Spasticity assessment and management Flashcards

1
Q

what would you include in the subjective assessment regarding patient’s condition?

A
  • impact on patient e.g., pain, sleep, function
  • frequency and severity
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2
Q

what factors would you check for and ask about in the subjective assessment?

A
  • any aggravating or easing factors
  • check for exacerbating factors
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3
Q

what details do you need to know about medication?

A
  • type
  • frequency
  • effectiveness
    -side effects
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4
Q

what should you look at regarding medical history?

A
  • previous spasticity management
  • was this effective?
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5
Q

what are the 6 exacerbating factors?

A
  • pain/ discomfort
  • infection e.g., UTI, chest infection
  • constipation
  • poor postural management
  • in growing toenail
  • pressure sores
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6
Q

what are the 9 assessments you would complete in an objective assessment?

A
  • posture
  • AROM/PROM
  • strength
  • sensation
  • co-ordination
  • reflexes
  • tone
  • balance
  • function
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7
Q

what is the main scale used for assessment of spasticity?

A
  • modified Ashworth scale
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8
Q

what is the MAS scores meaning?

A

0= no increase in muscle tone
1= slight increase in tone, manifested by catch and release at the end of ROM
1+= slight increase in tone, manifested by a catch, followed by minimal resistance throughout the remainder of range
2= marked increase in tone throughout most of range, still easily moved
3= considerable increase in tone, passive movement difficult
4= affects part(s) rigid in flexion/ extension

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9
Q

how do you test spasticity?

A
  • inner to outer range
  • assess slowly at first to assess PROM available
  • then move limb more quickly to assess spasticity
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10
Q

what is the other scale used to measure spasticity? when it is commonly used?

A
  • modified Tardieu scale
  • commonly used in children with spasticity- type cerebral palsy
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11
Q

what does the MTS involve?

A
  • ankle moved into dorsiflexion
  • knee moved into extension
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12
Q

what does R1 and R2 represent in the MTS?

A

R1= angle of muscle response (catch) as joint is moved at the fastest velocity possible
R2= angle of muscle response (end range) at the slowest velocity possible

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13
Q

what does the difference between R1 and R2 indicate? what does a large difference show?

A
  • difference between R1 and R2 indicates the relative contribution of spasticity versus contracture
  • large difference shows more spasticity whereas small difference shows contracture
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14
Q

what are the 9 contra- indications and cautions for testing spasticity?

A
  • haemophilia
  • osteoporosis
  • fracture
  • joint instability
  • severe pain
  • osteomyelitis
  • acute joint infection
  • DVT
  • flaccidity
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15
Q

what are the two things that treating spasticity reduces?

A
  • reduces pain
  • reduces carer burden
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16
Q

what are the two factors that treating spasticity improves?

A
  • improves functional activity e.g., gait, transfers, UL function
  • improves passive activity e.g., improve ROM for care tasks
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17
Q

what does treating spasticity help to maintain? (3)

A
  • maintains skin hygiene/ integrity
  • maintains seating and positioning
  • maintains current ROM/ activity status
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18
Q

what does treating spasticity support?

A
  • supports other interventions e.g., physiotherapy/ orthotics with the benefits it brings
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18
Q

how does spasticity relate to the ICF model level of impairment? (3 problems)

A
  • muscle spasms
  • abnormal trunk and limb posture
  • pain
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19
Q

what is the effect of muscle spasms? (3)

A
  • pain
  • difficulty with seating and posture
  • fatigue
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20
Q

what is the effect of abnormal trunk and limb posture? (3)

A
  • contractures
  • limb deformity
  • pressure ulcers/ other tissue viability problems
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21
Q

what is the effect of pain from spasticity? (2)

A
  • distress and low mood
  • poor sleep patterns
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22
Q

what are the two problems that arise on the activity level of ICF regarding spasticity?

