Pancreatitis, TPN, NG and enteral tube therapy Flashcards

1
Q

Side of the pancreas that is responsible for hormones that regulate body system.

A

Endocrine

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2
Q

Side of the pancreas making digestive enzymes and that plays a role in pancreatitis.

A

Exocrine

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3
Q

What makes stool brown?

How can stool become clay coloured?

A

Bile

If a stone blocks the common bile duct, stool will become clay-coloured

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4
Q

How does pancreatitis occur?

A

An obstructoin blocks the common bile duct - cyst, gallstone, tumour, etc. - and blocks the pancreatic enzymes - start to autodigest the pancreas and get excruciating pain.

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5
Q

What are the main triggers of acute pancreatitis?

A

Alcohol abuse and gallstones

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6
Q

Acute pancreatitis

  • ________ of the pancreas
  • varies from mild ______ to severe _______ necrosis
  • occurs most often in ______-age; and this gender is more greatly affected
  • most common cause is _________ and ___ disease
A
inflammation
edema, hemorrhagic
middle-age
men > women
alcoholism, GB
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7
Q

How are stones removed from the common bile duct?

A

Surgical removal or blasted out

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8
Q

What is the mortality rate of acute pancreatitis?

What surgery and what test may predispose someone to develop pancreatitis?

A

10%

GI surgery or ERCP - endoscope retrograde cholangiography

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9
Q

What drugs would be stopped if someone developed acute pancreatitis?

A

Oral contraceptives, thiazide diuretics and corticosteroids

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10
Q

Self-digestion of the pancreas by its own proteolytic enzymes, principally _______, causes acute pancreatisis

A

Trypsin

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11
Q

80% of patients with acute pancretitis have biliary tract disease, however, only 5% of patients with _____ develop pancreatitis

A

gallstones

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12
Q

What are the classic symptoms of acute pancreatitis?

A

Pain that buckles you over

Nausea and vomitting - doesn’t improve after vomiting

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13
Q

Clinical manifestation of acute pancreatitis
Pain:
- located at the ______, radiates (often to the left ______ or ____), sudden onset, severe, deep, piercing, increases with ______, increases in _______ position, not relieved with ______.

A

LUQ, shoulder, back

eating, recumbent (on back with knees bent), vomiting

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14
Q

What are the classical clinical manifestations of acute pancreatitis?

A

Epigastric/back pain

Nausea and vomiting

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15
Q

In acute pancreatitis, we will see no motility in the ______.
If pancreatitis is prolonged, we will start seeing these signs.

A

ileus
grey turner sign
cullen’s sign

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16
Q

hemorrhagic patches on the skin around the umbilicus.

A

cullen’s sign

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17
Q

bruising of the skin of the loin in acute hemorrhagic pancreatitis,

A

Grey turner sign

18
Q

Acute pancreatitis is the unique occasion when _______ is most effective.

A

demerol

19
Q

How is pain relief accomplished for acute pancreatitis?

A

Morphine and demerol

20
Q

Interprofessional care for acute pancreatitis

  • pain relief - with what?
  • diet?
  • transfusion?
  • purpose of bed rest?
A

pain relief with morphine and demerol

  • client is NPO to minimize pancreatic enzyme secretion
  • client is fed through enteral feeding or TPN
  • NG tube is used to suction
  • albumin is tranfused if shock is present
  • will give calcium gluconate by IV if tetany is present

Client is on bed rest to decrease metabolic rate and enzymatic secretion

21
Q

cramps, convulsions, twitching of the muscles, and sharp flexion of the wrist and ankle joints. A manifestation of abnormal calcium metabolism.

A

tetany

22
Q

Usually we give patients an isotonic solution - normal saline; or dextrose if we want a hypotonic solution. However, there is a solution that can legit do both, what is it?

A

Lactated ringers

23
Q

Drug that decreases pancreatic activity/acidity in stomach/intestine; one of the most common medications given in acute care
What other drugs are given to pancreatitis patients that also decrease pancreatic activity?

A

Pantoprazole

Zantac and tagamet (oral)

24
Q

Diagnosis of Pancreatitis:

  • Hx and characteristics of ________ pain
  • Serum _______ and _______ levels (__x higher than normal)
  • Urinary _______ elevated; stools ____ and _____ odour
A

abdominal
amylase, lipase - 3x higher
amylase
pale, foul

25
Q

Why would stools be pale in pancreatitis?

Why the foul odour?

A

Pale if the obstruction occurs for long enough (no bile)

Foul odour if fat is not being broken down

26
Q

What is steatorrhea and what are the four F’s?

A

Medical term for fatty stool

- Foul smelling, fatty, floats, frothy

27
Q

What do we do to minimize the foul odour that the patient may be experiencing from pancreatitis?

A

Charcoal or odour antagonizers

28
Q

What would bloodword indicate for a pancreatitis patient?

A

Serum amylase and lipase 3x elevated
Elevated WBCs
tetany (hypocalcemia)
hyperglycemia
Elevated serum bilirubin
Dehydration leading to elevated Hematocrit
(in the case of hemorrhagic necrosis - can get decreased Hb)

29
Q

What is a paracentesis?

A

Abdominal tap - done at bedside, sterile procedure with large needles to remove fluid

30
Q

This is done to relieve a pleural effusion.

A

Thoracentesis

31
Q

What tests/procedures are done to aid in the diagnosis of pancreatitis?

A

Ultrasound, X-ray, Computed tomography

MRCP (magnetic resonance cholangiopancreatography), paracentesis

32
Q

Nursing interventions - pancreatitis
Relieving pain:
- Give pain meds ____ pain is severe
- Assess _______ of pain medication
- Comfortable _______, frequent change in position
- Side lying with HOB elevated ___ degrees

A

before
effectiveness
positioning
45

33
Q
Nursing interventions - pancreatitis
Relieving N&V:
- person will be \_\_\_\_\_
- NG tube for \_\_\_\_\_\_
- \_\_\_\_ fluids
- Frequent \_\_\_\_\_ and \_\_\_\_\_\_ care
A

NPO
suctioning
IV
oral and nasal care

34
Q

When meds are due at 8, when can we give them?

A

Anywhere from 7:30 to 8:30

35
Q
Nursing interventions - pancreatitis
Prevent infection:
- Monitor \_\_\_\_\_\_
- Preventing pneumonia?
- Immobility - risk for?
A

vitals
Turning, DB and C, Semi-fowler’s
Monitor skin frequently and provide good skin care

36
Q

Progressive destruction of pancreas with fibrotic replacement, strictures and calcifications of pancreatic tissue

A

Chronic pancreatitis

37
Q

Describe the typical patient who has chronic pancreatitis.

A

Not a surgical candidate - e.g. poor heart
Often abused alcohol
Malabsorption - often very skinny
Often diabetic (since the pancreas cannot produce insulin correctly)

38
Q

What is the most important symptom to remember for chronic pancreatitis?

A

They will have steatorrhea

39
Q

How is chronic pancreatitis managed?

A

Diet - low fat, high carb, no alcohol
Pancreatic enzyme replacement (viokase and Cotazym)
Diabetes control

40
Q

How is chronic pancreatitis diagnosed?

A
ERCP
CT
MRI
US
leukocytosis
increased ESR
Secretin stimulation test
41
Q

test for pancreatic function. Given Secretin hormone and then volume and
bicarbonate concentrations are measured of pancreatic secretions.

A

Secretin stimulation test

42
Q

A reduction in these concentration is usually diagnostic of chronic pancreatitis (secretin stimulation test).

A

Amylase and bicarbonate