Restorative 1 Flashcards
what can impede calculus removal?
crowding/tilting root anatomy pocket size and shape access furcations
what are the effects of RSD?
health improved
gingival resolution
reduced pocket depth
long JE forms
when is RSD contraindicated?
poor oh compliance
acute inflammation
severe dentinal hypersensitivity
what does RSD do to the crown length?
makes it longer
what is calculus?
a calcified mass that adheres to hard surfaces not subject to friction
where is supra calc found?
opposing salivary ducts
e.g stensons/whartons
what is calc made of?
70-80% inorganic - calcium/phosphate
organic - lipids, carbs, proteins
what types of calc exist?
HA
octacalcium phosphate
brushite
magnesium whitelocke
where is brushite mostly found?
supra and new calc
where is HA mostly found?
older calc
where is mg whitelocke mostly found?
sub calc
what shapes does calc exist in?
needle, rod, platelet shape
what are the 3 calc formation theories and what do they mean?
Co2 theory - new saliva, increased CO2 conc = increased pH = crystallisation
ammonia theory - increased urea = ammonia = increased pH = crystallisation
Nucleation theory = crystallisation bc seeding agent
what are the indirect effects of calculus?
rough surface
hampers good oh
porous and holds toxins
what are anticalculus agents?
pyrophosphates = reduce new supra calculus
what is the critical pH of enamel ?
5.5
features of healthy gingivae?
pink, firm, uniform colour, stippling, knife edge margin, flat traiangular ID papillae, no BOP
features of gingivitis?
bleeding halitosis pain unpleasant taste inflammation starts ID and spreads
what happens to the tissues in gingivitis?
blood vessels dilate
tissue becomes red and swollen because of inflammatory exudate
fibres broken down by inflammation and stippling lost
false pocketing
what does long standing plaque irritation cause?
fibrous tissue
types of plaque disclosing tablets?
erythrocin - red fresh plaque
malachite green
blue - older
what TB technique is recommended?
modified stillman technique
45 degrees to gingival margin
what is the primary cuticle?
remnants of tooth development near g margin
usually worn away by natural friction
can persist and stain
what is the aqcuired pellice?
forms within seconds of cleaning, salivary glycoproteins
how is the acquired pellicle protective?
- glycoproteins and calcium phosphate adsorbed onto teeth - increased resistance
- restricts diffusion of acids from sugar breakdown
- antibacterial factors - IgA, IgM, complement, lysozyme
what is chronic periodontitis?
plaque induced inflammation of the tissues resulting in : pdl destruction
loss of alv crestal bone
apical migration of JE
what is a perio pocket?
deepened crevice, migration of JE onto root surface
how does perio disease progress?
BURST theory
bursts of disease activity can cause >3mm of attachment loss in a few weeks
what is the probing force used?
20-25g, modified pen grasp used
what is a biofilm?
microbial ecosystem adherent to a solid surface
what is a biofilm made of?
70% microorganisms, 30% interbacterial matrix
what is the intermicrobial matrix?
dead organisms, metabolic by products, enzymes, toxins
what does rubber dam do?
isolates tooth from salivary bac, protects airway
watertight junction around cervical margin, excludes moistures
what are the 2 types of disease progression?
linear - RAL/GAL
burst theory - bursts of disease with quiescent periods
describe a BPE probe?
WHO probe
ball end 0.5mm
coloured band 3.5-5.5mm
2nd coloured band - 8.5-11.5mm