Psych Flashcards

1
Q

Incomprehensibility (define)

A

When the general public can’t understand the motive behind someone’s behavior (Horowitz)

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2
Q

Cultural Relativity (define)

A

When the “normality” of behaviors is determined by a culture (wailing in mourning vs silent coping)

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3
Q

Dorothea Dix

A

Developed the concept of asylum (sanctuary), provided food, shelter, clothing to mentally ill.

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4
Q

What was the unintended consequence of deinstitutionalization

A

Thousands of mentally ill concentrated in prisons, state hospitals, homeless, and nursing homes.

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5
Q

Risk factors for violence among the mentally ill

A

History of violence
Command Auditory Hallucinations
Drug/Alcohol Abuse
Not compliant with treatment (or out of treatment)

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6
Q

Peplau believed…

A

That the work of psych nurses is interpersonal, and that using the relationship between nurse and patient can help move them towards wellness.

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7
Q

What are Peplau’s five roles?

A
Teacher
Resource
Counselor
Technical Expert
Surrogate
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8
Q

Transference

A

When a patient takes their baggage, especially feelings about a specific significant person, and places it upon the nurse/therapist.

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9
Q

Countertransference

A

When a nurse/therapist takes their history/baggage and it colors their perspective/opinions about the patient.

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10
Q

Milieu Therapy: Assumptions

A

Every individual has the ability to be healthy/realize health.
Every interaction is an opportunity for therapeutic intervention.
The client owns his own environment.
The client is accountable for his own behavior.
Inappropriate behavior comes with logical repercussions.

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11
Q

Milieu Therapy: Characteristics

A

Meeting of the patient’s basic physical needs
Facilities are home-like
Structured program of social and work-like activities
Community and family are involved
Democratic government in which patient is encouraged to participate.

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12
Q

What is the role of the nurse in the psychiatric setting?

A
Meeting the patient’s physiological needs
Provides reality orientation
Medication Administration
Therapeutic Interactions
Teacher 
Provide Hope
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13
Q

What are the recovery models and what are their shared components?

A

They’re based on the idea that recovery is possible.
The desire is for the patient to manage their illness the best as they are able.
The patient is provided a safe place to live.
The patient needs to feel as though they have meaningful activities.
The patient needs a social network for support.

Tidal Model
WRAP model.

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14
Q

What is the Tidal Model?

A

Recovery Model that uses the metaphor of water.
Focuses on the patient’s story, builds on their strengths.
Recognizes that change is constant (comes in waves)

Nurse should:

  • spend time with patient
  • be transparent.
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15
Q

What is the WRAP model

A

Wellness Action Recovery Plan (recovery model).

Structured process with concrete steps:

  • Builds a “wellness toolkit”
  • involves patient identifying their triggers/early warning signs and what tools they can use if it occurs.
  • making a maintenance list and reviewing it daily
  • spells out how to recognize when the condition worsens
  • incorporates crisis planning: how friends/family can manage so as not to call 911.
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16
Q

What are the four major groups of neurotransmitters?

A

Cholinergics
Monoamines
Neuropeptides
Amino Acids

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17
Q

What’s the basic physiology of neurotransmitter release?

A

Neurons talk to each other across the synaptic space
AP fires and the NT is released into the synaptic cleft
NT attaches to receptors on the postsynaptic membrane
AP continues to the next nerve cell
(Presynaptic neuron “reuptakes” extra/leftover NT for recycling).

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18
Q
What NT is the primary cholinergic? 
Is it excitatory or inhibitory?
Where is it located? 
What is it synthesized from? 
What does it do? 
What disease processes are associated with it?
A

Acetylcholine. (Synthesized from choline)
Both excitatory and inhibitory.
Location: Mostly PNS (also brain and spinal cord) - NMJs of skeletal muscles, especially.
Coordination of movement, sleep, arousal, pain, perception, memory retention.

Increased in: depression
Decreased in: Alzheimer’s, Huntingdon’s, Parkinsons.

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19
Q

What are the monoamines?

A

Norepinephrine
Dopamine
Serotonin/5HT
Histamine

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20
Q
What type of NT is neuroepinephrine?
What is NE synthesized from? 
Is it inhibitory or excitatory? 
Where is it synthesized? 
What are its functions? 
What disorders are associated with it?
A

Monoamine.
Synthesized from Tyrosine
Excitatory
Synthesized in pons, medulla, lymbic system, hippocampus (more?)
Functions: Mood, cognition, attention/vigilance, perception, cardiovascular function, sleep.

Increased in: anxiety, mania, schizophrenia

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21
Q

What metabolizes norepinephrine? What drugs prevent this?

