Small ruminants 2 Flashcards

1
Q

Peste des petits ruminants (PPR) what is it, transmission and signs

A
emergency disease in sheep
- Morbillivirus, like Rinderpest
○ Plan to eradicate worldwide
- Direct transmission from excretions
- Spread by movement of infected animals
- Signs
○ Fever
○ Oral erosions
○ Bronchopneumonia
○ Diarrhoea
○ Abortion
○ 10% case mortality rate
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2
Q

Maedi-Visna what is it, what does it cause, type of virus, transmission, 2 main types of clinical signs and daignosis

A

emergency animal disease in sheep
- Pneumonia and a wasting encephalitis, long incubation
- Small ruminant lentivirus, organ trophisms, slow viruses
- Direct transmission, maternally
- Clinical signs varied:
○ ‘Maedi’ dyspnoea, high mortality
○ ‘Visna’ subtle ataxia -> paresis
○ Indurative mastitis, arthritis
- Heavy lungs, diagnose with histo and virology

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3
Q

Sheep pox what is it, where is it found, type of virus, transmission and diagnosis

A
emergency animal disease in sheep 
- Middle East – severe losses expected in Australia
- Capripoxes host specific, resistant
- Aerosol transmission from papule stage
○ Lungs, skin, gut
- DDx skin diseases and pneumonias
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4
Q

Rift valley fever what is it, what type of virus, environment, transmission, importance, mortality rate and clinical signs

A
  • Bunyavirus, epidemics in wet seasons
  • Vectors vary, transovarial transmission
  • Zoonoses, severe human consequences
    ○ Exposure to infected tissues
  • Age dependent mortality rate
    ○ Lambs near 100%, adults lower
  • Signs
    ○ Abortion storms common
    ○ Fever, bloody diarrhoea, jaundice
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5
Q

Sheep scab what is it, lifecycle, clinical signs and is it in australia

A
emergency animal disease - sheep
- Psoroptes ovis mite
- Direct life cycle, short, fecund
○ Severe disease in 4-6 weeks
○ Dormancy at base of horns
- Rosette skin lesions, fleece loss
- Eradicated from Australia in 19th century
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6
Q

Ovine pulmonary adenomatosis what is it, what causes it, where found, what result in

A
emergency animal disease - sheep 
- Retrovirus – contagious neoplasm
- Australia, NZ, Iceland free
- Slow disease, progressive pneumonia
○ Lungs full of fluid
○ Wheel barrow’ test
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7
Q

Ovine enzootic abortion what is it, caused by, where found and main importance

A
emergency animal disease
- Chlamydia abortus
○ Abortion per se seen uncommonly in sheep in Australia
- Not reported in Aust.
- Zoonosis
○ Basque country
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8
Q

Brucella melitensis what is it, how important, what leads to in different species/sexes

A
emergency animal disease
- Commonest zoonosis in the world
- Ewes and does
○ Mastitis and abortion
- Rams and bucks
○ Epidydimitis and infertility
- Humans
○ Chronic disease, fever, aches
○ Intense ‘One Health’ vaccination campaigns
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9
Q

Schmallenberg virus what is it, where seen, transmission and results

A
emergency animal disease
- First seen in 2011 in Europe
○ All ruminant species
○ Simbu group of Orthobunyaviruses
§ includes Akabane
- Vector spread
○ Culicoides obsoletus complex
- Short viraemia and illness
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10
Q

What are the 6 important things to consider with hobby farms

A
  1. Their objectives
    ○ Just doing it for passion
    ○ Want to earn actual money, in for a profit? Higher stocking rate
  2. Knowledge? (husbandry and disease)
  3. Availability?
    ○ Treatment plan around how long spend on the farm
  4. Enthusiasm
  5. Write things down for clients -> simple treatment plan write it down
  6. Have good explanations/reasons for your recommended treatments (competing with salesmen, poor local knowledge, etc.)
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11
Q

What need to consider with facilities within a hobby farm

A

Safety is the most important -> to client, you and animal
o Restraint (want a good head stall, nose grips and tail jack)
○ Does the neighbor next door have better yards to use?
○ NO ASSUMPTIONS OF ABILITY AND KNOWLEDGE WITH HANDLING
o Operator safety
o Experience/ability of assistants
o Want crush, escape routes, less people to be trampled, backing gates
o Proper exit from crush -> possible exit from the raceway BEFORE crush and AFTER crush, no straight into run into truck, no sharp corners

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12
Q

What are the 4 main considerations when giving advise about animals on hobby farms and things within

A

1) what stock are easier/harder to mange
2) husbandry
- what must happen and when
- what can be more flexible
- mange potential inbreeding
- stocking rate
- condition scoring
- supplementary feed
3) flock animals need company - welfare
4) treatment and disease management consideration - individuals and flock/herd

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13
Q

In terms of knowing what stock are easier/harder to manage for hobby farmers what are

A

○ Dry stock, weather for wool and meat production - easier
○ Pregnant and lactating generally more susceptible to diseases - harder
○ Management calendar may be needed

