Spasticity Flashcards

1
Q

what is spasticity?

A
  • type of neurological driven muscle stiffness
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2
Q

is there a clear definition of spasticity?

A
  • inconsistently defined
  • constantly adapting
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3
Q

what is a definition of spasticity regarding motor movement?

A
  • motor disorder characterised by a velocity- dependent increase in tonic stretch reflexes (muscle tone) with exaggerated jerks
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4
Q

what does spasticity result from?

A
  • hyperexcitability of the stretch reflex
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5
Q

what is the definition of spasticity regarding sensory aspects?

A
  • disordered sensori- motor control, resulting from an upper motor neuron lesion
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6
Q

what does spasticity present with?

A
  • intermittent or sustained involuntary activation of muscles
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7
Q

what does spasticity increase?

A
  • increases resistance to movement
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8
Q

what are the two categories that cause spasticity?

A
  • neural (hypertonia)
  • non- neural (biomechanical)
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9
Q

what are the 3 further divisions of neural hypertonia?

A
  • spasticity
  • rigidity
  • dystonia
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10
Q

what are the two divisions of non- neural cause?

A
  • soft tissue contracture
  • thixotropy
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11
Q

what is soft tissue contracture?

A
  • shortening of the muscle, tendon, joint capsule
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12
Q

what is rigidity? what does it affect?

A
  • neurological muscle stiffness or inflexible muscles
  • affects antagonist and agonist
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13
Q

what is rigidity not dependent on?

A
  • not velocity dependent
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14
Q

what are the two main types of rigidity?

A
  • cogwheel
  • lead pipe
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15
Q

what is thixotropy?

A
  • stiffness is impermanent because it forms only when the muscle is stationary for some time and is reduced upon active or passive movement
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16
Q

what increases with time and what is reduced in thixotropy?

A
  • resistance to movement increases with time
  • reduced by movement
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17
Q

what is dystonia?

A
  • movement disorder characterised by sustained or intermittent muscle contractions causing abnormal, often repetitive, movements, postures or both
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18
Q

what are dystonic movements typically?

A
  • typically patterned, twisting, and may be tremulous
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19
Q

what is dystonia often initiated or worsened by?

A
  • initiated/ worsened by voluntary action and associated with overflow muscle activation
20
Q

what is an upper motor neuron lesion?

A
  • designated as any damage to the motor neurons that reside above nuclei of cranial nerves of the anterior horn cells of the spinal cord
21
Q

what are positive signs?

A
  • addition to behaviour
22
Q

what are negative signs?

A
  • loss of behaviour
23
Q

what are the six positive signs of spasticity?

A
  • positive babinski
  • spasticity
  • clonus
  • spasms
  • hyperreflexia
  • associated reactions
24
Q

what are the three negative signs of spasticity?

A
  • weakness
  • loss of dexterity
  • fatigue
25
Q

what are three examples of upper motor neuron lesions?

A
  • stroke
  • traumatic brain injury
  • MS
26
Q

what are the three examples of lower motor neuron lesions?

A
  • guillain barre syndrome
  • polio
  • spinal muscular atrophy
27
Q

what condition affects upper motor neuron and lower motor neuron?

A
  • motor neuron disease (MND)
28
Q

what is clonus?

A
  • rhythmic oscillating stretch reflex
29
Q

what is clonus potentially linked to?

A
  • linked to hyperreflexia
30
Q

how many oscillations occur in clonus ?

A
  • 5 to 10 oscillations
31
Q

what is fatiguing clonus?

A
  • where the patient is unable to stop spasms by themselves
  • require the stretch to be stopped
32
Q

what is non- fatiguing clonus?

A
  • stops by itself
33
Q

what are associated reactions?

A
  • effort- dependent phenomenon causing an involuntary increase in upper limb muscle tone, with awkward and uncomfortable postures
34
Q

what are associated reactions useful indicatives of?

A
  • indicative of effort
35
Q

how prevalent is spasticity in stroke?

36
Q

how prevalent is spasticity in severe brain injury?

37
Q

how prevalent is spasticity in severe multiple sclerosis?

38
Q

what does presentation of spasticity vary with? (3)

A
  • size of lesion
  • location of lesion
  • age of lesion
39
Q

what are the three common lower limb pattern?

A
  • focal spasticity with hyperextension of the hallux
  • multifocal spasticity involving the lower limb
  • regional spasticity involving the left leg and arm
40
Q

what are the common upper limb patterns?

A
  • flexion/ adduction/ medial rotation of GH joint
  • elbow flexion
  • pronation
  • wrist flexion
  • finger flexion
41
Q

what is the pathophysiology of spasticity?

A
  • a lesion to the CNS causes the loss of descending inhibition and disrupts the balance between excitatory and inhibitory supraspinal control, leading to hyperexcitability or reflexes
42
Q

what are upper motor neuron lesion symptoms largely caused by? what contributes?

A
  • parapryradimal fibre dysfunction
  • only small input from lateral corticospinal tract
43
Q

what is the main tract that inhibits spinal reflex activity? where is it?

A
  • main tract that inhibits spinal reflex activity is the dorsal reticulospinal tract
  • runs very close to the lateral corticospinal tract
44
Q

what does a single lesion frequently affect?

A
  • frequently effects both tracts
  • produces a picture reflecting the combined lesion
45
Q

what tract makes a small contribution and what tract produces most of the symptoms?

A
  • corticospinal tract lesion makes a small contribution but the parapyramidal dorsal reticulospinal tract produces most of the symptoms & signs
46
Q

what is spasticity pathophysiology also potentially due to? what does this explain?

A
  • spasticity due to neuroplastic changes within the CNS
  • explains some delay in the onset of spasticity
47
Q

why does hyperexcitability occur?

A
  • reflex arc lost connection to the brain
  • muscle spindle senses lengthening so triggers another nerve to contract that muscle