Musculoskeletal Flashcards

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1
Q

what are contusions

A

occur when external force such as a fall or blow breaks capillaries without breaking the skin

bruising and swelling

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2
Q

contusions manifestation

A

bruising
pain
swelling

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3
Q

contusions care

A

first 24-48 hours, apply ice for 15 min, 3x a day

wrap the area to compress
color change to greenish-yellow after 3-5 days

should completely heal within 7-10 days

observe for a bruise that keeps spreading

observe any changes in mental status if bruise is realted to the head

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4
Q

what are strains

A

cause by overstretching, overexertion or miscuse of muscles

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5
Q

first degree strains manifestations

A

mild and gradual onset
feels stiff and sore locally

muscle will be tender to the touch
may experience muscle spams

no loss of range of motion
little to no edema or ecchymosis will be seen

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6
Q

second degree strain manifestation

A

sudden onset, with acute pain that eventually leaves the area tender

extreme muscle spams and passive motion will increase pain

edema will develop early and later, ecchymosis will appear

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7
Q

third degree strain anifestation

A

occurs when there is severe stretching of muscle with tear

feels sudden, snapping or burning sensation

muscle spams
joint tenderness
edema - extreme

cannot move the strained muscle voluntarily

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8
Q

1st degree strain care

A

ice
rest
possibly immobilize for short term
elevate it

oral, non-opioid analgesics or NSAIDs

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9
Q

2nd degree strain care

A

elevate limb
ice for first 24-48 hours
apply moist heat

limit mobility, using compression bandage

use muscle relaxants, analgesics and NSAIDs

implement physical therapy

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10
Q

3rd degree strain care

A

elevate
ice for first 24-48 hours

immboilize or limit mobility

limit weight bearing on lower limbs

use muscle relaxants, analgesics and NSAIDs
physical therapy

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11
Q

what are sprains

A

involves injury to ligament structure by stretching, overexertion or traums

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12
Q

1st degree sprain manifestations

A

localized edema or hematoma
some mild discomfort

increased pain when limb is palpated or bears weight

no loss of functioning or weakening the joint structure

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13
Q

2nd degree sprain manifestations

A

edema
possible hematoma

decreased active ROM
mild weakening of the joint and pain

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14
Q

3rd degree sprain manifestation

A

severe edema with hematoma
severe pain

dramatice decrease in their active ROM
loss of joint integrity and function

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15
Q

1st degree sprain care

A

wrap
keep limb raise
ice for 24-48 hours
analgesics

isometric exercise
- to increase circulation and resolve hematoma

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16
Q

2nd degree sprain care

A

dress splint and immboilize it

elevate limb
24-48 hours, alternate:
- ice for vasoconstriction
- moist heat to decrease swelling and provide comfort

analgesics
physical therapy

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17
Q

3rd degree sprain care

A

casting/immobilizations

surgery may be needed to restore the integrity of joint

same as 2nd degree:
ice then heat

compression banadage:
if tingling below area heppans, bandage was wrapped too tihgtly, remove then reapply

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18
Q

types of fractures

A

complete: when bone is broken into two or more pieces
incomplete: bone is broken but still in one piece
close: simple fracture that does not break the skin
open: breaks the skin

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19
Q

fractures manifestations

A

swelling
pallor and ecchymosis
loss of sensation to body parts
deformity

pain and/or tenerness
muscle spams

loss of function, abnormal mobility

crepitus
shortening of the affected limb

decreased or absent pulses
affected extrremity colder than the other

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20
Q

fractures care

A

closed reductions: procedure to set broken bone without surgery
- allows bone to grow back together
0 wroks best hen done as soon as the bone breaks
- x-rays are done to see if it was successful

immobilization: keeps the bone fragments from moving and relieves pain
- casting
- traction
- splints
- braces or external fixation