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14
Q

What are the 5 main husbandry things that need to be done on a sheep hobby farm

A

1) castration (timing)
2) tagging/NLIS - all need
3) tail-docking - rubber rings
4) kid disbudding - dehorning a goat
5) mulesing/clips (maybe advise better sheep bred)

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15
Q

In terms of castration on a hobby farm how should be done and what is important to consider

A

□ Surgical castration +/- local anesthetic up to 6 months old without ‘much’ hemostasis
® Just twist the spermatic cord to ensure proper hemostasis
◊ If not confident just use emasculator
□ Beware inexperienced owner using rubber rings in bigger lambs/calves
® Can only be used in calves between 2days and 2 weeks of age

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16
Q

Tail docking sheep on hobby farms what use, what age

A
  • rubber rings
    □ 2-8 weeks old with rubber rings
    □ IF older than gas knife with local anesthetic
    □ OVER 6 months of age only tail docked by vet with appropriate anaesthesia/analgesia
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17
Q

Mulesing/clips on hobby farms what need to advise

A

can you advise about a better sheep breed?)
□ May need to get contractors
□ Non-merino bred of sheep won’t require mulesing
□ Insecticides to help reduce fly numbers

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18
Q

what are 6 important flock conditions of sheep need to talk to hobby farms about

A

1) dystocia
2) worms
3) Lice
4) flies
5) footrot
6) OJD

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19
Q

Dystocia for hobby farmers what need to talk to owners about

A
  • Manage dam bodyweight -> appropriate condition score and nutrition
    • Electric fencing grazing a smaller area can help
  • Be able to describe stages of parturition
  • Know when to intervene
    • Heifers 2 hours then intervene
  • More sheep caesareans
    • Service is available in the clinic?
  • Prolapse repair
  • Advice about lamb survival, orphan management/rearing
    • Neonates lectures
    • Good fact sheet
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20
Q

worms for hobby farmers what do you do the same, what are the challenges, what are the shortcuts and what need to be aware of

A
  • What do you do the same?
    • Drenching - same treatments
    • Fresh faeces in the ground 10 x for bulk cell count - monitoring
  • What are the challenges?
    • Won’t need as much volume for drench
    • Rotating paddocks may or may not be possible
  • What are the ‘shortcuts’/simplifications/lifesavers?
    • Before and after testing for individual animals -> see whether working and if need
  • Beware overstocked pastures
  • Might need to resort to capsules more often
  • Wethers MUCH easier than lambing ewes
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21
Q

Lice for hobby farmers what is bad, what needed for eradication and what may be easier

A
  • Major welfare issue
  • Harder in limited facilities
    • Backliners easy (so are contract dippers)
    § Explaining long vs short wool
  • Usual rules to maximize eradication chances
    • Address biosecurity - PROPER FENCES
    • Chemical ‘quarantine periods’ and lambing
  • Easier to change over whole flock (beware buying back in)
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22
Q

Flies for hobby farms how to prevent, what aware of and what is important

A
- Can't prevent
• Mules? Clip?, specific breed?
- Long-acting IGRs are great
- Beware of overt strike 
- How to manage individual cases 
- Time crutching and shearing to maximum effect
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23
Q

Footrot in hobby farms what is the important thing to address and things to do

A
  • Biosecurity
  • Hysteria
    • Unreasonable hysteria often accompanies footrot
    • Small hobby farms might just be best off selling all animals and starting again (apply usual biosecurity/quarantine principles)
    • Need to understand epidemiology properly
    • Eradication is achievable on their flock if the process is worthwhile
  • Use antibiotics rather than eradication?
    • Cure rate only good enough in dry conditions (summer or shearing shed)
  • 7 day rule still applies
  • just cull and start again
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24
Q

OJD - ovine johne’s disease what is the important thing to consider with hobby farms

A
  • Operator safety FOR THE VACCINES - terrible lesions, if not competent then do it yourself
  • Probably will need to source small numbers of doses from a bigger farmer because minimum pack size is 100 doses and its expensive
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25
Q

Spotting the sick alpaca how is it done and how to care for sick alpaca

A
  • Alpacas are stoical, they try not to stand out
  • Changes in behaviour - get to know each one’s nature, hierarchical position
  • Body score your animals at every opportunity, and have your owners do the same
  • By the time that the owner thinks that something is wrong, it probably is
    sick alpaca
  • small yards under cover, companion animal for the sick one
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26
Q

Alpaca clinical examination what to be done, rectal temp, HR normal

A
- Follow an ordered and thorough routine
• Rectal temp 38.5c - 39.5c
• Heart rate 60 bpm
• Auscultate chest
• Auscultate C1 at last left intercostal space
• Check mucous membrane colour, CRT, inspect oral cavity
• Palpate superficial lymph nodes
• Perform Body Condition Score
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27
Q

