Patient viewing wrap up Flashcards

1
Q

5 risks for skin CA

A
  1. skin type
  2. sun exposure
  3. Fam Hx
  4. immunosupresion
  5. tanning beds
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

3 key dermatitis mgmt points

A
  1. Tx inflam
  2. moisture
  3. prevent/tx infection
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

6 systemic diseases with psoriasis

A
Psoriatic arthritis – up to 30% of patients
Lymphoma (e.g. Hodgkin’s, CTCL etc)
Depression
IBD
Uveitis 
Metabolic Syndrome
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

5 biopsy complications

A
infection
dehiscence
recurrence
bleeding 
pain
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

sun exposure diffs for BCC and SCC

A

BCC - intermittetn and intense

SCC - cumulative

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

5 types of non-scarring alopecia

A

Telogen effluvium – increased shedding of otherwise normal hairs in response to stress such as surgery, pregnancy, malnutrition, etc.
Androgenetic – male-pattern and female-pattern hair loss
Alopecia areata – autoimmune process
Anagen effluvium – classically caused by chemotherapy
Trichotillomania – self-induced plucking or breakage of hair

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

potential triggers of telogen effluvium

A

Post-surgery
Post partum
After severe illness (especially febrile)
Severe nutritional deficiency (extreme dieting, malnutrition)
Severe psychological trauma (rare)
Chronic illness can lead to chronic telogen effluvium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

5 signs of scarring alopecia

A
burning,
pain +/- itching 
erythema
perifollicular scale
atrophy, loss of follicular ostia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

2 types of pruritus

A

skin disease

systemic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

systemic causes of pruritus

A

Kidney disease
Liver disease (especially obstructive/cholestatic liver disease)
Medications (ASA, morphine, other opiates)
Thyroid disease (both hyper and hypothyroidism)
Malignancy especially lymphoma
Polycythemia vera
Infections: HIV, Hepatitis B and C, intestinal parasitism
Connective tissue disease
Neuropathic itch (point out that this is usually not generalized), which can be caused by nerve compression, posthepetic
Skin dryness or xerosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

4 clues to venous ulcer

A

location
underlying stasis changes/lipdermatosclerosis
edema
varicosities

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Tx of stasis

A

Most important treatment is COMPRESSION
Leg elevation
Topical steroids
Wound care

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

signs of dermatomyositis

A
  • Poikiloderma with “shawl sign” on the V of chest/upper back
  • Heliotrope sign: violaceous eruption around the eyes
  • Gottron’s papules: lichenoid lesions over the knuckles
  • Nail fold changes: cuticle dystrophy, nail fold telangiectasias etc.
  • Mechanics hands
  • Holster sign: rash to hips
  • Calcinosis cutis
  • Non scarring alopecia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

MDSOAPBRAIN for lupus

A

*Malar rash
*Discoid rash
Serositis (pleuritis or pericarditis)
Arthritis (non erosive, > or equal to 2 peripheral joints)
*Oral or nasopharyngeal ulcers
*Photosensitivity
Hematologic disorder including:
Renal dysfunction
ANA positivity
Immunological studies with positivity in 1 or more of:
* Anti-DsDNA (correlates with disease activitity) OR
Anti-Smith OR
Anti-cardiolipin antibodies, lupus anticoagulant or false positive VDRL
Neurological disease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly