psychology psychopathology Flashcards

1
Q

what are the 4 definitions of abnormality

A

statistical infrequency- when an individual has a less common characteristic

deviation from social norms
- behaviour that is different from the accepted standards withing society

failure to function adequetely - occurs when someone is unable to cope with day to day living

devaiation from ideal mental health- not meeting criteria for ideal mental health

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2
Q

what are the two treatments for phobias?

A

systematic desensitization is gradually reducing phobic anxiety through classical conditioning (learn to relax in the presence of the phobic stimulus)

flooding - immediate exposure to phobic stimulus, fightening with not build up

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3
Q

what are the 3 processes involved in systematic desensitization

A

anxiety heirarchy made by patient and therapist, a list of situations concerning the phobia from least to most frightening

relaxation - therapist teaches patient to relax as deeply as possible e.g through breathing excercises

exposure, patient is exposed to the phobic stimulus while in a relaxed state across multiple sessions from the bottom of the heiracrhy moving up

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4
Q

what does flooding lead to

A

extinction - we learn the phobic stimulus is harmless if we are unable to avoid it

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5
Q

evaluation of phobia treatments

A

Behavioural treatments dont treat the underlying cause of phobias (irrational beliefs) but just symptoms like panic so cbt may be better as it targets the underlying cause.

research supporting sd.gilroy followed 42 patients treated for arachnophobia with 3 sessions of sd compared to a control group with no exposure and just relaxation. groups were assessed at 3 months and 33 months and SD group showed less fear at each point. this leads to LT solutions

a strength of flooding is its quick and cost effective. this is due to the immediate exposure to the stimulus so it can take up to one session. patients can be free of their symptoms asap

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6
Q

what is the two process model

A

this states that phobias are aquired through classical conditioning and maintained through operant conditioning.

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7
Q

explain classical conditioning of phobias with the white rat

A

loud noise (ucs) -> fear response (ucr)

white rat + loud noise (ns + ucs) -> fear response (ucr)

white rate (cs) -> fear response (cr)

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8
Q

how are phobias negatively reinforced

A

The behaviour is strengthened because the unpleasant consequence is removed.

we continue to maintain a behaviour (running away) in order to avoid the negative consequence (phobic stimulus)

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9
Q

what does biological preparedness suggest

A

innate predisposition to aquire certain fears (e.g heights and deadly animals) which will increase our chances of survival

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10
Q

evaluations of phobia explanations

A

two process model cant explain all phobias. evolutionary phobias dont have to be learned through classical conditioning . outline biological preparedness. theres more to acquiring phobias than conditioning.

weakness is the behavioural approach to phobias doesnt explain cognitive
characteristics e.g selective attention and irrational beliefs. behaviourist approach doesnt consider the role of internal mental processes. cbt may be better

real life application to support behaviourist approach. watsona nd rayner taught little albert a phobia of white rats by repeatedly pairing a loud bang noise with the neutral stimulus of the white rat which then became associated with the fear of the loud bang

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11
Q

behavioural characteristics of phobias

A

avoidance

panic

endurance

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12
Q

emotional characteristics of phobias

A

anxiety and fear

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13
Q

cognitive characteristics of phobia

A

cognitive distortions

irrational beliefs

selective attention to the phobic stimulus

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14
Q

behavioural symptoms of ocd

A

compulsions and avoidance

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14
Q

emotional symptoms of ocd

A

anxiety, distress, depression, guilt and disgust

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15
Q

cognitive symptoms of ocd

A

obsessive thoughts e.g catastrophic thinking, hyper vigilance, insight into excessive anxiety and thoughts

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16
Q

aol for the genetic explanation ocd

A

individuals inherit specific genes from their parents that make them vulnerable to the onset of ocd

polygenic - taylor found up to 230 genes involved

COMT gene is common in ocd patients. low activity of COMT gene means high dopamine

sert gene affects the transport of serotonin which causes low levels of serotonin linked with ocd

17
Q

evaluation of the genetic explanation of OCD

A

theres support for genetic. nestadl et al in their twin studies found that 68% of mz twins shared ocd whilst only 31% of dz twins did. closer related means more chance of ocd.however the mz twins could be more similar in terms of shared environments so env. factors may play more of a role, we cant condclude that its genes.

environmental risk factors may increase risk of ocd. cromer et al found more than half patients had a severe traumatic event so ocd cant be entirely genetic and theres a greater risk if theres an environmental trigger. diathesis stress model biological reductionism

18
Q

neural explanation of ocd : neurotransmitters

A

Abnormal levels of neurochemicals like dopamine and serotonin are associated with the abnormal transmission of mood related information and obsessive thoughts.

people with OCD have:
high dopamine and low serotonin which leads to low moods –> accompanying depression

19
Q

evaluations of the neural explanation of OCD

A

theres an issue with cause and effect. explanation says cause for ocd is the abnormal functioning of the lateral frontal lobe causes impaired decision making. however what if the ocd is what cause the abnormal functioning of lfl

real life application. some anti depressants work by increasing serotonin. Drugs help reduce ocd symptoms proving low levels of serotonin is included in ocd. however lots of people with ocd also have depression so these low serotonin levels could be that they have depression aswell and they are just treating derpression

20
Q

Explain the process of the neurotransmitter serotonin and how do ssris stop reuptake

A

When serotonin is released from the pre-synaptic neuro into the synapse, it travels to the receptor sites on the post-synaptic neuron. Serotonin which is not absorbed into the post-synaptic neuron is reabsorbed into the pre-synaptic neuron.

