Respiratory Control & Group Pacemaker Hypothesis.

Question 1

Central Chemoreceptors

Answer 1

Increased PCO2

Sensitive to changes in pH of the cerebrospinal fluid

Question 2

An increase in arterial PCO2 results in…

Answer 2

diffusion of CO2 from the blood into the brain which lowers pH and stimulates the central chemoreceptor to send signals to the respiratory control center to increase breathing
Results in increased alveolar ventilation and the the elimination of CO2

Question 3

Peripheral Chemoreceptors:

Answer 3

Carotid Body (Increased PCO2, Decreased pH and PO2); Aortic Body (Increased PCO2, Decreased pH)

Question 4

Carotid Body

Answer 4

Sensitive to changes in arterial PO2, PCO2, and pH; Increase in arterial PCO2 stimulates the carotid body to send signals to the respiratory control center to increase breathing

Question 5

Relative role of O2 vs CO2 in stimulating VE

Answer 5

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Question 6

Motor Cortex

Answer 6

Sends out impulses to activate skeletal muscles to contract and increase ventilation in proportion to the amount of exercise being performed; Neural impulses pass through the medulla and “spill over” causing a VE that reflects the number of muscle motor units being recruited

Question 7

SKM Mechanoreceptors

Answer 7

Muscle Spindles, Golgi Tendon Organs, and Joint pressure receptors

Question 8

Respiratory Muscle Training & the Metaboreflex.

Answer 8

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Question 9

Ventilatory Adaptations to Endurance Exercise Training.

Answer 9

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