Shoulder Flashcards

1
Q

shoulders are complex

A

} Lots can go wrong and numerous structures can cause shoulder pain
} To assess and treat shoulders effectively you need to
} Understand the anatomy } Joints
} Muscles
} Ligaments etc
} Understand the complexity of movement and control } Joint interactions
̈ Any painful or weak link can decrease effectiveness of shoulder function } Complex co-operative muscle interactions
̈ Any single weakness can disrupt kinetic sequencing } Be able to effectively differentially diagnose
} Assessment is the key!!!

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2
Q

4 joints of the shoulder

A
} Sternoclavicular
} Acromioclavicular
} Glenohumeral
} Scapulothoracic
} All 4 joints work co- operatively
} ROM largely produced by ST & GH...
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3
Q

sternoclavicular joint

A

} Clavicle → mechanical strut for scapula through its attachment to sternum
} Links axial skeleton to appendicular skeleton
} Primary purpose → position scapula optimally to receive head of humerus
} Highly stable – ligamentous structure
} Injury due to large forces } Commonly = fracture
BUT
} Potential SC joint dislocation ̈ Anterior vs posterior….

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4
Q

acromioclavicular joint

A

} Stabilised by
} Superior & inferior acromioclavicular
joint capsular ligaments
} Coraclavicular ligament
} Articular disc (of varying form, mostly present)
} Deltoid & upper trapezius muscles
} Proposed rotational adjustment
motions
} Optimally aligns scapula against thorax
} Adds to scapula motion
} Clinically important – don’t neglect!

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5
Q

glenohumeral joint

A
} Multiaxial synovial joint
} Design favours mobility over
stability
} Producing extensive ROM with ST
} Bony fit offers little to no stability
} Glenoid fossa covers approx 1/3 HOH
} * Mechanical integrity maintained by muscles and capsular ligaments
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6
Q

glenohumeral joint

A

} Provided by:
} Joint capsule & capsular ligaments
} Inherently loose capsular ligaments
̈ superior GH, middle GH, inferior GH ligs (esp ant & posterior bands in functional abd position)
} Reinforce capsule to assist maintenance of negative intra- articular pressure
} Taut in varying positions → please review!
} Coracohumeral ligament } Glenoid labrum
} Expands glenoid cavity } Rotator cuff muscles
} LH biceps brachii

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7
Q

scapulothoracic joint

A

} Specific ST ROMs
} BUT important element of overall shoulder biomechanics } Essential involvement in GH ROM & overall shoulder function

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8
Q

scapulohumeral rhythm

A

• *If there is one movement you can assess well…
• Consider ALL joints & relevant muscles
• Overall ratio of 2:1 = GH:ST
• Initial movement = purely GH
• Clavicle retracts & posteriorly rotates
• Scapula tilts & ER
• GH ER
• Why??
• Supraspinatus contracts - taut superior
capsule & depression of HOH
• Auxillary pouch stretches → inferior
sling for HOH

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9
Q

GH joint muscles

A
} Glenohumeral joint } Deltoid
} Anterior, middle & posterior } Coracobrachialis
} Biceps brachii
} Latissimus dorsi
} Pec major
\+
} Rotator cuff
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10
Q

rotator cuff musculature

A
} Supraspinatus (!!!)
} Superior roll of HOH
} Compression into fossa
} Restricts superior translation
} Infraspinatus, teres minor, subscap } Depression force on HOH
} Infraspinatus, teres minor
} External rotation of humerus
abduction
    } *Counteract pull of deltoid → elevation of HOH } Deltoid vs suraspinatus injury
} Consider ability to abduct...
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11
Q

Scapulothoracic joint muscles

A

} Elevators
} Upper trapezius
} Levator scapulae } Rhomboids

} Depressors
} Lower trapezius
} Latissimus dorsi } Pectoralis minor } Subclavius

} Protractors
} Serratus anterior

} Retractors
} Middle trapezius
} Rhomboids
} Lower trapezius

} Upward rotators
} Serratus anterior
} Upper & lower trapezius

} Downward rotators } Rhomboids
} Pectoralis minor

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12
Q

Shoulders are tricky

A

} Difficult to differentially diagnose
} Complex joint interplay
} Complex co-operative muscular control
} Special tests often don’t have high specificity & sensitivity
} Regularly painful > 90o
} Mechanical vs non-mechanical pain
} Pain can be referred from other regions
} May not be MSK issue BUT due to more serious problem…
} Some tips for success
} Be thorough in your history taking } Assess effectively
} Get your patient to do their work!