Ophthalmic examination for alpaca what need, drug and differences with dogs/cat

A
  • Find a dark area
  • Use magnification
  • Tropicamide (Mydriacyl) may take 20-45 minutes to effect dilation, so plan for that
  • Differences
    ○ Large pupillary riff (corpora nigra)
    ○ Iris colour dark brown
    § light brown -­ non-­ pigmented (“blue”)
    ○ Fundus lacks tapetum
    ○ Retinal blood vessel pattern-­ one pair dorsally, two laterally, one ventrally
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28
Q

what are 4 common congenital ocular conditions

A
  • Persistent pupillary membranes
  • Ectropion (esp llamas)
  • Nasolacrimal puncta agenesis
  • “Blue eye” deafness in white animals
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29
Q

Aural examination for alpaca what is important, what equipment needed

A
  • The vertical ear canal becomes very narrow near the transition to horizontal
  • Sedation is necessary for a thorough examination
  • Small rigid endoscopes are very useful to examine the lower ear & eardrum
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30
Q

What are some common diagnostic procedures done on alpacas and some pitfalls

A
  • Blood analysis
  • C1 fluid analysis - pH 6.5 - 7.5, multiple forms of protozoa
  • Paracentesis
  • Liver biopsy – Ultrasound guided, laparotomy
  • Radiography
  • Ultrasound – reproductive tract
  • Cytology and biopsy – particularly useful for dermatological disease
    Diagnostic pitfalls
  • In house clinical pathology – biochemistry is reliable but red blood cell shape renders benchtop cell counters useless
    ○ Need to send away
  • Peritoneal Tap - avoid midline fat
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31
Q

Landmakrs for venepuncture on a alpaca and how to perform

A
  • The alpaca jugular is quite superficial but harder to palpate as don’t become visible (within muscle)
  • The ventral process prevents lateral movement
    ○ Jugular farrow is medial to the ventral process
  • 30 degrees at the level of the process
  • Jugular or carotid??
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32
Q

Llamas vs alpacas with venepuncture

A
  • Jugular skin is very thick - cannot use a vacupuncture needle
  • Use an 18g needle
  • Preferably have the llama in a chute
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33
Q

what are the 2 main energy metabolism disorders of sick camelids

A

1) hyperglycaemia - driven by adrenalin
2) lipid metabolism - espeically in lacting and preganncy females
- hypoalbuminea, hepatic lipidosis

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34
Q

hyperglycaemia in alpaca what caused by, therefore what need to beware of and fluid movements associated

A

Hyperglycaemia driven by adrenalin, to a less extent cortisol, and peripheral insulin resistance
○ Crias are less so than adults – they have a higher insulin response and peripheral insulin sensitivity
○ Sick alpacas usually hyperglycaemic – beware of diagnosing diabetes mellitus -> could just be driven by adrenal when stressed
○ Fluid movements associated with persistent hyperglycaemia
§ Hyperglycaemia stimulates diuresis – primary method of lowering glucose
§ Dehydration causes relative hypernatremia and hyperosmolality worsens

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35
Q

Responding to a sick camelid what are some early signs of metabolism disorders and what to suspect/do

A
  • The early sign of energy metabolism disorders may simply be increased frequency of urination
  • Maintain hydration – “Normosol” (low sodium balanced electrolyte)
  • Suspect hypoalbuminaemia – give plasma!
    ○ If not doing well and done blood test, especially in CRIAS
  • Regular insulin ? – No protocol in place yet. See the work of Chris Cebra, OSU
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36
Q

in terms of injecting alpacas what should you do more commonly and how

A
  • Sterile technique
  • Most injections given subcutaneously
    ○ Less painful than intramuscular (will probably be kicked)
    § High on the thigh (avoid sciatic) if give IM but don’t do often
  • The skin of alpacas is quite tightly attached compared to other species
  • Use the skin in front of and behind the “shoulder blade” – there is very little “loose” skin on the camelid
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37
Q

What are 5 unlikely disorders of camels

A
  1. • Bloat -> generally don’t get
  2. • Footrot
  3. • Fly strike
  4. • Mastitis
  5. • Respiratory infections
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38
Q

What are 7 main likely diagnosis of alpacas

A
  1. some skin problems, often allergic
  2. occasional gastro enteric disorders
  3. occasional neurological upsets - pasture toxins generally
  4. occasional parasitic problems
  5. cancer likely in older animals
  6. Ear infection, foreign bodies - ear carriage will be down, may shake head.
    ○ Alpacas have narrow ears and may need sedation for adequate examination
  7. Weepy eyes, suspect grass seeds. May be hidden by third eyelid
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39
Q

what are the 8 main neurological diseases of alpaca

A
  1. Congenital abnormalities
  2. Pasture toxicities - perennial rye & phalaris - most common
  3. Snakebite
  4. Ixodes holocyclus
  5. Heat stress - high temp & humidity in combination
    ○ Heat cooling mechanisms -> cool grass or wet area to lay on to dissipate heat (only area without dense fibre)
    ○ THEREFORE - to treat create a wet area for them to lay on
  6. Polioencephalomalacia
  7. Hepatic encephalopathy - copper injection toxicity
  8. Meningitis, otitis media - not retrieved grass seed
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40
Q

Sporidesmin what is it, does it do for alpacas

A
  • Pithomyces chartarum - Facial eczema in sheep
  • Potent hepatotoxin
  • Spore counts useful in high risk areas
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41
Q

