Paediatric Orthopaedics Flashcards

1
Q

What is ‘flat foot’?

A

When the arch of the foot doesn’t fully develop, and this may appear as weak ankles which turn inwards

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2
Q

What is the treatment for ‘flat foot’?

A

Orthotics to aid the arch with stretching or surgery

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3
Q

What are the potential causes of flat feet?

A

Muscle imbalances or issues with the tarsal bones

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4
Q

What may persistent toe walking in older children indicate?

A

Cerebral palsy, DMD or nervous system disorders

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5
Q

How may toe walking be corrected?

A

A cast for the foot and ankle or stretching of the calf muscles

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6
Q

What is club foot?

A

Where the foot looks like a foot; also known as talipes equinovarus which means there is a fixed varus and equinus deformity and calf underdevelopment

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7
Q

What is the epidemiology of club foot?

A

1 in 1,000 children have it and it’s more common in boys

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8
Q

What are the risk factors for developing club foot?

A

Breech presentation, connective tissue disorders, too little amniotic fluid, Edwards Syndrom

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9
Q

How is club foot treated?

A

Ponseti method - manipulative technique to correct the foot using braces and stretching

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10
Q

What is the Ponseti method?

A

Method of manipulative treatment for club foot

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11
Q

What is Edward’s syndrome?

A

Trisomy 18

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12
Q

What is hip dysplasia?

A

When the hip joint is mal-aligned

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13
Q

How common is congenital hip dysplasia?

A

1.5 in 1,000 children

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14
Q

What are the risk factors for congenital hip dysplasia

A

Breech delivery and family history of club foot or scoliosis, and is commoner in females

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15
Q

How may congenital hip dysplasia present in infants?

A

Tends to be double creases when the leg is turned outwards, with asymmetric gluteal folds (double crease on affected side)

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16
Q

Why is the use of x-ray limited in the diagnosis of congenital hip dysplasia in infants?

A

As the femoral head doesn’t ossify until 4-6 months and therefore won’t be visible on x-ray

17
Q

How may an ultrasound be used to diagnose congenital hip dysplasia?

A

The femoral head will move back on the gluteal muscles, causing the fibres to look more vertical

18
Q

How is ‘Barlows test’ used to diagnose congenital hip dysplasia?

A

Knees are brought together and pushed on slightly whilst the infant is lying down. If the hip can be dislocated like this, this is a positive test

19
Q

How is ‘Ortolani test’ used to diagnose congenital hip dysplasia?

A

This usually follows on from Barlows test, and involves abducting the hip to see whether the hip will pop back in, to confirm the diagnosis

20
Q

How is ‘Galeazzi test’ used to diagnose congenital hip dysplasia?

A

Flex the hip and knee and see if there is a leg length discrepancy in the height of the knee, affected side will be shorter

21
Q

How is congenital hip dysplasia treated?

A

Under 6 months = Pavlik harness to keep the femoral head in the true acetabulum
Over 6 months or doesn’t work = closed reduction with hip spikes or open reduction

Often resolves spontaneously though, however.

22
Q

What is Perthes’ disease?

A

Self-limiting avascular necrosis of the femoral head often due to loss of the acetabular branch of the obturator artery which supplies the femoral head

23
Q

Is Perthes’ disease usually unilateral or bilateral?

A

Unilateral

24
Q

What are the four stages of Perthes’ disease?

A

Necrosis, fragmentation, reossification and remodelling

25
Q

Describe the necrosis stage of Perthes’ disease

A

A portion of the femoral head dies and the shape of the femoral head changes, causing pain, stiffness and inflammation and this takes up to one year

26
Q

Describe the fragmentation stage of Perthes’ disease

A

The dead cells from the necrosis of the femoral head are then absorbed and replaced by new bone, and as the new bone forms it produces varying femoral head shapes, this takes 1-3 years to take place

27
Q

Describe the reossification stage of Perthes’ disease

A

The femoral head continues to growth with new bone cells, taking 1-3 years

28
Q

Describe the remodelling stage of Perthes’ disease

A

New bone cells are gradually replaced by normal bone cells and remodelling continues, and this can take 1-3 years or more

29
Q

What is slipped upper femoral epiphysis (SUFE)?

A

Where the epiphysis (growth plate) of the femur stays in place, but the femoral neck and shaft displace

30
Q

Which side is more likely to be affected by SUFE?

A

Left hip

31
Q

What are risk factors for SUFE?

A

Obesity, hypothyroidism, deficiency or increased androgen production and trauma

32
Q

How may SUFE appear on x-ray

A

Hip joint may appear as ice-cream falling off it’s cone

33
Q

How may Klein’s line be useful in diagnosing SUFE on x-ray?

A

This is a line drawn along the superior border of the femoral neck, and should cross at least a portion of the femoral head, if the femoral head drops below this line, this is indicative of SUFE

34
Q

How may SUFE be treated?

A

Rest, analgesia, surgery (to close growth plate with screws or bone removal)

35
Q

What is Blount’s disease?

A

where there is a growth problem of the distal tibia (in the medial compartment) which causes tibia varus)

36
Q

What is thought to be the cause of Blount’s disease?

A

Effect of weight (obesity) on the growth plate

37
Q

What is Osgood Schlatters disease?

A

Quadriceps insert onto the tibial tuberosity, which causes the tuberosity to be pulled due to exercise. This causes the bone to grow anteriorly at the insertion point.

38
Q

What type of condition are Severs’ and Schlatters disease?

A

Osteochondroses

39
Q

What may cause Osgood Schlatters disease?

A

Repeated traction from patellar ligament on tibial tuberosity (due to heavy exercise) or avulsion fractures to part of the tibial tuberosity