Spinal cord Flashcards

1
Q

a) Primary injury
b) Secondary injury

A

a) initial physical trauma
unlikely to be changed by surgery
b) Ongoing, after primary injury
the focus of nursing care

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2
Q

Classification of SCI 3

A

Level of injury
Mechanism of injury
Degree of injury

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3
Q

Spinal cord name
form top to the bottom

A

Cervical
Thoracic
Lumber
Sacral

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4
Q

a)cervical damage ?
b) thoracic,lumber,sacral damage?

A

a) Tetraplegia
C1-T1 Level of injury
Involvement of all 4 extremities
b)Paraplegia
Below T2
Lower extremities paralyzed

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5
Q

Which part is parakysis
a) C4 injury?
b) C6 injury?

A

a) complete below the neck
b) partial hand,arm and lower body

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6
Q

Which part is parakysis
a) T6 injury?
b) L1 injury?

A

a) below the chest
b) below the waist

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7
Q

Mechanism
Flexion

A

Force comes anteriorly
posterior ligaments rupture
Neurogic deficts
Head snaps forwad (cervical)
Snapping at waist (thoracic)
Herniated disl (lumber)

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8
Q

Extension

A

Forse comes posteriorly
Disruption of anterior ligament
Tension or shearing of involved tissue with potential for fracture injury

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9
Q

Flexion-Rotation

A

Force comes from the side Spinalligaments torn
Neck/spine twisitng
Often happen inmotor vehicle crashes where vehicle is hit from the side
Also occur in motorcycle crashes

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10
Q

Vertical Compression

A

Force comes from top or bottom
Wedge fracture
Fractures of vertebral(shattered)
Rupture of supporting ligaments d/t bone gragments

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11
Q

Simple fracture

A

Singular break/fracture
Aligment(stright line) generally remains intact

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12
Q

Dislocation

A

Over vertebra overrides another
Cord becomes ischenic

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13
Q

Anterior cord syndrome
a) Typically results from?
b) what effected?

A

a) flexion injury
b) Motor paralysis
pain
temperature sensation
Sensations of touch, position, vibration

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14
Q

Central cord syndrome
a) Damage to?s
b) what effected?

A

a) central cord
Most commonly cervical region
Neck hyperextension
More common in older adult
b) Motor weakness
altered sensation in upper extremities
Lower extremities usually not affected

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15
Q

Brown-Sequard Syndrome
a) damage to?
b) what effected?

A

a) half the cord
by penetrating injury
Loss of motor function + pressure, position, vibration sense on same side of body
Loss of light touch, pain, temperature sensation on opposite side

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16
Q

a) Conus Medullaris Syndrome
b) Cauda Equina Syndrome

A

a) lowest portion of cord
Conus Medullaris Syndrome
Pain is uncommon
Areflexic bladder and bowel
b) lumbar.sacral nerve roots
Areflexic bladder and bowel
Severe, radicular, asymmetric pain
Clean intermittent catheterization

17
Q

Clean intermittent catheterization
a) how many times?
b) Urine residuals less than?

A

a) 4-6/day
b) 500mL
No prophylactic antibiotics
Clean technique – no sterile procedure

18
Q

Emergency Management
Prehospital
If suspected SCI, always suspect head injury as well

A

Ensure patent airway
No head-tilt, chin lift, no neck hyperextension
keep SaO2 > 90%
Stabilize/immobilize cervical spine
Establish IV access x 2 / NS or LR

19
Q

Immobilization of spine
a) Maintain what poosition?
b) Reduction with skeletal traction
c) Halo vest

A

a) neutral position
b) for a day, maybe before surgery
c) for a longer time
Spinal immobilization should not affect resuscitation

20
Q

After admitted hospiral
Respiratory
a) above C3
b) below C4

A

a) absence of the ability to breathe independently
-require ventilator
b) hypoventilation
-administer O2
-aggressive chest physiotherapy

21
Q

Cardiovascular
a) Any injury above what dec/absent cardiac system
b) What HR is a concern?
c) Any vagal stimulation can cause what?
d) what is a treatment goal?

A

a) T6
b) Bradycardia
c) cardiac arrest
d) constrict vessels w/vasopressors

22
Q

Urinary System
a) what immediately occurs?
b) acute phase? Chlonic phase?

A

a) urinary retention
b) catheter but want to remove ASAP
colonic intermittent catheter or surgery(bladder neck revision)

23
Q

GI System
a) any injury above what causes hypomobility?
b) what concern?

A

a) T5
b) paralytic ileus and gastric distension
NG tube to intermittent suction

24
Q

Neurogenic shock
a) what part is caused?
b) s/s?
c) TX?

A

a) T6 or higher SCI
b) Peripheral Vasodilation
Hypotension
Bradycardia
dec CO
c) Vasopressor(norepinephrine, phenylephrine, or dopamine)
Blood pressure will not be restored by fluid infusion alone

25
Q

Autonomic Dysreflexia
a) what part causesd it?
b) s/s?

A

a) level at or above T6
distended bladder is the most common cause
b) masive Hypertension
Bradycardia
Blurred vision
Throbbing headache
Flushing of skin above level of injury
-Especially face, neck, shoulders
Anxiety and nausea

26
Q

Autonomic Dysreflexia
Treatment
a) first?
b) then?

A

a)elevate head of bed 45 degrees or sit patient upright
Super important!!!
b) Immediate catheterization to relieve bladder distension
If indwelling urinary catheter present, look for kinks, constrictions, plugs – irrigate if suspected
Check for stool impaction

27
Q

The nurse is caring for a patient admitted with a spinal cord injury after a motor vehicle accident. The patient has a complete loss of motor, sensory, and reflex activity below the injury level. The nurse recognizes this condition as:
central cord syndrome.
spinal shock syndrome.
anterior cord syndrome.
Brown-Séquard syndrome.

A

spinal shock syndrome.
Rationale:
About 50% of people with acute spinal cord injury develop spinal shock, a temporary loss of reflexes,sensation, and motor activity. Central cord syndrome is manifested by motor and sensory loss greater in the upper extremities than the lower extremities. Anterior cord syndrome results in motor and sensory loss but not loss of reflexes. Brown-Séquard syndrome is characterized by ipsilateral loss of motor function and contralateral loss of sensory function.

28
Q

Which assessment finding would the nurse interpret as a manifestation of neurogenic shock in a patient with acute spinal cord injury?
Bradycardia
Hypertension
Neurogenic spasticity
Bounding pedal pulses

A

Bradycardia
Rationale:
Neurogenic shock is caused by the loss of vasomotor tone after injury and is characterized by bradycardia and hypotension. Loss of sympathetic innervation causes peripheral vasodilation, venous pooling, and decreased cardiac output. Thus hypertension, neurogenic spasticity, and bounding pedal pulses are not seen in neurogenic shock.