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21
Q

stages bone healing

A
  1. hematoma formation
  2. fibrocartilage/ granulation tissue formation
  3. callus formation
  4. ossification
  5. consolidationg/remodeling
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22
Q

bone healing complications

A
immediate:
shock
fat embolism
- can occur 24 hours after the injury and more common in pelvic/femur fractures
DVT
PE

delayed:
joint stiffness and post-traumatic arthritis
reflex sympathy dystrophy
myostitis ossificans
malunion
- fractured bone heals in an abnormal position

delayed union
- bone is slow to heal

nonunion
- when gracture does not heal

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23
Q

bone healing management

A

check neurovascular status
palpate for pulses, sensation, skin temp, ability to move appendages

elevate limb above level of heart, unless compartment syndrome is suspected

apply cold

immboilize the client as soon as possible
turn every 2 hours
- use pressure air mattress
- position with proper alignment

incentive spirometry frequently
proper coughing and deep breathing

monitor for signs of infection
proper wound care and antibiotics

VTE prophylaxis is key for clients who are immobilized after a frature

  • hydration
  • comopression stockings
  • pneumatic devices

fat embolism cannot be prevented with these interventions

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24
Q

skeletal traction care

A

perform daily pin care
inspect traction apparatus every 8 hours
ensure weights are free hanging

teach how to bear weight and how much weight is permitted

teach how to use assistive device

teach cast care:

  • keep dry
  • dont put anything inside cast
  • itch with hair dryer on cool setting
  • report swelling and anything abnormal

use incentive spirometyr

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25
Q

5 P’s

A
pain
paresthesia
pallor
paralysis
pulse
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26
Q

factors that enhance healing

A

fracture near a good blood supply
minimal damage to soft tissue

anatomic rduction
fragments are in a good position to heal
immobilization

weight can be borne on lone bones

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27
Q

factors that delay healing

A

poor blood supply
severe damage to soft tissue

separation of fragments

improper fixation allows bones to move or rotate

pre-exisiting factors:

  • obesity
  • diabetes
  • steroid use
  • smoking

severely comminuted fractures
bone loss
infection

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28
Q

what is rhabdomyolysis

A

acute condition involving the breakdown of skeletal muscle tissue

occurs when trauma to the muscle compresses tissue, it causes ischemia and necrosis

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29
Q

rhabdomylosis manifestation

A

myalgia - muscle pain
weakness
myoglobinuria - tea colored urine

triad of symptoms

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30
Q

rhabdomylosis diagnostic studies

A

elevated CK level = most sensitive

serum creatinine - elevated
- can indicate kidney injury

serum electrolytes - hyperkalemia

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31
Q

rhabdomylosis care

A

early recognition and management of ABCs

intervention will focus on:
- preserving renal function
- preventing acute kidney injury
by administering large volumes of IV fluid

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32
Q

what is osteoarthritis

A

degenerationg of the aritcular cartilage and formation of bones

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33
Q

types of osteoarthritis

A

primary OA:

  • idiopathic and is most often related to aging process
  • symptoms appear in middle age and progresses with age

secondary OA:
resulting from predisposing condition or trauma
- obesity, family hx of degenerative joint disease
- joint abnormality
- excessive wear
- repetitive stress common in certain occupations
- mostly in caucasian women

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34
Q

osteoarthritis manifestations

A

joint stiffness after period of rest
pain in a movable joint, typically worse with action and relieved by rest

paresthesia
joint enlargement
- Heberden’s nodes: joint closest to fingernail gets enlarged
- Bouchard’s nodes: middle joint of the finger and closest to body gets enlarged

joint deformities
tenderness on palpation

joint surfaces no longer fit together
muscle spasm and contract

joints are blocked by osteophytes and loose bodies
creptitation and crunching ocurrs when joints are moved

eventual ankylosis or stiffening of joint

changes to gait occur
shortneed stance
abnormal antalgic gait
- limp that help avoid pain

widened base of support
shortened step length

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35
Q

osteoarthritis diagnostic studies

A
x-rays
bone scans
MRI and/or CT scan
history 
physical exam
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36
Q

osteoarthritis management

A

functional and mobility assessment
pain management
correct use of assistive devices

implementation of prescribed heat and/or cold therapies
proper posture and body mechnics
weight reduction if obese

collaboration with physical and occupational therapy
referral to support agencies
home safety