A

Monoamine oxidase

MAO inhibitors

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22
Q
What kind of NT is dopamine? 
What is it synthesized from? 
Where is it located? 
Is it excitatory or inhibitory? 
What does it do? 
What disorders is it associated with?
A

Monoamine
Synthesized from Tyrosine
Located in: brainstem (mostly), especially at the substantia nigra
Usually excitatory. (Inhibits prolactin release: gynecomastia)
Controls complex movements, motivation, cognition, regulates emotional responses, involved in pleasure.
Associated with addictions, movement disorders, psychosis.
Increased in mania and schizophrenia
Decreased in depression and Parkinson’s

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23
Q
What kind of NT is Serotonin? 
What is it synthesized from? 
Where is it located? 
Is it inhibitory or excitatory? 
What are its jobs? 
What disorders is it associated with?
A

Monoamine
From tryptophan
Located in the brain (especially at the raphe nuclei of the brainstem)
Mostly inhibitory
Plays a role in sleep/arousal, libido, appetite, mood, aggression, pain perception, temperature regulation.
Decreased in depression
Increased in anxiety

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24
Q

What kind of NT is Histamine?

What does it to?

A

Monoamine
Plays role in mediating allergic reactions
Associated with wakefulness

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25
Q

What are the Amines?
What are they synthesized from?
Where are they located?
What functions are they associated with?

A

GABA, Glycine (Inhibitory)
Glutamate, Aspartate (Excitatory)

Synthesized from tyrosine, tryptophan, histidine
Found in the brain
Involved in learning, emotions, motor control

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26
Q
What type of NT are GABA and Glycine? 
What do they do? 
What are they synthesized from? 
Where are their receptors located? 
What drug class is GABA associated with?
What disorders are they associated with?
A

Amines
GABA is the major NT of post-synaptic inhibition: it interrupts the impulse at the synaptic junction.
(Synthesized from glutamate?)
Most CNS neurons have receptors
Associated with Benzodiazepines
Decreased in Anxiety, Schizophrenia, some Epilepsy, Huntingdon’s

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27
Q
What type of NT are Glutamate and Aspartate? 
Are they excitatory or inhibitory? 
What do they do? 
Where are they located? 
What is the risk? 
What disorders are they associated with?
A

Amines
Excitatory
Relay sensory info and regulate motor/spinal reflexes
Synthesize structural/functional proteins
Located in the CNS
Too much can be neurotoxic
Decreased in Schizophrenia
Increased in anxiety, depression, temporal lobe epilepsy

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28
Q

What are Neuropeptides? Examples?

What does “second messenger” mean?

A

Opioid peptides (endorphins and encephalines)
Substance P
Somatostatin

They are “Second messengers”: they modulate the messages of NTs that aren’t peptides.

29
Q

What type of NTs are Endorphins and Enkephalines?
Where are they located?
What do they do?
Are they inhibitory or excitatory?

A
Neuropeptides (opioid peptides)
Located in the CNS and GI tract
Endorphins help us deal with stress
Enkephalines help reduce pain
They’re generally inhibitory.
30
Q
What is Substance P? 
Is it excitatory or inhibitory?
What is it’s job? 
Where is it located? 
What drug interrupts its release?
What disorders are associated with it?
A

Neuropeptide
Excitatory
Works in the transmission/regulation of pain
Located in the spinal cord, brain, sensory neurons associated with pain, GI tract, salivary glands
Interrupted by morphine
Increased in the CSF of patients with depression and PTSD.

31
Q

Which NTs are most closely associated with anxiety disorders?

A

Serotonin (5HT), Norepinephrine, GABA

32
Q

How do Monoamine Oxidase inhibitors work?

A

They prevent the intracellular destruction of monoamines (NE, 5HT, DA)

33
Q

How do Donepezil and Rivastigmine work?

A

They inhibit acetylcholineesterase in the synapse, thereby increasing available acetylcholine.

34
Q

How do tricyclics work?

A

They block the reuptake of norepinephrine and 5HT/Serotonin

35
Q

How do antipsychotics work?

A

They block dopamine receptors on the post-synaptic neurons.

36
Q

How do antianxiety drugs work (generally)

A

They activate GABA receptors/potentiate GABA.

37
Q

How are norepinephrine and dopamine related?

A

Dopamine is the precursor to catecholamines.

38
Q

Anxiety basics

A

Subjective experience detectable by objective behaviors.
SNS response to a perceived threat.
Psychological (thoughts) and physiological (NTs as well as Catecholamines).

39
Q

Define Anxiety Continuum

A

Anxiety exists along a continuum, from mild through panic. At the mild end it can be useful and isn’t pathologic. At the panic end it is debilitating.

