SM02 Mini4 Flashcards

1
Q

why is it importnat to talk about sex w/patients?

A
  • talking about sex history helps improve overall health
  • reduce disease & death by preventing & treating STDs, HIV, & hepatitis
  • improve patients’ mental health & well-being thru identifying & tx of sexual problems
  • improvement in physician-patient relationship
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2
Q

barriers in taking sexual hx

A
  • time
  • reimbursement concerns
  • privacy
  • confidentiality
  • provider discomfort
  • unfamiliarity w/content or language
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3
Q

5 Ps of sexual hx

A
  1. partners: type & number
  2. practices: type of sexual contact
  3. protection from STDs
  4. past hx of STDs
  5. prevention of pregnancy
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4
Q

questions for how to determine cause of infertility

A
  • is ovulation occurring?
  • is there adequate sperm w/adequate moltility?
  • are Fallopian tubes patent?
  • is uterine cavity ok?
  • is there any pelvic disease
    • pelvic inflammatory dz can damage hair cells
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5
Q

classic workup for infertility

A
  • documentation of ovulation
  • semenanalysis of partner
  • hysterosalpingogram evaluates uterine cavity & Fallopian tubes
  • diagnostic hysteroscopy to evaluate for pelvic disease
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6
Q

how should the endometrium appear on ultrasound around the time of the LH surge?

A

trilaminar

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7
Q

cause of luteal phase defect

A

probably abnormal formation of cellular adhesion molecules (CAMs), ex. integrins, & abnormal hormonal response of endometrium to estrogen & progesterone

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8
Q

what studies are included in a semenanalysis?

A

sperm density (#/volume)

sperm motility

sperm morphology

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9
Q

oligozoospermia

A

decreased # of spermatozoa in semen

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10
Q

aspermia

A

complete lack of sperm

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11
Q

hypospermia

A

reduced seminal volume

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12
Q

azoospermia

A

absence of sperm cells in semen

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13
Q

teratozoospermia

A

increase in sperm w/abnormal morphology

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14
Q

asthenozoospermia

A

reduced sperm motility

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15
Q

WHO criteria for normal sperm parameters

A

2010

1.5mL ejaculate

15million sperm/mL

32% motility

4% strict morphology by Kruger criteria

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16
Q

how is sperm chromatin so tightly packed compared to somatic cells?

A

histones are replaced by protamines that allow DNA to form toroidal structures

17
Q

what causes Asherman’s Syndrome?

A

vigorous curettage of uterus or manual extraction of the placenta

ligher or more painful periods develop after such a procedure

may see uterine adhesions

18
Q

what is the difference between an polyp & a fibroid?

A

polyps are soft & of endometrial origin

fibroids are hard muscular growths

19
Q

other than the pelvis, where can signs of pelvic inflammatory disease be found?

A

perihepititis can be caused by PID

adhesions around the liver

20
Q

adenomyosis

A

endometrium grows into the pelvic wall

21
Q

tests to estimate ovarian reserve

A
  • day 3 of menstrual cycle FSH <9IU/dL & estradiol <60pg/dL
  • Clomid challenge test
  • antral follicle count
  • anti-Mullerian hormone
  • inhibin B
22
Q

health risks for PCOS pt

A

**pt is estrogen replete**

endometrial hyperplasia

CV dz

HTN

diabetes

lipid abnormalities

obesity

sleep apnea

23
Q

treatment for infertility of PCOS pt

A

induce ovulation w/clomiphene citrate

24
Q

health risks for exercised induced hypothalamic amenorrhea

A

hypogonadotrophic hypogonadism→ very estrogen deficient

CV plaque formation

oesteoporosis

stress fracture

dysparunia

25
Q
A