Perennial ryegrass staggers in alpaca how common, clinical signs, and greatest risk

A
  • Most common neurological problem in southern Australia
  • Early stages may be mild head tremor
  • Often progress to generalised tremor
  • Some familial groups seem predisposed
  • Some animals are left with permanent disability
  • Risk greatest January to June
42
Q

Skin disorders in alpacas how common, what presents as, causes and how to diagnose

A
  • Uncommon
  • Sunburn on ears and dorsal midline if closely shorn (occasional flystrike)
  • Allergies – generalised or localised – feet, nasal plane
  • Pemphigus foliaceous
  • Tumours eg mast cell
    perform punch biopsy under local and sedation to pursue a diagnosis
43
Q

Biting lice in alpacas most common species, where mainly found, lifecycle and transmission

A
  • Presence of Bovicola breviceps documented in Australian alpaca herds
    ○ Affects all camelids in South America
  • Most common base of tail, thorax, abdomen & upper limbs
    ○ Feed on scurf (“chewing lice”) -> need easily killed with injectables
  • Life cycle 2-5 weeks – adults live 30-50 days
    ○ Host specific for camelids
  • Transmission – close contact (e.g. mating) or grooming instruments
44
Q

Biting lice in alpacas diagnosis, control and treatment

A
  • Diagnosis – 1.0 – 1.5 mm long, photophobic so often hard to find, examine hairs for eggs attached at the base of the shaft
  • Control – no products registered – must be safe and avoid fleece residues & staining
    ○ Advisable to add wetting agents
  • Treatment - Spinosad (Extinosad® - Elanco) – a jetting fluid + surfactant at 17 day intervals was effective in our herd
    ○ Note – Off label use
    ○ The active ingredient kills adults and nymphs but not eggs
45
Q

neonatal/cria assessment what are the 2 main thing to assess

A
1) Assess maturity
○ Eruption of lower incisors - feel at least 2 teeth, if not then premature 
○ Floppy ears - premature 
○ Laxity of flexor tendons - premature 
2) Some congenital problems
1. Angular limb deformities
2. Umbilical hernia
3. Cardiac murmur - VSD common 
4. choanal atresia - obligate nasal breathers - will open mouth 
5. congential cataracts
46
Q

Choanal atresia what common in, what is it, results in, diagnosis and treatment

A

crias - congenital problems
○ Failure to establish a connection between the caudal nasal cavity and the pharynx
○ Usually have difficulty suckling from birth or cannot suckle if complete atresia
○ Air flow through nostrils absent (or minimal)
○ Diagnosis – instil radiographic contrast in nasal cavity and tilt skull ; attempt to pass stiff catheter into pharynx
§ Contrast does not progress to pharynx just into frontal sinus
○ Attempts to surgically repair usually causes horrible facial defects
○ Euthanasia usually considered the best option

47
Q

Congenital cataracts what common in, cause, presentation

A

crias - congential condition
○ Consequence of intra-uterine infection
○ BORN BLIND but generally clinical well -> as long as other animals around to follow can survive well

48
Q

Geriatric (old) camelids what is the main cause of death and most common

A
  • Neoplasia is not an uncommon cause of death

- Lymphosarcoma is a relatively common neoplasm, and may not involve superficial lymph nodes

49
Q

What are the most common medical condensations for alpaca

A
  • Skin disease - mange, atopy, insect worry
  • Endoparasites
  • Tooth root abscess
  • Ill-thrift
  • Hepatopathy - sporidesmin
50
Q

what are the main surgical conditions of alpaca

A
  • Uterine torsion/caesarean/prolapse/uterine flush
  • Castration
  • Surgical repair of dog attack
  • Corneal ulcers
  • Tooth root abscess
  • Bone sequestrum/ fracture repair
  • Drainage of abscess
51
Q

Following up a sick alpaca what is important to do and get the owner to do

A
  • Try to set an expected time line for recovery
    ○ Monitor the animals progress
  • Be prepared to reconfirm a diagnosis or look for a new one
  • Have the owner monitor body weight and body condition score
  • Also have the owner monitor rectal temperature if infection is involved
52
Q

Faecal egg counts in crias and weaners what is low, moderated, high and when treat for GIT count, nematodirus count and combines

A
  • Low when <90 egg per gram
  • Moderate when between 90-240 eggs per gram
  • High if > 390 eggs per gram
  • Treatment when
    ○ GIT Count > 390 eggs per gram
    ○ Nematodirus count > 90 eggs per gram
    ○ Combined count is >210 eggs per gram
53
Q

Faecal egg count in adult and tuis what is low, moderate, high and when is treatment needed for GIT count, nematodirus and combined

A
  • low when < 30 epg,
  • moderate between 30 - 75 epg,
  • high if > 120 epg.
  • Treatment is for these animals recommended only when
    ○ the GIT count is > 120 epg, or
    ○ the Nematodirus count is > 60 epg, or when
    ○ the GIN + Nematodirus count is > 75 epg.
54
Q

Reproduction vetarinry intervention during unpacking how common, what is normally needed and what need to avoid