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37
Q

osteoarthritis pharmacological interventions

A

aspirin
- can cause tinnitus, gastritis, peptic ulcer disease

NSAIDs

  • ibuprofen, naproxen
  • celexicob, indomethacin
  • meloxican
  • may cause GI bleeding

corticosteroid injections
hyaluronic acid injections
opioids analgesics

38
Q

osteoarthritis nonpharmacologic itnerventions

A

assistive devices
TENS has been shown effective for pain relief
physical exercise
relaxationtechniques
appropriate nutrition and weight loss
active stretching and non-weight bearing activities like swimming

acupuncture and massage therapy may help lessen the pain

39
Q

what is carpal tunnel syndrome

A

compression of the median nerve that neters the hand through a tunnel of ligaments and bone at wrist

repetitive strain injury

dibaetes, peripheral vascular disease and rheumatoid arthritis have higher incidence of CTS

40
Q

carpal tunnel syndrome manifestation

A
weakness
pain, tingling
numbness or decreased sensation in the affected hand
difficulties with fine motor movement
atrophy of thumb muscles
41
Q

carpal tunnel syndrome diagnostic studies

A

history
physical exam
identification of risk/precipitating factors

corresponding clinical symptomaology
postitive Tinels sign
- compress with 2 fingers at wrist and see if there is tingling

positive phalen’s sign
- put both wrists at a 90 degree downward and see if there is tingling

42
Q

carpal tunnel syndrome nursing management

A

relieving underlying cause of nerve compression
wrist splint

ergonometry with workstation modification
physical therapy

hand/wrist exercise
NSAIDs
rest, ice

if conservative treatment does not work, then more invasive treatment may includes:

  • corticosteroid injection
  • surgery
43
Q

what is plantar fascitis

A

inflammation of the plantar fascia, located on the arch of the foot

often seen in middle age and older adults, esp runners

obesity is a contributing factor

44
Q

plantar fascitis manifestations

A

severe pain in arch of foot, esp when getting out of bed
pain worsens with weight bearing

most have unilateral plantar fascitis, it can affect both feet

45
Q

plantar fascities diagnostic studies

A

history and physical exam
identification of risk factors
corresponding clinical symptomalogy

46
Q

plantar fascitis nursing management

A

rest, ice
physical therapy

stretching exercises
shoes with good support
orthotic shoe inserts

NSAIDs
steroid for pain and inflammation

if these are unsuccessful then endoscopic surgery is needed to remove the inflamed tissue

if taking corticosteroid, important to NOT abruptly stop taking them
- take with food to prevent GI upset

if diabetic, monitor for higher than usual blood sugar levels

47
Q

what is rheumatoid arthritis

A

chronic, systemic inflammatory disease of the connective tissue

does have genetic tendency
- more common in women than men

irreversible and will lead to ankyloses of the joint
- abnormal stiffening and immobility of a joint

48
Q

rheumatoid arthritis manifestations

A
general signs:
fatigue
loss of appetite
weight loss
enlarged lymph nodes

early RA:
painful, stiff joints
warm, red, swollen and incapable of full ROM

late RA:
bony ankyloses
destruction of joint
adhesions
inflammation and effusions
Raynauds syndrome

may present with ulnar deviation
- deformed hands and fingers, including swan neck of fingers and joints

49
Q

rheumatoid arthritis diagnostic studies

A

history and physical exam
radiographic studies
aspiration of synovial fluid

labs:
decreased ESR
increased RBC
positive c-reactive protein
positive antinuclear antibody
positive rheumatic fever
50
Q

rheumatoid arthritis care

A

surgical intervention
splinting
adequate rest

ice for joint inflammation
heat for joint stiffness

nutrition therapy:
weight reduction if obese
calcium supplements

51
Q

rheumatoid arthritis complementary health

A
gamma-linolenic acid
fish oil
ginger, boswelia
green tea
turmeric
- the three have ongoing research
52
Q

rheumatoid arthritis management

A

psychosocial support
education related to corticosteroids
promotion of self care and independence

pain control is priority assessment

administration of meds
balancing exercise and rest, energy conservation
assistive devices

speak HCP before using herbal or OTC medications

53
Q

what is systemic lupus erythematosus

A

chronic, systemic inflammatory disease of the collagen tissues

african americans, hispanics, asians and antive americans are 2-3 time more likely to have lupus
- most cases are found in women

may experience kidney damage that is progressive and can lead to end-stage renal dsease
- requiring dialysis or kidney transplantation

