangl- ASIS, pubic tubercle, 3G (groin, gluteal, greater trochaner)

35
Q

osteitis pubis-

A

overuse injury causing localised tenderness over pubic symphysis, pubic symphysis instability caused by microtrauma due to repetitive muscle strains, thought to be from abd and add muscles as they attach to pubic bones and act antagonistically, reduced IR= risk factor

36
Q

osteitis pubis- symptoms

A

pain exacerbated by running, pivoting on 1 leg, kicking, or pushing off to change direction, pain=walking, climbing stairs, coughing or sneezing
clicking/popping- DTS, turning i nbed or walking
weakness and difficulty ambulating
fever, chills orrigors along with pubic pain

37
Q

hip bursitis- subcutaneous trochanteric bursitis

A

pain in buttock and lateral aspect thigh, onset gradual, walking uncomfortable, full pass hipflex/ER/ADD= painful, resisted MR/ER/EXT/ABD= pain

38
Q

hip bursitis- trochanteric bursitis

A

can sometimes be inflammation of the insertion of glut med, pain proximal and lateral thigh, patient will have tender ITB, myofascial pain from secondary muscles

39
Q

hip bursitis- iliopsoas bursitis

A

uncommon, no specefic agg, +ve passive and active add (90°), =ve LR, -ve all resisted movements

40
Q

myositis ossification-

A

formation of bone within a muscle, caused by returning to activity too quickly post dead leg

41
Q

myositis ossification- cause

A

when muscle injured or bruised, bony tissue can be deposited into the bruise during healing process
not taking measure to reduced inflammation, calcification occurs 2-4 weeks after initial injury

42
Q

myositis ossification- symptoms

A

unsually slow recovery, pain and ROM often improve- pain and muscle stiffness worsen as bone matures, hard bumps

43
Q

pre patella bursitis-

A

can occur from infectious nature (30%) or a non-infectious nature (70%)

44
Q

pre patella bursitis- causes

A

a direct fall on the patella, an acute trauma, repeated blows or friction on the knee, infectious or low grade inflammatory condition, prolonged knee flexion

45
Q

pre patella bursitis- symptoms

A

pain, swelling, differential warmth around the knee, painful and limited ROM at the knee, if bursitis’s caused by an infection, pain is associated with fever and chills

46
Q

foot compartment syndrome

A

compartment syndrome is a condition where bleeding or oedema develops in area of the body which is surrounded by non-expandable structures of bone and fascia, increasing local pressure, 4 compartments= interosseous, medial, lateral, central)

47
Q

foot compartment syndrome- causes

A

crush injuroes, burns, tightly fitting bandages, fractures, steriods

48
Q

foot compartment syndrome- symptoms

A

pain- sever and spontaneous or occurring during exercises, pain with passive stretch, pain during DF, increased soreness caused by moving toes, pain on palpation, swelling or tightness, pale skin, sensory deficits, firmness, muscle weakness, 5 p’s(pain, pallor, paresthesia, paralysis, paralysis, pulselessness