A

○ Not common
○ Caesarean section RARE
○ Foetal realignment usually all that is necessary
○ Epidural and episiotomy (cut made at opening of vagina during childbirth) more commonly useful
○ Avoid cervical and vaginal trauma
○ Assume all birthing after 5pm will be a problem

55
Q

Periparturient problems in alpacas what are 6 and which is rare

A

1) RFM - oxytocin infection
2) Hypocalcemia - RARE, dilute 4 in 1 with saline before giving subcutaneous
3) Post-partum bleeding if tear uterine artery
4) Prolapsed uterus - excellent prognosis if attend promptly
5) Tears - rectovaginal as repair in others
6) Uterine torsion in last 2 weeks of gestation is common so if colic looking and meant to be giving birth but not

56
Q

Uterine torsion in alpacas when generally occur, which way, types, diagnosis and treatment

A

○ Up to 6 weeks before due date
○ Most are clockwise - gravid horn twists over non gravid horn
○ Post cervical or pre cervical
○ Vaginal palpation - rectal palpation (problematic due to rectal tears)
○ Correction - sedate and de torse or caesarian
§ When de torsing need to rotate neck as well so need 3 people instead of 2

57
Q

What are 6 main surgical procedures for alpacas

A
  1. • Orthopaedic
  2. • Abdominal
  3. • Tumour removal
  4. • Ear and eye procedures
  5. • Diagnostic radiology
  6. • Laparoscopic procedures
58
Q

Anaesthesia for alpacas what is used for induction and maintenance

A
- Induction
○ Xylazine/ Ketamine/ Butorphanol
○ Mask down cria
- Maintenance -
○ Intramuscular “top up”
○ “Triple Drip”
○ Gaseous anaesthesia
59
Q

Anaesthesia what to use for sick alpaca

A
  • Avoid Xylazine in sick animals
  • Diazepam 0.2 mg/kg I/v followed by Ketamine 2.2 mg/kg I/v
  • Can be mixed in the same syringe
  • Isoflurane is preferable to halothane
    ○ toxicity is documented with halothane
60
Q

Patient preparation for alpaca before anaesthesia what 4 things need to do

A

1) Starve 12 hours before elective procedures
2) Intubate where possible
○ Long bladed laryngoscope, rigid endoscope (8mm)
○ Retrograde guide system
○ HAVE NECK IN FULL EXTENSION
3) Have rostral tilt on head when not intubated
4) Keeping the fibre away
○ Clip as usual but use duct tape to direct fibres away from clipped area

61
Q

during alpaca anaesthesia what is important to monitor and recovery

A

Monitoring anaesthesia
- Normal resting heart rate 60 bpm
- Eye position not reliable when Ketamine used
- Jaw and limb movements are better indicators of depth
- Respiration - observe chest excursions and bag movement
- Saphenous artery pulse - medial to stifle
Recovery from anaesthesia
- Leave intubated as long as possible
- Return to sternal recumbency as soon as possible
○ Avoid perfusion/ventilation mismatch
- Support the head and neck

62
Q

Analgesia for alpaca how to know when in pain, what are the 3 common drugs to use and which use most commonly

A
  • Often start “tooth grinding” when in pain
    1) Butorphanol 0.1 - 0.5 mg/kg s/c
    2) Flunixin 1mg/kg s/c once daily for 5 days - MOST COMMON
    3) Meloxicam 0.2 mg/kg s/c once daily abdominal pain
63
Q

tooth root abscess in alpaca what present as, diagnosis and treatment

A
  • Present as swelling and hair loss around the jaw
  • Diagnosis -> radiograph of mandible, need to work out the exact tooth to extract
    Treatment -> tooth removal
  • Identify the correct tooth
  • Time consuming
  • Air driven burr
  • Dental papilla bleeds profusely
  • We tend to exhaust medical possibilities first - antibiotics
64
Q

Laparoscopy what are the 3 main reasons to do in an alpaca and what used for closure

A

○ Caesarian section
○ Uterine torsion correction
○ Exploratory (diagnostic) laparotomy
§ “Never let the linea alba stand between you and a diagnosis
Abdominal wall closure
- Routinely use 0 PDS for simple continuous closure of the linea alba, then 2/0 vicryl subcutaneously. Simple interrupted skin closure

65
Q

Orthopaedic disease in alpacas what is the main one, presents how old,

A

Bone cysts in growing camelids

  • Presented from 4 weeks to 14 months of age
  • Reported in metatarsus, metacarpus, femur, humerus, mandible
  • Present with lameness and pain on palpation of the affected bone
  • May or may not have a sinus tract
66
Q

Bone cysts in alpacas diagnosis and treatment

A

Diagnosis
- Radiograph showing typical characteristics of a sequestrum
- Cases documented so far involve only one discrete site (compared to hypovitaminosis D)
Orthopaedic surgery
- Surgical repair of fractures
- Plates are good!
- Transverse pins embedded in a fibreglass cast may be useful
- Correction of limb deformities

67
Q

What need to consider with single limb lameness in an alpaca

A
  • Always consider the possibility of the presence of a sequestrum in a young animal with a single limb lameness
  • The index of suspicion rises if there is a discharging sinus
  • Take a radiograph and if confirmed perform surgery – this is a surgical disease!
68
Q

