Reconstruction Flashcards

1
Q

What are type one sternal infections?

A

Occur within several days and present with serosanguinous drainage
Treated with irrigation, minimal debridement enclosure

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2
Q

What are type two sternal infections?

A

Present a few weeks postoperatively with cellulitis, mediastinitis, osteomyelitis, and drainage of pus
Treated with irrigation and debridement and may require reconstruction

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3
Q

What are type three sternal infections?

A

These occur months later with osteomyelitis chondritis and a chronically draining sinus tract
Can be treated with debridement irrigation, and possible reconstruction

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4
Q

What is the most common organism identified in sternal infections?

A

Staph

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5
Q

What structures are most commonly affected by primary lymphedema?

A

Lower extremity followed by genitalIa followed by upper extremities

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6
Q

What is the genetic cause of primary lymphedema?

A

VEGFR3, FOXC2, SOX18, CCBE1

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7
Q

Milroy disease

A

Lymphedema which presents at birth With a mutation in VEGFR3

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8
Q

Meige disease

A

Lymphedema presenting an adolescence with a family history of the disease, but no genetic mutation has been identified

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9
Q

What is the most common complication of lymphedema?

A

Cellulitis

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10
Q

Lateral compartment, muscles, and innervation

A

Perineus longus and brevis
Superficial perineal nerve
Ankle eversion

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11
Q

Superficial posterior compartment

A

Gastrocnemius, solus and plantaris
Posterior tibial nerve

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12
Q

Deep posterior compartment

A

Flexor Hallucis, flexor digitorum longus, tibialis posterior
Posterior tibial nerve

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13
Q

Anterior compartment

A

Tibialis anterior extensor digitorum longest extensor Hallucis longus, peroneus tertius
Deep perineal nerve

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14
Q

What nerve may accompany the lesser saphenous vein

A

Sural Nerve
Travels between the Achilles tendon and the lateral malleolus
Give sensation to the posterior lateral lower leg and lateral foot

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15
Q

Muscles of the superficial layer of the plantar surface of the foot

A

Flexor digitorum Brevis
Abductor hallucis
Abductor digiti quinti

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16
Q

What nerve is at risk during the harvest of the plantaris tendon?

A

The posterior tibial nerve
Dissection begins posterior to the medial malleolus

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17
Q
A
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18
Q

How many compartments exist in the thigh?

A

Three the anterior medial and posterior

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19
Q

Anterior thigh compartment

A

Femoral nerve and artery
Quadriceps sartorious illiopsoas pectineus

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20
Q

Medial thigh compartment

A

Obturator nerve
Profunda femoral artery
Adductor brevis longus and magnus
Gracilis, obturator externus

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21
Q

Posterior thigh comp

A

Sciatic nerve
Branches of the profunda femoris
Adductor Magnus, semi tendonosus and membranous, biceps femoris

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22
Q

What is the most common location of bone sarcoma of the lower extremity?

A

Proximal tibia

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23
Q

What to do if the common perineal nerve is disrupted?

A

You can do a tibialis posterior transfer because in an equinvarus deformity develops

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24
Q

What does the mangled extremity severity score consider

A

Shock, ischemia, age and skeletal or soft tissue damage

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25
Q

What is the ideal length that should be preserved for a below knee amputation

A

12 to 15 cm from the joint line

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26
Q

What is aLisfranc amputation

A

Amputation at the level of transmetatarsal joint

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27
Q

What is a symes amputation

A

Amputation just above the ankle joint

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28
Q

What lab test is useful in osteomyelitis

A

ESR above 120

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29
Q
A
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30
Q

What is the angle of protrusion of the ear?

A

25 to 30°

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31
Q

What is the lymphatic drainage of the ear?

A

Superior lateral ear and anterior external auditory meatus drain to the parotid
Superior medial ear and posterior external auditory, drained to the mastoid
Inferior ear and lower external auditory Meatus drain to superficial cervical
Concha and meatus drain to the pre-auricular nodes

32
Q

What is a banner flap?

A

Post ear flap from the superior sulcus, which is used in conjunction with a cartilage graft for upper 1/3 defects

33
Q

Dieffenbach flap

A

Post ear flap with contralateral cartilage support used for reconstruction of defects of the middle 1/3
Cartilage graft is sutured into the defect with the skin advanced over for skin coverage and the flap is then divided 2 to 3 weeks later

34
Q

Do patients with microtia have hearing difficulties

A

Most patients have a atresia of the external auditory canal, tympanic membrane and middle ear ossicles resulting in conductive hearing loss

35
Q

What happens to the dermis during tissue expansion?

A

Thins
Increased fibroblast and myofibro blasts
Increase collagen
Fragmentation of elastin fibers

36
Q

What happens to muscle during tissue expansion?

A

Increased number of mitochondria
Th thinning

38
Q

What is the significance of the temporal line of the calvarium?

A

It is the line along which the deep temporal fascia fuses with the pericranium

39
Q

What is aplasia cutis congenita?