40
Q

Mild Anxiety

A
Helpful/Adaptive
No intervention needed
Increased: 
-learning 
-alertness
-perception
-motivation
41
Q

Moderate Anxiety

A
Could be appropriate (depending on the threat)
Decrease in: 
-perceptual field
-alertness
-learning
-attention span
-concentration
42
Q

Severe Anxiety

A

Could be appropriate if threat is severe, as well.

Greatly decreased perceptual field. Focus on very specific details.
No:
Concentration
Effective learning

43
Q

Panic

A
NO: 
Focus
Learning
Concentration
Comprehension. 

Misperceptions and sense of doom.

44
Q

Defense mechanisms arise at what point(s) in the anxiety continuum?

A

Mild-Moderate

45
Q

Migraines, heart arrythmias and IBS arise from an extended time in what section(s) of the anxiety continuum?

A

Moderate to Severe

46
Q

Chronic supression of severe anxiety can lead to:

A

Psycho neurotic behavior patterns

47
Q

PTSD symptoms

A

Reliving trauma
Mental Stupor
Irritability
Nightmares

48
Q

Simple Phobias

A

Fear of blood/snakes/elevators/flying/etc

49
Q

Social Phobias

A

Fear of humiliation

Can manifest as fear of public speaking, fear of public toilets…

50
Q

Panic Disorder symptoms

A

Fear of dying, sense of doom
Chest pain
Shortness of breath

51
Q

Mixed anxiety/depressive disorder symptoms

A
Anxiety
Inner tension
Depression
Increased Aggression
Hostility
52
Q

OCD symptoms

A

Obsessions (fears) and compulsions (to help to relieve the fears). Hallucinations.
Can be fear of germs, public toilets.
Can be sexual or murderous thoughts.

53
Q

What are the four names we learned for anxiety disorders with physical manifestations?

A

Factitious disorder
Malingering
Somatic Symptom Disorder
Conversion Disorder

54
Q

What is a factitious disorder? Is it intentional or unintentional?

A

Intentional production of symptoms in order to attain primary gain (usually treatment/medication/high)

55
Q

What is Malingering? Is it intentional or unintentional?

A

Intentional production of symptoms made up for secondary gain (like a warm bed on a freezing night).

56
Q

What is somatic symptom disorder? Is it intentional or unintentional?

A

Somatic symptoms (like a stomachache) not related to the disorder that the person believes is caused by something else (staples in the stomach). No loss of function.

57
Q

What is conversion disorder? Is it intentional or unintentional?

A

Person actually loses function in a part of their body but no physiological cause exists. (Not intentional).

58
Q

What are some anxiety screening tools? When/why are they used?

A

Usually used in outpatient treatment. Good for establishing a baseline and marking progress (anxiety skews perception in the moment).

GAD-7
Hamilton Anxiety Screen (HAMa)
Burns Anxiety Screen
Sheehan Anxiety Scale
Pediatric Anxiety Scale
MOCI
59
Q

Behavioral Responses to Anxiety (Observable Signs)

A
Pacing
Hand-wringing
Hyper vigilance
Increased motor movement
Increased Startle Reflex
Difficulty problem-solving
Clenching jaw
Increased heart rate/BP respiration’s
60
Q

Physiological experience of anxiety (patient-experienced symptoms)

A
Palpitations
Trembling, Shaky
Chest pain/pressure
Paresthesias (numbness/tingling)
Faintness
Irritability/Restlessness
Derealization (doesn’t feel real)
Dread/Doom
Decreased concentration
Decreased appetite
Gut pain, IBS
Dry mouth
Lump in the throat
61
Q

Psychological responses to anxiety (Defense mechanisms)

A
Denial
Displacement
Rationalization
Intellectualization
Regression
Repression
Suppression
Dissociation
Obsessions
Compulsions
Abreactions/Flashbacks
62
Q

Define Displacement

A

Placing feelings (and associated actions) on someone other than the original target (yelling at the nurse because of anger at the doctor).

63
Q

Define Intellectualization

A

Using logic, reasoning and analysis to avoid addressing actual emotions.

64
Q

Define Repression

A

Involuntary blocking of feelings/experiences (can’t remember)

65
Q

Define suppression

A

Voluntary blocking of feelings/experiences (“I don’t want to think about that now”).

66
Q

Define Dissociation

A

No longer present - checked out completely.

67
Q

Define Abreactions/Flashbacks

A

Relieving an experience and the subsequent emotional release.

68
Q

Anxiolytics

A

Antianxiety medications. Most depress subcortical levels of the CNS (except buspirone).

69
Q

SSRIs (action, names, onset, side effects)

A

Increase serotonin in the synapse by slowing down the reuptake and recycling by the presynaptic neuron.

Onset: slow (3-4 weeks), peak effect (6 weeks).
Side effects: sedation (paroxetine), nausea, restless legs (fluoxetine)
Paroxetine
Citalopram
Fluoxetine
Sertraline
Escitalopram