54
Q

systemic lupus erythematosus manifestation

A

SLE more likely to present if client has 4 or more findings:

  • arthritis
  • butterfly rash: malar rassh
  • discoid lupus skin lesions
  • photosensitivity
  • oral ulcers
  • serositis
  • renal disorder: persistent proteinuria
  • seizures or psychosis in absence of drugs
  • hemolytic anemia
  • positive lupus erythematosus cell preparation
  • abrnomal titer of antinuclear antibody
55
Q

systemic lupus erythematosus care

A

control system involvement
monitor findigns
promote remission

pharmacological interventions:
salicylates
NSAIDs
corticosteroids
anti-infectives
anti-cancer drugs
antimalarial drugs
56
Q

systemic lupus erythematosus management

A

maintaining skin integrity
weight management
energy conservation techniques

sun protection
well balanced diet - no specific diet needed or avoided

57
Q

what is gout

A

metabolic disease marked by painful inflammation of joints

deposits of urates in and around the joints

usually an excessive amount of uric acid in the blood

peak incidence between 40-60 years old
does have familial tendency

abdnormal purine metabolism or excessive purine intake results

deposits most often found in the metatarsophalangeal joint of the greater toe or in ankle

58
Q

gout manifestations

A

tight, reddened skin over inflamed joint
elevated temperature

edema of involved area
severe pain in affected joint

hyperuricemia because uric acid levels are in bloodstream

acute attacks begin at night and last 3-5 days

may follow trauma, diuretics
increased alcohol consumption
a high purine diet
stress
suddently stopping maintenance meds

g

59
Q

gout diagnostic studies

A

synovial fluid analysis will revel uric acid crystals

blood work will show hyperuricemia

60
Q

gout care

A

pharamcological interventions
NSAIDs
- indomethacin

colchine

  • used when NSAIDs are contraindicated
  • enhances the excretion of uric acid

antihyperuricemic agents
- allopurinol
heat or cold therapy
dietary changes to low purine diet

61
Q

gout management

A

pain management
elevate affected limb
bedrest and immobilize joint

avoid putting pressure or bedding on affected joint

reinforce need for dietary management
low purine diet:
- protein is okay
- avoid organ meats
- shellfish (shrimp)
- sardines

incrase fluid intake to prevent renal calculi
use aspirin and diuretics cautiously and avoid if possible

HALLMARK of gout are: flareups or acute episodes of sevre pain, lasting days to weeks
- during an acute attack –> goal is to treat with colchine and manage pain

62
Q

what is osteomalacia

A

occurs when mineralization is delayed, resulting in a soft weak bonew

rickets caused by vitamin D deficiency
- not having enouhg calcium and phosphorus

63
Q

osteomalacia manifestation

A

generalized muscle pain and skeletal pain in hips and lower back
waddling gait

wide stance and may not want to walk at all

deformities in weight bearing bones
scoliotic or kyphotic deformities of the spine

bones tend to break easily

64
Q

osteomalacia diagnostic studies

A

radiiographic findings

lab tests:

  • decreased calcium and phosphorus
  • alkaline pshophatase will be moderately elevated
65
Q

osteomalacia care

A

pharmacological interventions;

