Parasitology - Lecture 12 (Miscellaneous Nematodes) Flashcards

1
Q

When considering Gi nematodes, discuss stomach worms

A
  • ollulanus
  • Physaloptera
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What type of stomach worm is Ollulanus and discuss the basics

A
  • Cat stomach worm
  • Usually feral cats and catteries
  • Transmission: Directly transmitted by ingestion of larvae or adults in vomitus (No fecal transmission)
  • internal autoinfection –> entire life cycle can occur in the stomach > number of worms keeps increasing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Discus the pathologic effects, diagnosis, and treatment of Ollulanus

A
  • Pathologic effects and clinical signs:
    - Heavy burdens - chronic gastritis and vomiting
    - auto infectiion can result in hhhigh worm burden
    - can result in emacation and death if left untreated
  • Diagnosis: demonstrate tiny worms in fresh (nonrefrigerated) vomitus by Baeerman
  • Endoscopic biopsies

Treatment: 5 day course of benbendazole
Tretramisole

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Discuss the basics of Phylasoptera

A
  • stomach worm of dogs and cats
  • Larvated egg shed in feces
  • L3 develops in bettle/crickets
    • required intermediate host
  • Intermediate host can be eaten by birds or rodents (paratenmic host)
    -Paratenic or intermediate host eaten by cat/dog
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Discuss the pathologic effects, diagnosis and treatment od phylasoptera

A
  • Pathologic effects and clinical signs: chronic gasttritis and chronic vomiting
  • Diagnosis: fecal floats often dont work; **direct smear ** is better for fidning eggs; adults in vomitus; **endoscopy **
  • Treatment: Pyrantel pamoatel; fenbendazole
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Which nematode lives in the small intestine

A

Strongylkoides

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Discuss strongyloides

A
  • intestinal threadworm in small intestine
  • Complicated life cycle
  • Propogation of infectious L3 by envionmental stages leads to highky contaimnated enviironments
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Discuss the clinical siogns of strongyloides

A
  • Usually asymptomatic –> immunity develops in immuncompetent hosts
  • Puppies and kittens are highly susceptible
  • Possible clinical signs:
    - dermatits due to L3 penetration
    - Bronchopneumonia due to lung migrations
    - Adults in intestine watery diahhrea, dehydrartion
  • Immune sytem controls infection so **immunosuppression ** can lead to hyperinfection resulting in emacation and death
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Discuss the diagnosi, treatment, and zoonosis for Strongyloides

A

In short:
- diagnosis: fecal float to look for larva in feces
- Treatment: ivermectin, fenbendazole

Control: dry environment: free living stages are susceptible to cold and dry

potentially zoonotic - can be transmitted from dogs to humans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Which nematode lives in the large intestine

A

Trichuris vulpis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Discuss trichuris vulpis

A

Trichuris vulpis –> whipworm
- whip shaoed worm in large intestine of **dog ** and cat
- Thin hair like anterior and embeds in the large intestinal mucosa

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Discuss the trichuris vulpis lifecycle

A

Transmission: ingestion of larvated egg from environment
Egg shed in feces larvate in environment:
- takes about 1 month in the environment to become infectious

Eggs hatch when eaten, larvae develop to adults in intestine
- PPP is 3 months

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Discuss the clinical signs and diagnosis of trichuris

A

Pathologic effects and clinical signs:
- usually asymptomatic
- large numbrs can cause bloody diahhrea, weight loss, dehydration

Diagnosis: Fecal floats –> floats well but egg outpout is often low
Antigen ELISA –> if no eggs on float but you supect Trichuris, a **fecal ELISA test ** is available

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Discuss the treatment of trichuris

A

Treatment: Fenbendazole; macrocyclic lactones –> milbemycin, moxdectin

  • Larva are not suspectibe to anthelmintics
  • Control is difficult –> eggs survive in enviornment for a long time and dogs tend to get reinfected
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Discuss the different lungworms in dogs and cats

A

Dogs: Oslerus and Eucoleus
Cats: Aelurostrongylus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Discuss Oslerus Osleri

A
  • Dog lungworm –> found in nodules at bifurcation of trachea
  • Life cycle: Female worm deposits egg in trachea which hatches immediately > L1 coughed up and swallowed > **infectious L1 larvae ** are shed in feces
  • Direct transmission by ingesdtion of feces
    - Major mode of infection = nursing pups when dam licks them and L1 transferred vai septum
17
Q

Discus the clinical signs, diagnsois, and treatment of Osleri osleri

A
  • Clinical signs: hard dry cough broughton by the cold or exercise; nodules can eventually obstruct air passages
  • Diagnosis: bronchoscopy –> nodules are pathognomic; tracheal wash; fecal floatation: look for LARVAE IN FECES

Treatment: –> difficult; goal is to **reduce nodule size and lcinical signs ** –> cure if often times not achieved;
- fenbendazole
- ivermectin

18
Q

Discuss Aelurostrongylus

A

Cat lungworm: relatively common; found in the lung parenchyma

19
Q

Discuss the life cycle of Aelurostrongylus

A
  • L1 shed in feces (not infectious)
  • L2 developsin snail/slug
    - required intermediate host
    • Intermediate host eaten by birds/rodents/reptiles (**paratenic hosts)*
  • paratenic or intermeidate host is eaten by cat
20
Q

Discuss the clinical signs, diagnosis, and treatment of Aelurostyrongylus

A
  • Clinical signs:
    - asymptomatic
    - moderate: coughing to anorexia
    - Severe: coughing, panting, shortness, of breath, death

Diagnosis: fecal float to find larvae in feces
Baeerman to look for L1 in larvae in feces

Treatment: Fenbendazole
Ivermectin, selamectin - 2 treatments one month apart

21
Q

Discuss the Eucoleus species

A
  • E. aerophila - bronchial capillarid primarily in foxes (also dogs and cats); clinically inapparent to mild respiratory signs; cough; nasal discharge
  • E. boehmi –> nasal capillarid (chronic rhinitis)
  • diagnose by fecal float for eggs

Both treatable with ivermectin and fenbendazole

22
Q

Discuss the different urinary system worms

A
  • Dioctophyma renale
  • ## Pearsonema plica
23
Q

Discuss Dioctophyma

A
  • the giant kidney worm of dogs
  • the **largest nematode ** in veterinary medicine
  • Lives in the kidney–> almost always in the right kidney
  • normal defintiive host is the mink
24
Q

Discuss the diagnosis, treatment, and transmission of Dictophyma

A

Transmission:
- eggs shed in urine
- Intermediate host: aquatic worms
- Paratenic hosts: usually freshwater fish

Transmission to definitive host usually via undercooked freshwater fish or water containing infected aquatic worms
Diagnosis: eggs found in urine sedimentation
Treatment: surgical removal

25
Q

Discuss the diagnosis of Dictophyma

A

Diagnosis: eggs can be seen in urine sediment

26
Q

Discuss Pearsonema Plica and the basics

A

Adults in bladder mucosa
- transmission: eggs released in urine
- Earthworms are the intermediate host
- Clinica;l sogms: usually asymptomatic
- Treatment: fenbendazole; ivermectin; levamisole
-

27
Q

NOTE THAT THE LAST FEW NEMATODES UNDER THE “MISCELLANEOUS NEMATODES - FYI CATEGORY” were NOT included on this PPT such as ocular ____ and spirocerca

A