Vitamin D deficiency in alpacas when occur, what is the most at risk group and exceptions to this rule

A
  • Animals growing rapidly during winter suffer the “double whammy” - high needs and decreasing day lengths
  • UV levels at low altitudes less than the altiplano
    The Most At Risk Group
  • Animals born in the current calendar year
  • This group have not been through a summer, were born into decreasing day lengths and have not had the opportunity to accumulate vitamin D
  • Are actively growing
    The exceptions to the rule
  • Occasional adults will present with lameness attributable to inadequate Vitamin D
  • Adults are considered to have higher levels than younger animals at any particular time of the year
69
Q

Vitamin D deficiency clinical signs and diagnosis in alpacas

A

Clinical signs
- “Proppy” gait
- Failure to keep up with the group
- Arched back
- “Walking on eggshells”
- Multiple limbs affected or shifting lameness
Making a diagnosis
- Measure 25-OH Vitamin D - expensive, not readily available
- Blood P - correlates with Vitamin D levels, cheap can be performed “in-house” - IF LOW GIVE VITAMIN D
- <1 mmol/L is consistent with hypovitaminosis D – screening test only

70
Q

Vitamin D deficiency prevention in alpacas, when to give/who to treat

A
  • 2000 IU Vitamin D per kg s/c late autumn and repeat in 8 weeks
  • Treat growing cria from one month of age
    ○ Some growth continues until 2 yrs old , so treat all alpacas under 3y.o.
  • Poor transplacental transfer, but treat all females due to give birth
  • Vitamin D levels are low in the milk of other species
    Some Vitamin D present in cured hay
71
Q

Alpaca nutrition how much to feed and length of grass

A
  • 1.5% body weight dry matter as maintenance diet.
  • Remember , growing , late pregnant and lactating females need more. 2-2.5%
  • > 4 cm best to keep fore stomachs functioning
72
Q

Livestock emergencies what are the 6 main ones

A

1) Burns – bush & grass fires
2) Floods
3) Animal welfare crises
4) Off-shears losses - severe hypothermia
5) Grain overload (‘lactic acidosis’)
6) Transport accidents

73
Q

What are the main roles of vets after emergencies

A

○ Assess & give prognosis for burnt, injured or affected livestock
○ Consider welfare of:
§ Livestock
§ Farmer
○ Minimise suffering of unsalvageable animals
○ Minimise unnecessary destruction of affected but otherwise healthy animals

74
Q

Humane destruction of sheep during emergencies what can use and how to ensure the animal is dead

A
  • Gunshot
  • Captive bolt
  • Blunt trauma (1 day old)
  • Bleed out (sheep & goat)
  • Lethabarb
    5-finger head check’:
    a. no corneal reflex
    b. dilated pupils
    c. slack jaw tone
    d. flaccid tongue
    e. no breathing
75
Q

Burns during emergencies for sheep what are the 2 main things need to consider and what makes it hard

A
- Intensity &amp; type of fire
○ Amount of fuel - bush, forest, grass 
○ speed &amp; direction of wind
- Which animals more susceptible?
○ Size (weaners vs. adult)
○ No. in mob, position in mob
○ Wool length in sheep:
§ is a good insulator &amp; protects from radiant heat
HARD - Yards &amp; fences often destroyed - hard to inspection of animal
76
Q

What are the classifications of burns and common signs within

A

1st Degree:
- Superficial layers affected only
2nd Degree:
- Superficial:
○ All skin layers, but to varying depths
○ Blisters are a common sign
- Deep:
○ All skin layers but not hair follicles or sweat glands
○ Eschars common - attractive for flies
3rd Degree
- All layers damaged including hair follicles
4th Degree
- All skin layers are affected including deeper tissues (muscle, tendons, fascia, nerve endings)
- Pain is often absent due to damaged nerve receptors
- [oedematous swelling can also reduce pain responses]

77
Q

Assessing burns in sheep what are the 2 main things need to do and what will occur

A
1) Assessment of non-wool (bare) areas:
○ Ewes – udder
○ Rams – pizzle &amp; testes (guarded prognosis)
○ Eyes
- After the fire – hot &amp; inflamed
- Over the next 3-4 days:
○ Tend to look worse
○ Blacken, leathery, hard &amp; dry
○ 2o infections, loss of plasma protein
2) Divide survivors into 3 categories
1 - destroy immedaitely 
2 - emergency slaughter or retain in flock and reassess 
3 - minimal damage
78
Q

Category 1 in categorizing sheep after burns what need to do and what defines

A
- Destroy immediately:
○ Unconscious
○ Recumbent
○ Extensive lung damage → Dyspnoea
○ Extensive burns to lower leg
○ Burns to >15-20% of body area
79
Q

Category 2 in categorising sheep after burns what are the 2 options and what defines within