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13
Q

Hx

A

} Try to pinpoint the site of pain
} Easier said than done!
} AC & bicipital pain often localised
} Define pain
} Type, severity…
} Sensation } Neural
} Referred
} “Dead arm” – baseball pitchers → labral injury
} Onset of pain
} Acute or insidious
} If MOI
} Shoulder position provides clues to structures injured
} Night pain common in some presentations
} Aggravating & easing
} Impact on ADLs

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14
Q

PE

A

} Takes the usual form and covers all bases } LOADS of special tests to choose from…
} Diagnose effectively
} What is your hypothesis?
} What are your differential diagnoses?
} What is contributing to the pain and presentation? } All joints and muscles!!!

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15
Q

6 categories of shoulder pain

A
} Rotator cuff (including impingement)
 } Labral injury
} Instability
} Stiffness
} AC joint pathology
} Referred pain } Cx
} Tx
} Visceral
} Nerve lesions: e.g suprascapular nerve, axillary nerve, long thoracic nerve
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16
Q

referred pain from viscera

A

} Cardiac dysfunction
} Diaphragm dysfunction, lung disease
} Pneumoperitoneum/Perforated ulcer } Disease of the oesophagus
} Aneurysm of the subclavian artery
} Gallbladder and spleen (left shoulder) } Axillary vein thrombosis

17
Q

differential Dx

A

} Pancoasts tumor – pulmonary apex, non small cell Ca

} Bone tumor (young) } More commonly 2o

18
Q

referred pain

A
} Commonly } Cx spine
} Tx spine
} Associated musculature...
BUT
} Shoulder dysfunction can alternatively result in
} Traps pain and fatigue
} Pain radiation into } Neck
} Posterior scapula } Upper arm
} Forearm
} Wrist and hand
19
Q

rotator cuff pathology

A
} One of the most common shoulder problems you will treat
} Supraspinatus most commonly affected
} Tendinopathy
} As per tendon continuum...
} Calcification may occur } Most commonly within
supraspinatus
} Investigation of choice } MRI
} Alternatively US
20
Q

Rotator cuff pathology clinical presentation

A
} Pain often considerable } Rest
} Seemingly minor movements } Night
} Potential for associated instability & “dead arm”
} Pain with overhead activities
} < 90o painfree
} Painful arc – 70-120o
} Catch/difficulty on return
} TOP supraspinatus tendon at insertion point
} If supra tendinopathy.... } ↓ IR – consider HBB
ROM...
} Pain on
} Empty can > full can
} Impingement tests
} EOR passive flexion
21
Q

impingement syndrome

A

} Impingement syndrome - ?overuse, tendinopathy of supraspinatusàpainàRC muscle dysfunctionàpotenital bursitis
} 3 types of impingement
} 1 ̊ External
} Encroachment of subacromial space from superior structures } Spurs may be seen on X-Ray
} 2 ̊ External
} Inadequate muscle stabilisation of scapula
} Internal (glenoid)
} Overhead sports à undersurface of RC against posterosuperior surface
of glenoid during late stage cocking,
̈ Overuse may cause pathological process and superior labrum injury

22
Q

Rx and rehab for impingement

A
} Pain relief
} Mobilisation, STT, medication...
} Correction of abnormalities
} GH instability
} Muscular weakness and imbalances
} ST tightness
} Posterior capsule tightness
} Impaired scapulohumeral rhythm
} Correct training errors or functional errors
23
Q

Labral lesions

A

} Excess traction on labrum through LHB } FOOSA
} Classification
} SLAP – stable vs unstable, 4 grades
} Non-SLAP – degenerative, flap, vertical labral tears, Bankart
} Bankart – unstable lesion anterior inf labrum, repetitive dislocations } Hill-Sachs – damage to posterior humeral head