A
  • loss of active function
  • loss of passive function
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23
Q

what effect does active function loss have? (3)

A
  • reduced mobility and dexterity
  • difficulty with sexual intercourse
  • difficulty with continence
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24
Q

what effect does passive function loss have? (3)

A
  • difficulty with care and hygiene
  • increased carer burden
  • difficulty with wheelchair seating or bed positioning
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25
Q

how does the participation level of ICF relate to spasticity? (1)

A
  • impact of muscle spasms, loss of active function, etc
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26
Q

what are the 4 ways that lack of participation due to spasticity effect the patient?

A
  • poor self- esteem/ self- image
  • reduced social interaction
  • impact on family relationships
  • impact on work
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27
Q

what are the three factors that treatment should be focused on?

A
  • focused on a goal
  • focused on independent functions
  • focused on improvement or maintenance
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28
Q

when would you not treat spasticity?

A
  • when extensor activity in lower limbs may be helpful for transfers so reducing spasticity may cause a loss of function for the patient
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29
Q

what should you discuss with the patient before treating spasticity? what should you explain?

A
  • discuss the balance between the benefits and harms of treating spasticity
  • explain that some people use their spasticity to maintain their posture and ability to stand, walk or transfer in which muscle relaxants will adversely affect this
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30
Q

what is spasticity useful for and why would you not treat in this case? (2)

A
  • may be useful for function or circulation
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31
Q

what is the medication used to treat focal/ multi- focal spasticity?

A
  • botulinum toxin
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32
Q

is there evidence of botulinum toxin effect?

A
  • evidence that it is safe and effective for treating upper and lower limb spasticity for both active and passive goals
33
Q

what effect does botulinum toxin have?

A
  • only has a temporary effect
34
Q

how does botulinum toxin work?

A
  • works by blocking the vesicle- dependent release of acetylcholine and other neurotransmitters from the presynaptic nerve terminal at the neuromuscular junction
35
Q

what does botulinum toxin result in?

A
  • results in partial and temporary muscle weakness in the treated muscles
36
Q

what is suggested before BoNT- A is used? (3)

A
  • all aggravating factors have been addressed
  • an appropriate physical management programme is in place
  • suitable programme of ongoing appropriate management is planned
37
Q

what should all botulinum toxin injections be accompanied by?

A
  • formals assessment outcome
38
Q

what review should the therapist do after the injection? when should this be done?

A
  • therapist review in 7-14 days for assessment
  • and if necessary orthotics/ splinting
39
Q

when do the MDT review the patient after the injections?

A
  • MDT review at 4-6 weeks to assess effect and patient status
  • also review at 3-4 months to plan future management
40
Q

what does follow up MDT review after injection vary due to? (3)

A
  • re- injection intervals
  • trajectory
  • types of goals
41
Q

what is used to treat regional spasticity?

A
  • nerve blocks
42
Q

what is an example of a nerve block?

43
Q

what is phenol?

A
  • long- acting, local neurolytic agent which denatures protein causing tissues necrosis
44
Q

what is the effect of phenol dependent on? (2)

A
  • dependent on volume and concentration
45
Q

what are the potential side effects of phenol?

A
  • sensory symptoms e.g., pain
46
Q

what is intrathecal baclofen?

A
  • manages severe spasticity of cerebral or spinal origin
  • reduces muscle hyperactivity
47
Q

do intrathecal baclofen have more side effects than oral medication?

A
  • intrathecal baclofen has less CNS side effects than oral medications e.g., fatigue
48
Q

what do patients receive before implementation of intrathecal baclofen?

A
  • patients receive a test dose so patient and clinician can evaluate potential effects
49
Q

how is intrathecal baclofen administered?

A
  • medication delivered directly to intrathecal space via implanted pump
50
Q

what needs to happen to the pump of intrathecal baclofen regularly?

A
  • needs to be refilled
51
Q

what are the two many surgery options?

A
  • selective dorsal rhizotomy
  • orthopaedic surgery
52
Q

what does SDR stand for? what is it used for?