A

Localized, absence of skin, dermal, appendages, and subcutaneous tissues
Can occur anywhere on the body, but majority occur on the scalp
And 15 to 30% of cases, it can’t be associated with defects of the underlying skull or Dura, exposing the brain and sagittal sinus

40
Q

When do lips begin to develop?

A

At 4 to 5 weeks

41
Q

What muscle planes does the facial artery travel within?

A

Deep to the platysma , Risorius and zygomaticus major and superficial to the buccinator and levator Anguli Oris

42
Q

How do you find the mental nerve?

A

It is located below the second mandibular bicuspid

43
Q

Where are the majority of malignancies of the lip?

44
Q

What is the blood supply of the tongue?

A

Lingual artery arising from the external carotid artery drainage into the internal jugular vein

45
Q

What is the lymphatic drainage of the tongue?

A

Level two nodes are most commonly affected
Level one and three are also at risk

47
Q

What antidotes are available for extravasation injuries?

A

Phentolamine should be given within 12 hours of alpha adrenergic agents
Derazoxane should be given within six hours of anthracycline like Doxorubicin

48
Q

In what extravasation injuries do you want to use a warm compress?

A

When you want to have a dilute and disperse effect
With TPN calcium potassium, mannitol, phenytoin, and contrast media

49
Q

In what extravasation injuries do you use a cold compress?

A

DNA binding vesicants
Chemotherapeutic agents except non-DNA binding vesicants

50
Q

When should you not use cold compresses?

A

Vika alkaloids, which are a non-DNA binding vesicant like vincristine and vinblastine

51
Q

What is purple gloves syndrome?

A

Phenytoin related soft injury
Three phases
Pain and purple discoloration, distal access site
Glove like circumferential erythema with edema
Bullae formation, skin, necrosis, and possible compartment syndrome

52
Q

What are non-DNA binding vesicants

A

Vinca alkaloids and taxanes

53
Q

What are DNA binding agents?

A

Anthracycline Alkylating agents
Mitomycin

54
Q

What are the risk factors associated with pharyngo cutaneous fistula formation?

A

Hemoglobin less than 12.5
Neck dissect
Prior radiation and chemo radiation

55
Q

What head and neck tumors are treated with radiation

A

Sarcoma like rhabdosarcoma Ewing sarcoma neurofibro sarcoma
Retinoblastoma
Leukemia

56
Q

At what radiation dosages is cranial facial growth altered

A

Soft tissue effects are seen at four
Bony effects are seen at 30

57
Q

What are the different fields targeted in breast radiation?

A

The chest wall and the three noodle basins, including the axillary supraclavicular and internal mammary artery

58
Q

What is pathologic gynecomastia?

A

Related to underlying disease
Adrenal tumor
Hypogonadism
Tumor
Liver disease

59
Q

What is the pathogenesis of senescent gynecomastia?

A

Primary testicular failure, resulting in hypogonadism and increased adipose tissue leading to increase production of estrogen by aromatase

60
Q

Elevated levels of which gonadotropin’s lead to gynecomastia

A

LH and HCG

61
Q

How does primary testicular failure cause increased estrogen to androgen ratio and gynecomastia?

A

Reduced androgen production and increased aromatase activity in Leydig cells results and increased estrogen levels

62
Q

How do testicular tumors increase estrogen?

A

Steroid producing tumor cells and paraneoplastic production of hCG

63
Q

How is gynecomastia associated with hyperthyroidism?

A

Increase peripheral conversion of androgens to estrogen and increased sex hormone binding globulins

64
Q

What two mechanisms result in gynecomastia and liver patients

A

Increased hCG secretion and increased aromatization of circulating adrenal androgens

65
Q

What chronic disease diseases are associated with gynecomastia

A

Ulcerative colitis
Cystic fibrosis
HIV
Renal failure
Liver disease
Malnutrition

66
Q

Where does the medial canthal ligament attach

A

Frontal process of the maxilla, the thicker posterior portion inserts on the posterior lacrimal crest

68
Q

Describe location of para spinous muscles

A

Deep to LD except @ T 10 -l1 where the serratus poster is sandwiched

69
Q

Describe the development of reproductive organs

A

Female / male differentiation begins @ 6 weeks
Female is default which comes from paramesonephric
Male comes from mesonephric which develops because of mullerian inhibiting substance from Sertoli cells
The male organs develop due to testosterone from Leydig cells

70
Q

Types of osseous callus

A

Periosteal = appears day 3 very important for comminuted fracture non union healing
Medullary = appears day 4 important for stable non displaced, time to union is shortest

71
Q

Most common pathogens in open joints

A

Pseudomonas and klebsiella

72
Q

EDL flap

A

Anterior tibial artery
For <5cm wounds
Incision 2cm lateral to tibia
Located lateral to tibial artery
Preserve superficial peroneal nerve

73
Q

Cross leg flap

A

Local flap necrosis 40%
Infection 28%
Axial blood supply of the post. Descending subfascial cutaneous branchof popliteal artery

74
Q

What tissue does not have lymphatics

75
Q

Meige’s disease

A

MOST COMMON primary LE
Associated with vertebral, cvs malformation, hearing loss.double row of eyelashes
Ad
FOXC2 mutation