A
  1. Emergency slaughter
    OR
  2. Retain in flock and reassess:
    ○ Burns to face
    § Unsightly but heal well
    ○ Mild leg burns → may lose hoof but will recover
    § Highly attractive to flies again
    ○ Axilla or inguinal area (anus, vulva & udder):
    § May heal, but scarring
    ○ Reassess
    § initially every 2-3 days - destroy recumbent/ dyspnoeic
    ○ Treat (more valuable) animals?
    § Antibiotics (broad-spectrum + topical sulfadiazine)
    § NSAIDS & Tri-solfen®
    § Avoid c’steroids
80
Q

What are some issues with placing sheep into category 2 with sheep after burns and what generally isn’t done anymore and why

A
  • Some sheep/ cattle may need destruction later on
  • Assess udder & teats for damage
  • Palpate scrotum & examine pizzle
    ○ Obvious fibrosis
    ○ Reduced sperm quality & fertility
    ○ Assess @ 2-6 months
  • Keep close to yards if possible – on good feed & water, shade
  • Salvage slaughter: - generally not done anymore
    ○ High condemnation rate?
    ○ Distance? - important for welfare
    ○ Abattoirs may not be interested (wool sheep)
81
Q

Category 3 in categorising sheep after burn what sheep within and what need to do

A
  • Some burnt wool, but bare & haired areas OK
  • Let out, minimal care required
  • Reassess at 7 days
    ○ Burns to inguinal & axilla areas overlooked?
82
Q

Cattle burns what places do fine and which need to euthanise

A
  • Burnt feet - generally do fine

- Bull prepuce, testicles, feet swollen and separation at the coronet -> euthanise

83
Q

What are the 5 main complications after a fire

A
1. No feed/ yards/ fences
○ Pipe melting - water can be an issue 
2. Fly strike:
○ Coronary band
○ Tx: Cyromazine (Spinosad if struck) -> FOOTBATH 
3. Infections
4. Worms
○ Decreased immunity?
5. Insurance?
84
Q

Floods and trying to save the animals what are the 6 main options

A
  1. Muster or remove to higher ground with feed -> easier with cattle
  2. Create access – open gates/ cut fences
  3. Fodder drops? -> dropping in round bails -> if large areas of land, wait and monitor
  4. Air lift? -> expensive!
  5. No immediate action needed -> monitor
  6. Euthanasia - can be difficult to access
85
Q

Prompt euthanasia in a flood what sheep should do on, how?

A
- Injured, exhausted, debilitated:
○ Standing in water for > 5d
○ Bogged (heavy black soils)
○ Septicaemia or neurological disease.
○ Prolonged recumbency
○ Full wool (fleece waterlogging, green)
○ Poor avoidance behaviour
○ Scattering, not in mobs
- How - challenge
86
Q

Animal welfare disasters what generally occurs and why

A
- Many sheep moribund or dead:
○ No or minimal feed/ water
CAUSE
1) Fail to recognise or treat Disease:
§ Internal parasites
§ Fly-strike
§ Footrot
2) Fail to conduct routine management procedures:
○ Inexperienced
○ Sheep traders/ dealers
○ Elderly
87
Q

Animal welfare disasters who handles this and what is commonly involved

A

○ DPI – handle ‘in-house’
○ RSPCA may call a Private Vet to help sort problem out
§ Post-mortems, worm counts
§ Ageing of carcasses
§ Assessment of feeding & management
○ Legal responsibilities & powers (entry/ treatment) under Acts:
§ POCTA
§ Vet Surgeons Act
- often a human crisis - - Appropriate counselling by trained people can help resolve the animal welfare problem

88
Q

Hypothermia as a diaster when generally occur, highest risk and what generally occurs

A
  • Losses can occur any time, but disasters tend to occur with summer storms
  • Sheep losing weight & recently off-shears at greatest risk
  • In wet, windy weather:
    ○ Sheep move downwind
    ○ Stop eating
    ○ Become ‘hunched up’, shiver & reluctant to move - know you have an issue
  • If bad weather continues:
    ○ Core body temperature decreases
    ○ Recumbent → Coma & death
    ○ Sheep found in a heap in a downwind corner of the paddock
89
Q

What are the 6 main things that influence losses via hypothermia and how do this

A
  1. Weather:
    ○ Rain + wind speed (+ temperature)
  2. Time after shearing:
    ○ 75% deaths occur within 2 weeks
    ○ 95% deaths within 4 weeks
  3. Shelter:
    ○ Plantations often located upwind
    § Downwind better - may have to cut fences to let sheep in
    ○ In/ behind buildings, under shearing sheds
    ○ Long grass (Phalaris) & Tussocks give some shelter
  4. Body size
    ○ Weaners & lambs have increased surface area/ weight ratio
  5. Body condition
    ○ Minor effect - fat provides some insulation but can’t be mobilised quickly enough
    ○ Sheep losing condition more at risk
  6. Feeding?
    ○ Hay feeding can be some help (but is not fully protective)
    ○ access to high quality feed in fasted newly shorn sheep can decrease ‘lower critical temperature’
    § Temperature below this need to start metabolising own reserve - more susceptible
90
Q

With hypothermia disaster what need to do to sheep that have collapsed

A
• Move into shed (priority for those recently lost condition)
• Dry with towels or cloths
• Insulate &amp; warm with anything:
○ Hay, wool, bags
○ Industrial heaters - useful
91
Q