24
Q

Labrum assessment and management

A

} Clinical presentation SLAP } TOP anterior shoulder
} Pain on resisted biceps contraction – Dynamic labral shear, O’Brien’s
} Management
} Stable SLAP/nonSLAP = arthroscopic debridement
} Unstable SLAP/non-SLAP = arthroscopic fixation
} Conservative: >50 yo; young, minor SLAP; ?non-overhead sports
} NSAIDs, scapula stabilisers/dynamic stabilisers, capsule stretching } AVOID heavy weights early

25
Q

GH instability

A

} Post-traumatic, atraumatic or combination
} Most commonly anterior (90%) > posterior > inferior or
multidirectional } Due to
} Trauma – FOOSA
} Anterior = abd + ER
} Posterior = flex + add + IR } Inferior = traction
} Multidirectional
} Atraumatic – overhead athletes, ligamentous laxity } Combination – traumatic episode in lax shoulder

26
Q

anterior instability clinical presentation

A
S &amp; S + Assessment
}S&amp;S
} Recurrent dislocation or subluxation
} may incr in frequency &amp;/or occur with minor activities
} Shoulder pain
} Impingement of RC à weakening &amp;
inadequate stabilisation cycle
} “Dead arm”
} Potential catching – labral
detachment
} Assessment
} Apprehension&amp;relocation
} Load &amp; shift (Drawer)
} Pain &amp; apprehension noted on
Anterior Drawer
} Minor instability – perform ant drawer + abduction &amp; ER
Treatment
} Atraumatic
} Muscular stabilisation
} Correct technique in athletic population
} Surgery if required } Capsular shift
} Traumatic
} }
Correct underlying mechanism
Surgery – esp for recurrent anterior subluxation
} Bankart repair
} Bone graft if Hill-Sachs’ lesion
present
} Nil tendon transfer in athletes  ̈ Loss of ER ROM + ↓ power
27
Q

posterior and multidirectional instability

A

Multidirectional
} Combination of 2-3 instabilities
} Most commonly atraumatic BUT can be traumatic
} ExtremeROMsordirectblow(rare) }S&S
} Generalisedligamentouslaxity–hypermobility syndrome
} +ve on all instability tests
} Apprerehension;Drawer;Sulcus
} PainintranslationinmidROMs
} Alteredmuscleactivationplaysconsiderablerole
} Scap retraction = decr pain
} Rx
} Relief of symptoms
} Therapeuticexerciseprogram–avoidstretching!
} Possiblysurgery–results????
Posterior
} Most commonly atraumatic as part of multidirectional
} Subluxation & dislocation may damage post labrum
} Clinical presentation
} May be able to voluntarily
sublux
} Posterior Drawer = marked displacement

28
Q

adhesive capsulitis

A

} No consensus on aetiology – 3 theories } Inflammatory
} Fibrosing
} Algoneurodystrophiccondition
} Reflex sympathetic dystrophy } Idiopathic or post-traumatic } Decalcification of HOH
} Presentation
} 40-60 yo, female > male
} Diabeticàhigher incidence } Evaluate ER PROM in neutral } May last 18 months+
} 3 phases
} Phase 1: onset of severe pain, but movement not stiff.
} Phase 2: pain at EOR and night, movement more restricted
} Phase3:paindecreasing,slowlyincreasingmovement;“thawing”

29
Q

adhesive capsulitis management

A

} Exclude primary causes
} Assessment by orthopaedic surgeon
} Intra-articular cortisone injection may be helpful in early stage } Late stage:
} MUA – may lead to intra-articular damage } Capsulotomy
} Physiotherapy
} No evidence that PT impacts on outcome
BUT
} Assists in managing pain and symptoms } Education essential

30
Q

AC J dislocations

A

} Clinically → inherently susceptible due to:
• Sloped joint
• High probability of large shear force
} Mechanism: direct fall on the shoulder with arm in adduction } Common in rugby codes…
} Presentation
} Pain directly over AC joint
} All extremes of movement aggravate pain
} Joint sprain
} Gr I = damage to capsule &
acromioclav lig
} Gr II = rupture acromioclav & damage to coracoclav lig
} Gr III = rupture acromioclav & coracoclavic ligs

31
Q

management of ACJ dislocation

A

• Mx = RICE, isometric strength, taping particularly on RTS