Spinal Cord Compression Flashcards

1
Q

describe the corticospinal tract

A

2 neurone tract
UMN = from motor cortex > anterior grey horn
decussates at medulla
ipsilateral tract
LMN = anterior horn cell > site of action

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2
Q

features of UMN lesion?

A

increased tone
no real muscle wasting
hyperreflexia
no fasciculation

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3
Q

features of LMN lesion?

A

decreased tone
muscle wasting
fasciculation
diminished reflexes

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4
Q

describe spinothalamic tract?

A

carried info on pain, temp and crude touch
contralateral tract
decussates at spinal level

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5
Q

describe dorsal columns

A

carried info on fine touch, proprioception, vibration
ipsilateral tract
decussates at medulla

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6
Q

which spinal joint is most commonly affected in rheumatoid arthritis?

A

C1/C2

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7
Q

what is cord transection?

A

conceptual term meaning spinal cord is injured
can be complete (all motor and sensory modalities below level of injury affected)
can be incomplete (some power/sensation preserved)

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8
Q

how does cord transection present?

A

hypotension (due to loss of sympathetic outflow below lesion)
initially a flaccid areflexic paralysis/”spinal shock” (no reflexes, floppy paralysis)
classic UMN signs develop later (spastic paralysis, hypereflexes etc)

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9
Q

how does borwn sequard syndrome present?

A

ipsilateral motor level affected
ipsilateral dorsal column sensor level affected
contralateral spinothalamic sensory level affected

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10
Q

how does central cord syndrome generally present?

A
usually an elderly person after a fall
cant move hands
band of numbness/pins and needles over tops of hands
distal upper limb weakness
cape-like spinothalamic sensory loss
normal lower limbs and dorsal column
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11
Q

what usually causes a central cord syndrome?

A

hyperflexion or extension injury to already stenotic neck

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12
Q

why does ischaemia often occur in central cord syndrome?

A

pinching of medial spinal cord = most vulnerable part to ischaemia due to blood supply

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13
Q

describe sensory loss in central cord syndrome?

A

not true sensory loss as there isn’t complete loss of sensation everywhere below injury, only at that level

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14
Q

how does chronic spinal cord compression present?

A

same as acute but UMN signs predominate

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15
Q

most common areas of spine injured in trauma?

A

cervical

- most mobile

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16
Q

commonest cause of spinal tumours?

A

metastases

17
Q

what causes extradural tumours?

A

metastases (Lung, breast, kidney, prostate)

18
Q

what causes intradural tumour?

A

extramedullary (meningioma, schwannoma)

intramedullary (astrocytoma, ependymoma)

19
Q

how can tumours damage spinal cord?

A

can slowly compress

can cause acute compression by collapse or haemorrhage

20
Q

features of spinal stenosis?

A

osteophyte formation
bulging of IV discs
facet joint hypertrophy
subluxation

21
Q

causes of spinal cord infection?

A
epidural abscess (bloodborne staph, TB)
surgery or trauma
22
Q

what types of bleeding can cause spinal cord compression?

A

epidural
subdural
intramedullary

23
Q

causes of bleeding leading to spinal cord compression?

A

trauma
bleeding diatheses
anticoagulants
AVM

24
Q

how is spinal cord trauma managed?

A

immobilise (collar, board, blocks etc)
investigate (CT/X-ray, MRI)
decompress and stabilise (surgery, traction, external fixation)
methylprednisolone (acute inflammation decompression but generally not used due to side effects)

25
Q

how are metastatic tumours causing spinal cord compression managed?

A

IV dexamethasone in acute presentation
radiotherapy usually
chemotherapy if chemosensitive (multiple myeloma etc)
surgical decompression and stabilisation if needed but try to avoid

26
Q

how are primary spinal tumours managed?

A

mainly excised

27
Q

how is infection in the spine managed?

A

antimicrobial therapy
surgical drainage (always drain pus)
stabilisation where required

28
Q

how is haemorrhage in spinal canal managed?

A

reverse anticoagulation

surgical decompression

29
Q

how is degenerative disease managed?

A

surgical decompression +/- stabilisation