A
  • selective dorsal rhizotomy
  • used to reduce spasticity in children with cerebral palsy with benefit to impairment and function
53
Q

what does orthopaedic surgeries involve? (3)

A

-muscle lengthening
- tendon transfers
- multi- level surgery

54
Q

what is the response to treatment?

A
  • variable
  • takes time to optimise
55
Q

what do you need to regularly do with the management plan?

A
  • reassess and review chosen measures
  • adequate follow up is essential to ensure treatment meets the needs and goals of the individual patient
56
Q

what should you consider when handling a patient?

A
  • consider patient’s position
  • comfort
  • support
  • surface area
57
Q

what should the therapist’s hand be when handling patient? how should the therapist be?

A
  • should have warm hands
  • therapist should be calm
58
Q

how do you handle the limb?

A
  • cradle the limb with the whole han
59
Q

what speed should you complete handling in?

A
  • slow supportive handling to try and reduce spasticity
60
Q

what should individuals at risk of contracture/ loss of joint range receive? what does it provide?

A
  • receive interventions e.g., splints, casts or positioning
  • to provide passive stretch of sufficient duration and intensity where there is stull potential for reversibility
61
Q

what does postural management/ 24 hour positioning enhance? (4)

A
  • comfort
  • respiration
  • communication
  • visual ability
62
Q

what does postural management reduce? (3)

A
  • pressure sores
  • adaptive muscle shortening
  • pain
63
Q

what are the three factors we need to consider in postural management/ 24 hour positioning?

A
  • positions across the day e.g., for sleep, for eating
  • is position for rest or activity?
  • 24 hour approach required
64
Q

when is splinting/ orthotics used?

A
  • for function e.g., AFOs for standing/ walking
  • at rest e.g., resting hand splint
65
Q

when is splinting/ orthotics effective?

A
  • improves effectiveness of chemo denervation (botulinum toxin injections)
66
Q

what does the COT & ACPIN guidelines cover?

A
  • cover splinting (thermoplastic) and (serial) casting fibreglass casting tape or plaster of paris
67
Q

what did the COT and ACPIN guidelines suggest about ankle casts?

A
  • ankles casts are used with botulinum toxin (in people with stroke and ABI) to improve ROM when there is clinically significant spasticity
68
Q

what do ankle splints prevent?

A
  • prevents loss of ROM at the ankle when positioning at PG (for people with stroke)
69
Q

should splints be used for the upper limb?

A
  • for upper limb, splints should not be used routinely to prevent loss of ROM in the writs/ hand (for people with stroke and ABI), but maybe beneficial in some cases
70
Q

what does weight bearing help with?

A
  • helps normalise tone
71
Q

what should weight bearing be considered for?

A
  • considered for both upper and lower limb spasticity
72
Q

what is a good option for lower limb spasticity?

A
  • standing frames can be a good option for lower limb spasticity as well as providing other benefits
73
Q

how do you improve activity performance and motor control?

A
  • improve activity performance by task practice training
  • repetitive practice
74
Q

what is functions electrical stimulation used for? (2)

A
  • used to activate muscles
  • used for upper limb/ lower limb
75
Q

what is FES most effective for?

A
  • most effective for people with UMNLs
76
Q

what is there good evidence for regarding FES?

A
  • good evidence base for effectiveness of addressing dropped foot
77
Q

what do the National Stroke Guidelines advise about treatment for spasticity?

A
  • people with spasticity in the upper or lower limbs after stroke should not be treated with electrical stimulation
78
Q

what should people with spasticity in wrist/ fingers be considered for?

A
  • considered for ES after injection to maintain ROM and/ or to provide regular stretching as an adjunct to splinting or when splinting is not tolerated
79
Q

what are some outcome measures? (7)

A
  • pain scale
  • spasm frequency scale
  • ArmA (arm activity measure)
  • LegA (leg activity measure)
  • AARS (associated reaction rating scale)
  • SQoL- 6D (spasticity related quality of life tool)
  • GAS (goal attainment scaling)
80
Q

what do objective measures alone fail to identify?

A
  • fail to identify the complexity of spasticity