SSRIs increase the level of serotonin available in the synapse by preventing it from being reabsorbed into the presynaptic neuron. This increases level of serotonin in the synapse and results in more serotonin being received by the post-synaptic neuron

21
Q

ao3 evaluation for the treatment of ocd

A

Drug therapy is supported by research. Soomro et al found that SSRI’s were more effective than placebos. however they found the best results when ssris were combined with cbt. however evidence may be biased by drug companies who dont publish all of their answers.

drugs are quick and cheap. ssri perscription is £9 when cbt is £60 a week. ssris are non disruptive to the patients daily life cbt requires time in a therapy session. however once they stop taking ssris theyre more likely to relapse so its not a long term solution

22
Q

behavioural symptoms of depression

A

over and under eating

reduced activity levels

disruption of sleep

insomnia

agression and self harm e.g cutting and irritable

23
Q

cognitive symptoms of depression

A

poor concerntration

dwelling on the negative

absolutist thinking

23
emotional symptoms of depression
belief of being worthless hopeless and inadequete low self esteem anger/lowered mood
24
ao1 explain becks cognitive theory of depression
beck believes people are vulnerable to depression dt: faulty information processing :only focusing on the bad side of things negative self schemas: interpret everything about themselves badly negative triad: negative views about the world negative views about the future negative views about oneself
25
what does cbt and Rebt do?
cbt identifies the irrational belief and then challenges it rebt is a form of cbt abc + de for dispute and effect aim: break the link between negative life event and depression logical argument - dispute using facts empirical argument - dispute if theres evidence to back up their beliefs
25
ao1 explain ellis' abc model
A- activating event e.g poor grade B - beliefs e.g i will never succeed c- consequences e.g feeling worthless and depressed musturbation - belief we must always succeed utopianism - belief life should always be fair
26
ao3 evaluation of becks cognitive theory of depression
evidence to support.grazioli and terry assessed 65 pregnant women for cognitive vulnerability and depression before and after birth. They found that those women judged to have been high in cognitive vulnerability were more likely to suffer post-natal depression. however not representative one group of women. doesnt explain all depression. some patients have cotard syndrome and hallucinations. becks theory is incomplete as it cant explain these symptoms like a biological explantation can.
27
ao3 evaluation for ellis abc model
partial explanation. it states depression is caused by an activating agent but that only explains reactive symptoms of depression in response to an event when its not always clear what lead to someones depression at a given time beck and ellis contributed to treatments for depression. cbt is identifying and challenging irrational thoughts. rebt utilises abc adding dispute and effect to breakdown evens and thoughts. march found that Cbt was effective at treating symptoms and risk of relapse at 81%
28
A01 depression treatments
cognitive therapy uses thought catching and challenging it. reality testing : clients have to gather evidence e.g when they enjoyed an event or recieved praise and compare evidence with thought to t=see if they match. This is used to prove clients wrong. rebt behavioural activation : activities to improve mood
29
A03 evaluations of treatment of DEPRESSION
Cbt takes time and is expensive. average of 12 sessions and £60 per session and they may have to miss work to attend so ssris at £9 may be better. However cbt is cheaper in the long term as its done in 12 sessions but drugs are forever. cbt may not be effective for sever and disabled cases. It requires complex rational thinking and with a lack of motivation this may be hard to engage with making them feel more hopeless. cbt has economic impacts. depression is costly to the economy through absenteeism , low productivity and unemployment. In the long term Cbt decreases risk of relapse and helps people go back to work.
30
what are the a03 evaluations of statistical infrequency
doesnt distinguish between desiraable and undesirable behaviours. it suggests behaviour shown less often is abnormal but some abnormal behaviours are desirable like having an IQ over 150 though very few people do we would say its undesirable and some normal behaviours are undesirable like depression. Real life application SI is used to assess the severity of symptoms e.g iq below 70 means intellectual disability.beck depression inventory a score of 30+ means sever depression
31
a03 evaluations of deviation from social norms
incomplete definition. in uk hearing voices and hallucinating is unacceptable whereas it may be normal as in african cultures they believe they're communicating with ancestors.its not universal useful in clinical practice.recklessness, aggression, violating the rights of others. These signs of the disorder are all deviation from social norms. ​acts as a criteria for mental illeness
32
a03 evaluation of deviation from ideal mental health
this is unrealistic. no one can master their environment everyday and not be insecure some days not a universal definition because collectivstic cultures dont really value self actualization and autonomy thats mainly westeren cultures
33
a03 evaluation for failure to function adequetely
this may be deviation from social norms. if a person doesnt live a normal life e.g hold down a job they may be abnormal however are they failing to function or deviating from social norms e.g not having a job or permanent life could be an alt lifestyle like new age travellers. represents a threshold for professional help. most people carry on with life despite 25% of people having mental health problems this criterion allows people who cant function to get professional help as they need it the most.
34
neural explanation of ocd: abnormal brain structures
OCD is associated with impaired decision making and illogical thinking which could be due to abnormal functioning of the lateral frontal lobes of the brain There is also evidence that the left Para hippocampal gyrus, associated with processing unpleasant emotions, functions abnormally in OCD patients
35
neural explanation of ocd: abnormal brain circuits
people with ocd have a damaged caudate nucleus which suppresses worry signals from the orbitalfrontal cortex. when the caudate nucleus is damaged it fails to supress minor worry signals from the OFC and the thalamus is alerted which sends signals back to the OFC acting as a worry circuit PET scans show when a person with a germ obsession holds a dirty cloth, there is heighted activity in the OFC.
36
What are 2 treatments for OCD other than serotonin
Tricyclics block the transporter mechanism that re absorbs both serotonin and noradrenaline, prolonging their activity in the synapse SNRI's increase the levels of serotonin and noradrenaline and are tolerates by those for whom ssris are not effective for
37
outline the 6 aspects to jahodas criteria
positive attitude towards the self self actualization autonomy mastery of the environment accurate perception of reality resistance to stress