With hypothermia disaster what need to do to sheep that are still standing

A
-  Sheep still standing -> move to shelter:
○ can be very difficult
○ need dogs &amp; plenty of people
○ priority for low CS mobs
○ cut fences
- Grazing between squalls is a good sign
92
Q

prevention for hypothermia disasters

A
- Watch for sheep weather alerts
○ Rain &amp; strong wind
○ Move unshorn sheep out of shed
○ Protective coats?
- Longer term:
○ Downwind shelter belts with gates
93
Q

Grain engorgement what are the risk factors

A

1) Rapid introduction of cereal grains:
a) Supplementary rations
b) Spilt grain – around silos or when first introduced to stubbles
c) Break into standing cereal crop
d) Rolled or cracked grains
2) Type of grain:
○ High risk - wheat, barley, (bread, dough)
○ Medium - triticale & maize
○ Low - oats & sorghum
○ Virtually no risk - lupins & field peas

94
Q

Grain engorgement with sheep what clinical signs within first 18-36 hours and within rumen

A
- Within 18-36 hours:
○ Listless &amp; anorexic
○ Diarrhoea (scant faeces later)
○ abdominal pain – hunched up
○ Tender feet (acute laminitis)
○ dehydration
- Rumen:
○ Firm &amp; doughy
○ Stasis
○ Characteristic smell
○ pH <5 in live or recently killed sheep (optimum 6.5-7.0)
○ Rumen full of grain, papillae may strip from mucosa (chemical &amp; fungal rumenitis)
95
Q

Grain engorgement treatment for recumbent, acute cases

A
  • Shift away from source of grain
  • Destroy recumbent animals
  • Acute cases:– rumen buffers:
    ○ 40-50g Magnesium hydroxide into rumen (600 mL of 7-8.5% solution – ‘milk of magnesia’)
    ○ 10-20g Sodium bicarbonate orally
  • More intensive treatment? (valuable ram or pets):
    ○ Rumenotomy (& cud transfer)
    ○ i/v Fluids:
    § 1-2 L of glucose saline
    § Sodium bicarbonate – 500 mL of 2.5% i/v over 30 mins, then isotonic (1.3%) over 6-12 hrs (100 mL/ kg BWt)
    ○ NSAIDS for toxaemia
96
Q

Prevention of grain engorgement

A
  • Restrict access to grain silos/ feed dumps
  • Gradual introduction onto grain:
    ○ 50 g/ head/ day
    ○ ↑ 50 g/ head every 3rd day to 300 g/ head/ d
  • Include Virginiamycin? (Eskalin®, [Founder Guard®])
    ○ An S4, costs ~10 c/day
    ○ Useful for emergencies (where rapid introduction of grain needed):
    § After fires
    § Pregnancy toxaemia
97
Q

Transport accidents how common, who attends, role of vet and disposal

A
  • Regular occurrence on some sections of road
  • Local Gov’t & DPI will usually attend
  • Assist with destruction of injured animals?
  • Often very difficult to deal with
  • Council usually digs pit at site to bury carcasses
98
Q

Abdominal signs what are the 3 main clinical presentations and causes for each

A
  1. Diarrhoea
    ○ Worms
    ○ Coccidia
    ○ Bacterial - Yersinia, campylobacter, salmonella
    ○ Dietary - early weaning, sudden change, specific plants, ruminal acidosis
  2. Tenesmus
    ○ Alimentary - often causes of diarrhoea
    ○ Urinary - urothiasis or cystitis
    ○ Genital - penile trauma/obstructive infections (pizzle rot), abortion, pregnancy, parturition
  3. Abdominal distention
    ○ Younger - dietary change, overfeeding, abomasal bloat (over milk feeding - bloat not as common in sheep than cattle)
    ○ Older - dietary change, bloat, impaction, hernias, abdominal accident (redgut, dilation or volvulus - GENERALLY HINDGUT IS THE PROBLEM), urinary tract rupture
99
Q

What is the general approach to abdominal issues in sheep

A
  • Clinical signs as for cattle
  • Do TWC (total worm counts) via necropsy
  • Necropsy -> VERY USEFUL
    ○ Not exploratory laparotomy like in cattle
  • Faecal examination, flotation culture, sedimentation
    ○ Liver fluke, strongyles
  • Worm egg count
    ○ Sampling of mobs or individual
    § Individual get sample per rectum and place ear tag number next to sample
    □ Give idea about spread, for drench, treatment
    ○ Collect 10-20 fresh samples in paddock OR YARDS
100
Q

Redgut pathophysiology what causes the issue then what results

A

○ Especially on highly digestible legume diets
§ Moves quickly through the rumen -> hypermotility -> relative decrease in size of reticulorumen
§ Carbohydrate spillover from rumen -> Increased hindgut fermentation -> enlargement, displacement and twists
○ Displacement and torsion/volvulus of large bowel
§ Disruption of blood supply - infarction - collapse and sudden death
§ Usually found dead after intestinal crisis -> can reach 20% if not action is taken