Psychopathology: What is Abnormal? - Exam Review Flashcards

1
Q

Clinical psychology

A

attempts to identify pathological mental processes and behaviour.

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2
Q

How to Distinguish Abnormal from Normal

A
  1. ) Deviation from a statistical/cultural norm
  2. ) Societal disruption/observer discomfort
  3. ) Emotional distress
  4. ) Maladaptiveness of behaviour
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3
Q

Deviation from a statistical/cultural norm

A
  • if people normally get sad once a week, but one person gets sad 7 days a week, we would assume this person has depression.
  • deviance doesn’t necessarily lead to disturbance, ex: homosexuality.
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4
Q

Societal disruption/Observer discomfort

A
  • whether a person’s behaviour causes societal stress or discomfort.
  • fails to distinguish people who have a pathology from people who are revolutionary (ex: MLK).
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5
Q

Emotional Distress & Maladaptiveness of behaviour

A
  • criteria used in DSM to identify mental illness.

- problematic because its hard to find objective standard of impairment.

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6
Q

Mental disorder

A

syndrome characterized by clinically significant disturbance in an individual’s cognition, emotional regulation, or behaviour.

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7
Q

Category model

A
  • a mental disorder is qualitatively distinct from normal psychological functioning.
  • individual either has the disorder or does not.
  • ex: Ted Bundy.
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8
Q

Continuum model

A
  • a mental disorder consists of traits that are at an extreme point along a particular dimension.
  • an individual has more or less of a certain trait.
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9
Q

Costs of Diagnosis

A
  1. ) Expectation Biases
  2. ) Social Stigma
  3. ) Potential for mis/over-diagnosis
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10
Q

Rosenhan’s Study

A
  • shows that diagnostic labels distort how other people see a patient.
  • labels can also produce self-fulfilling prophecies.
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11
Q

Job Applicant vs Patient Study

A
  • the label of patient changes the way that a therapist interprets an individuals behaviour.
  • therapist rated those labelled as “patients” as less well adjusted.
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12
Q

Diagnostic Inflation

A
  • Allen Francis
  • argues that the qualification for a disorder is becoming more loose, so more people are being diagnosed.
    ex: ADHD
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13
Q

Benefits of Diagnosis

A
  1. ) Validation for patient.
  2. ) Allocation and coordination of resources.
  3. ) Predicting behaviour of individuals.
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14
Q

Evolution of Abnormal Psych

A

mental illness thought to arise from being possessed by evil spirits.

treatments included:
1.) torture to make body inhabitable.

  1. ) scare evil spirits away by hanging person over snake put.
  2. ) drill holes in scalp to release demons.
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15
Q

Diathesis-Stress Model

A

A model of mental disorders that recognizes the roles and interactions of both predispositions and environmental or situational factors.

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16
Q

Major Depression Facts

A
  • 15-20% of the US population will experience major depression at least once.
  • 75% of individuals will experience at lease one more episode.
  • women are twice as likely to experience an episode than men.
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17
Q

Symptoms of Major Depression - DSM

A

Must have one of these:
- sad mood or anhedonia.

At least four of these:
- sleep changes, suicide ideation, loss of energy, appetite changes, feelings of worthlessness, etc.

18
Q

DSM-5 Criteria for Major Depression

A
  1. ) Symptoms must have been present during a two-week period.
  2. ) Symptoms must produce clinically significant impairment in areas of functioning.
  3. ) Symptoms must not be due to the effects of a substance or a medical condition (i.e hypothyroidism).
19
Q

Melancholic Depression

A

At least one of these:
- anhedonia, lack of mood reactivity.

At least three of these:
- early morning awakening, excessive guilt, worse mood in the morning, appetite loss, etc.

20
Q

Biological Causes of depression

A
  • genetic relatedness (monozygotic twins have 50% chance)

- serotonin dysfunction

21
Q

Serotonin

A
  • plays a role in regulating mood.
  • reduced serotonin signalling in raphe nuclei in depressed ppl.
  • lower availability of serotonin in synapses between neurons in depressed ppl.
22
Q

5-HTT

A
  • serotonin transporter gene, that affects the re uptake of serotonin.
  • influences a persons vulnerability to become highly stressed when exposed to difficult life circumstances.
23
Q

Cognitive Factors

A
  1. ) Attributional reformulation.

2. ) Rumination.

24
Q

Learned helplessness

A

lack of motivation to avoid unpleasant stimuli after one has failed before to escape similar stimuli.

25
Q

Attributional Reformulation

A

the explanations we give to stressors is what determines whether we will develop depression.

  • depressed people make attributions that are internal, stable, and global.
26
Q

Cognitive Triad

A

automatic thoughts from depressed people about themselves, the world, and their future.

  • negative views about one’s self –> negative views about the world –> negative views about one’s future.
27
Q

Dysfunctional Attitude Scale

A

measures the intensity of dysfunctional attitudes, a hallmark feature of depression.

ex: I am nothing if a person I love doesn’t love me.

28
Q

Rumination

A

the tendency to recurrently self-generate negative thoughts about one’s past.

  • predisposes people to depression and relapse.
29
Q

Cognitive Therapy

A

Goal is cognitive restructuring:

  1. ) Become more aware of distorted patterns of thinking.
  2. ) Challenge distorted patterns of thinking.
  3. ) Implement more realistic modes of thinking.
30
Q

Effectiveness of cognitive therapy

A
  • generally equally as effective as SSRIs.

- better at preventing relapse.

31
Q

Subgenual Cingulate

A
  • central area in depression-relevant brain networks.
  • rich in serotonin transporters.
  • activation is specific to sadness.
32
Q

Subgenual Cingulate has projections to:

A
  • prefrontal cortex (attributions).
  • nucleus accumbens (pleasure).
  • hypothalamus (hunger, sex, sleep).
33
Q

Deep-brain stimulation (DBS)

A

electrodes are implanted in the subgenual cingulate, to stimulate overactive brain circuits.

34
Q

Treatment resistant depression

A
  • includes 20% of the depressed population.

- DBS has been shown to relieve depression in many of these patients.

35
Q

Psilocybin

A
  • acts through serotonin neurotransmitter.
  • has no affinity for dopamine receptors.
  • can help decrease depression in treatment-resistant patients.
36
Q

Results of Rosenhan’s Study

A
  1. ) patients sanity was undetected by any of the doctors or staff.
  2. ) normal behaviour was interpreted as being abnormal.
  3. ) patients were treated as less than human.
37
Q

Variations of 5-HTT gene

A
  1. ) short/short allele
  2. ) short/long allele
  3. ) long/long allele
38
Q

Short/short allele

A
  • s/s allele is associated with altered efficiency of serotonin re-uptake.
  • people with s/s allele have greater amygdala responses to emotional faces.
39
Q

Forms of Attributional Reformulation

A
  1. ) Internal vs. External
  2. ) Stable vs. Unstable –> problem will persist forever vs. temporary slip up.
  3. ) Global vs. Specific –> consistent factor vs. context-specific factor.
40
Q

Sternberg Working Memory Task

A

asses rumination by examining a persons ability to replenish and update WM after negative events are deemed to be no longer relevant to the current task.

41
Q

Steps of Cognitive Therapy

A
  1. ) therapist guides patient in identifying and correction cognitive distortions.
  2. ) patients keeps journal and gathers data on their beliefs,