Seriously Ill Child Flashcards

1
Q

A women comes in saying her baby sleeps to much, what are some questions you can ask to know if its pathological?

A
  • Sleep up to 18h / day
  • Fix but don’t follow
  • Wake regularly for feeds
  • Suck strongly on the breast
  • Rousable to activity (e.g. limb movements)
  • Normal sounding cry
  • Flexed posture w. some time - Preterm babies Ø until ~32wks
  • May have some chest indrawing (esp. premature)
  • May have periodic respiration
  • Ø apnoea = period of no respiration that is abnormally long
    • serious sign:
      • ​RSV (bronchiolitis)
      • Croup
      • Acidosis
      • Sepsis
      • Patent ductus arteriosis
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2
Q

What are some system observations to measure to determine wellness of a child?

A

CNS

  • State
  • Arousal
  • Posture

Resp

  • Resp distress
  • Hypoxemia
  • UAO

CV

  • Adequate end-organ perfusion
  • Hydration
  • HR
  • BP
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3
Q

A seriously ill child is in ED and you have to start standard resus, what are some considerations you should consider?

A
  • DO NOT INTUBATE - unless the child has been resuscitated with fluid, can cause circulatory collapse.
  • start with 20ml/kg
  • once you get to 40ml/kg and the child still has signs of shock do something else (vasopressor, ionotrope, ICU)
  • Hartmann’s is good in acidosis (lactate converted by bicarb) - vs normal saline which causes high chloride and exacerbates acidosis.
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4
Q

Types of Tachypnea on examination?

A
  • expiratory wheeze (long expiratory phase)
    • asthmatics
    • bronchiolitis
    • anaphylaxis
  • expiratory cough/wheeze (short expiratory phase)
    • pneumonia - maintain lung volumes
  • inspiratory stridor
    • croup
    • foriegn body inhalation
    • anaphylaxis
  • Effortless tachypnea
    • acidosis (diabetes, sepsis, dehydration, methylene glycol ingestion)
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5
Q

What type of rash do you get in GAS disease?

A
  • scarlatinoform red erythematous rash

other rashes not to miss?

  • non-blanching petechial rash (rub they don’t go away), <2cm petechial, >2cm purpuric - meningococcal infection
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6
Q

What are the types of shock? What are some signs?

A

Signs:

  • Tachycardia
  • Increased RR
  • pulse (brachial, femoral then carotid)
  • capillary refill (push for 5 seconds, <3seconds normal)
  • Hypoperfusion - cool peripheries
  • Altered concious state
  • hypotension is a late sign in kids

Types:

  1. neurogenic (loss of sympathetic input, above T6)
  2. septic/anaphylaxis (distributive)
    • Warm shock
  3. obstructive (Cardiac - coarctation, duct dependent)
  4. hypovolemic
    • Cool shock
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7
Q

What is the management of someone who is in shock?

A

DRS ABCDE

  • A - steth out - look and have a listen - not sure assume not breathing
  • B - neonatal resus - give O2 and some breaths
  • C - circulation - cap refill central, BP, pulse (femoral/brachial - not carotid).
    • Bolus - 20mls/kg. 2IV lines.
    • Ionotropes - +/- cardiogenic.
  • D - disability
  • E - exposure
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8
Q

What is a quick way to approximate someones weight based on age in an emergency?

A

(age+4) x 2

often underestimates in devloped countries, but is quick for approximating.

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9
Q

A child with DKA has hyperglycemia, acidemia, hypocapneoa, low bicarb, low sodium and low potassium. Explain the sodium result. Define DKA.

A
  • metabolic acidosis with some compensation
  • dehydration would expect it to be higher
    • corrected sodium not actual?
    • pseudohyponatremia - dilutional.
      • in hyperglycemia and hypercholestrolemia

What is DKA?

  1. low pH
  2. high glucose
  3. presence of ketones
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10
Q

What is the Management of someone in DKA? What specifically do you need to monitor?

A
  • fluid bolus:
    • on conservative side - 10mls/kg bolus + maintenance (see guideline)
  • insulin +/- dextrose (aim for 5-12 range)
    • disconnect pump
    • IV access - 2.4units/hr generally.
  • Potassium - always give. 40mmol/L KCL. insulin causes intracellular shift.
  • NBM
  • ECG - baseline and potassium
  • Monitor GCS
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11
Q

Why is it so important to monitor GCS in a DKA patient?

A
  • Sodium - overcorrected.
  • DKA cerebral oedema (one theory). Lower blood sugar in blood than in CSF water flood brain.
    • Patients at risk fo cerebral oedema - really sick, younger, concurrent meningitis or resp infections.
    • Monitor for papilloedema, frontanelles, Obs - cushings triad (bradycaridia, hypertensive, mixed RR)
  • Ketotic seizures.
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12
Q

How do you classify a child’s hydration status?

A
  • Mild - dry membranes, mild tachy, decreased UO
  • Moderate - sunken eyes, respiratory rate increase, tachycardia, reduced tissue turgor, lethargy
    • delayed CRT >2secs (centrally)
  • Severe - shock, mottled limbs, altered consciousness
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13
Q

What is Reye’s Syndrome? How does it present?

A
  • a non-inflammatory encephalopathy
  • Presentation:
    • abrupt vomiting 12hrs-3days after viral illness
    • associated with aspirin
    • neurological Sxs (lethargy, diarrhoea)
  • complications:
    • brain herniation
    • SiADH
    • ARF
    • sepsis etc…
  • 20-50% mortality
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14
Q

What are the common organisms that cause meningitis in infants?

A
  • <2mths old:
    • GBS
    • E. coli
    • listeria
  • >2 mths:
    • streptococcus pneumoniae
    • neisseria meningitidis
    • HIB
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