psychology psychopathology Flashcards
what are the 4 definitions of abnormality
statistical infrequency- when an individual has a less common characteristic
deviation from social norms
- behaviour that is different from the accepted standards withing society
failure to function adequetely - occurs when someone is unable to cope with day to day living
devaiation from ideal mental health- not meeting criteria for ideal mental health
what are the two treatments for phobias?
systematic desensitization is gradually reducing phobic anxiety through classical conditioning (learn to relax in the presence of the phobic stimulus)
flooding - immediate exposure to phobic stimulus, fightening with not build up
what are the 3 processes involved in systematic desensitization
anxiety heirarchy made by patient and therapist, a list of situations concerning the phobia from least to most frightening
relaxation - therapist teaches patient to relax as deeply as possible e.g through breathing excercises
exposure, patient is exposed to the phobic stimulus while in a relaxed state across multiple sessions from the bottom of the heiracrhy moving up
what does flooding lead to
extinction - we learn the phobic stimulus is harmless if we are unable to avoid it
evaluation of phobia treatments
Behavioural treatments dont treat the underlying cause of phobias (irrational beliefs) but just symptoms like panic so cbt may be better as it targets the underlying cause.
research supporting sd.gilroy followed 42 patients treated for arachnophobia with 3 sessions of sd compared to a control group with no exposure and just relaxation. groups were assessed at 3 months and 33 months and SD group showed less fear at each point. this leads to LT solutions
a strength of flooding is its quick and cost effective. this is due to the immediate exposure to the stimulus so it can take up to one session. patients can be free of their symptoms asap
what is the two process model
this states that phobias are aquired through classical conditioning and maintained through operant conditioning.
explain classical conditioning of phobias with the white rat
loud noise (ucs) -> fear response (ucr)
white rat + loud noise (ns + ucs) -> fear response (ucr)
white rate (cs) -> fear response (cr)
how are phobias negatively reinforced
The behaviour is strengthened because the unpleasant consequence is removed.
we continue to maintain a behaviour (running away) in order to avoid the negative consequence (phobic stimulus)
what does biological preparedness suggest
innate predisposition to aquire certain fears (e.g heights and deadly animals) which will increase our chances of survival
evaluations of phobia explanations
two process model cant explain all phobias. evolutionary phobias dont have to be learned through classical conditioning . outline biological preparedness. theres more to acquiring phobias than conditioning.
weakness is the behavioural approach to phobias doesnt explain cognitive
characteristics e.g selective attention and irrational beliefs. behaviourist approach doesnt consider the role of internal mental processes. cbt may be better
real life application to support behaviourist approach. watsona nd rayner taught little albert a phobia of white rats by repeatedly pairing a loud bang noise with the neutral stimulus of the white rat which then became associated with the fear of the loud bang
behavioural characteristics of phobias
avoidance
panic
endurance
emotional characteristics of phobias
anxiety and fear
cognitive characteristics of phobia
cognitive distortions
irrational beliefs
selective attention to the phobic stimulus
behavioural symptoms of ocd
compulsions and avoidance
emotional symptoms of ocd
anxiety, distress, depression, guilt and disgust
cognitive symptoms of ocd
obsessive thoughts e.g catastrophic thinking, hyper vigilance, insight into excessive anxiety and thoughts
aol for the genetic explanation ocd
individuals inherit specific genes from their parents that make them vulnerable to the onset of ocd
polygenic - taylor found up to 230 genes involved
COMT gene is common in ocd patients. low activity of COMT gene means high dopamine
sert gene affects the transport of serotonin which causes low levels of serotonin linked with ocd
evaluation of the genetic explanation of OCD
theres support for genetic. nestadl et al in their twin studies found that 68% of mz twins shared ocd whilst only 31% of dz twins did. closer related means more chance of ocd.however the mz twins could be more similar in terms of shared environments so env. factors may play more of a role, we cant condclude that its genes.
environmental risk factors may increase risk of ocd. cromer et al found more than half patients had a severe traumatic event so ocd cant be entirely genetic and theres a greater risk if theres an environmental trigger. diathesis stress model biological reductionism
neural explanation of ocd : neurotransmitters
Abnormal levels of neurochemicals like dopamine and serotonin are associated with the abnormal transmission of mood related information and obsessive thoughts.
people with OCD have:
high dopamine and low serotonin which leads to low moods –> accompanying depression
evaluations of the neural explanation of OCD
theres an issue with cause and effect. explanation says cause for ocd is the abnormal functioning of the lateral frontal lobe causes impaired decision making. however what if the ocd is what cause the abnormal functioning of lfl
real life application. some anti depressants work by increasing serotonin. Drugs help reduce ocd symptoms proving low levels of serotonin is included in ocd. however lots of people with ocd also have depression so these low serotonin levels could be that they have depression aswell and they are just treating derpression
Explain the process of the neurotransmitter serotonin and how do ssris stop reuptake
When serotonin is released from the pre-synaptic neuro into the synapse, it travels to the receptor sites on the post-synaptic neuron. Serotonin which is not absorbed into the post-synaptic neuron is reabsorbed into the pre-synaptic neuron.
SSRIs increase the level of serotonin available in the synapse by preventing it from being reabsorbed into the presynaptic neuron. This increases level of serotonin in the synapse and results in more serotonin being received by the post-synaptic neuron
ao3 evaluation for the treatment of ocd
Drug therapy is supported by research. Soomro et al found that SSRI’s were more effective than placebos. however they found the best results when ssris were combined with cbt. however evidence may be biased by drug companies who dont publish all of their answers.
drugs are quick and cheap. ssri perscription is £9 when cbt is £60 a week. ssris are non disruptive to the patients daily life cbt requires time in a therapy session. however once they stop taking ssris theyre more likely to relapse so its not a long term solution
behavioural symptoms of depression
over and under eating
reduced activity levels
disruption of sleep
insomnia
agression and self harm e.g cutting and irritable
cognitive symptoms of depression
poor concerntration
dwelling on the negative
absolutist thinking