  • calcium gluconate
  • vit D daily, until signs of healing take place
  • high protein diet
  • UV radiation therapy
66
Q

osteomalacia management

A

teach about intake of dietary vit D

importance of safe sun exposure to gain vit D

67
Q

what is osteporosis

A

reduction in bone mass, loss of bone strength and an increased risk of fracture

68
Q

contributing factors to osteoporosis

A
aging
heredity
nutrition
lifestyle
medications
69
Q

risk factors in osteoporosis

A
low bone density
history of scoliosis
female - northern european and asian
neruological impairement
- stroke
parkinsons
decreasd vision
complications of diabetes
70
Q

osteoporosis manifestations

A

acute fractures
hisotyr of falls

pain greater when active and typically occurs in mid-low thoracic spine

kyphosis: dowagers humps
- may lose 2 or more inches in height

71
Q

osteoporosis manifestations

A

CBC of calcium, phosphate, alkaline phosphatase
x-rays
DEXA scan
- assess cortical and trabecular bones in the spine and hip
T-score of -2.5 ore lower indicates osteoporosis

72
Q

osteoporosis care

A

weight bearing exercises
nutritional changes

pharmacological interventions:
antiresportive agents
- do not increase bone mass but prevetnt further bone loss

estrogen therapy
calcitonin
peptide hormone
biphosphonates
- inhibit bone resoprtion

androgen
-taken long term

surical intervention

  • Vertebroplasty
  • kyphoplasty

clients taking oral biphosphonates should be instructed to:

  • take them with a full glass of water
  • take them at least 30 minutes before food and other meds
  • stay upright for at lesat 30 min after taking them
73
Q

what is pagets disease

A

slow progressing resporption and irregular remodeling of bone

cause is unknown
- viral implication and family tendencies

bone is resolved and new bone develops poorly

bone is weak and fractures easily
- affects the skull
- femur
0 tibia
- pelvis
- vertebrae
74
Q

pagets disease manifestation

A

initially asymptomatic

as disease progresses:
- pain and point tenderness of affected limbs develops

pathologic fractures - femur or tibia

deformity of long bones

75
Q

pagets disease diagnostic studies

A

radiographic studies

  • show a bowing of long bones
  • thickened areas of bones

labs

  • increase in alkaline phosphataste
  • increase in urinary hydroxyproline
  • calcium will be normal
76
Q

pagets disease care

A

pharmacological interventions:
NSAIDs
biphosphonates - alendronate
- slows bone resportion

calcitonin - slows bone resportion
plicamycin
- used when nerve ares damaged and the client is unresponsive to other treatments

surgicla interventions

77
Q

pagets disease management

A

administer meds
pain management
increase mobility

physical therapies cllaborate
collaborate with nutrition therapy

firm mattress for sleep
some may need to wear a corset or back brace

78
Q

what is oteomyelitis

A

bacterial infection of the bone

staph aureus is the most common

79
Q

osteomyelitis manifestation

A

pain, loclized, tenderness
erythema over the involved bone
decreased ROM at affected bone

irritability
restlessness
fever

80
Q

osteomyelitis diagnostic studies

A
lab tests:
- increased ESR
blood cultures and bone aspirate cultures
CBC
increased WBC

radiographic tests
- often negative for 10-14 days
bone scan

81
Q

osteomyelitis care

A

IV atibiotics will be administered
long term IV access
-4-6 weeks

surgery and/or immobilization

82
Q

osteomyelitis management

A

monitor antibiotic levels
- peak and trough
monitor level of comfort

encourage to perform ROM exercises if possible
- but DO NOT allow weight bearing exercises

monitor clients nutritional intake and status

hpysical therapy at home

IV access for antibiotic therapy

83
Q

what is hip arthroplasty

A

replacing the entire hip joint, either total hip of the femoral head and acetabulum

or partial hip replacement - femoral head only

84
Q

indications for surgery

A
OA
RA
femoral neck fractures
avascular necrosis of femoral head caused by steroids
failure of previous prosthesis
85
Q

hip arthroplasy post op care

A

immediate post op care:

  • benefite from PCA using peripheral or epidural catheter for first 48 hours after
  • PCA will aid in early mobilization

intermediate to long term post op care:

  • monitor neurovascular status of leg
  • pulmonary hygiene
  • prevent abduction of hip
  • used in the first postoperative week while the clients sits in a chair or bed
  • turn client by logrolling
  • use fraacture bedpan
  • monitor for hip dislocation
  • low molecular weight heparin
  • collaborate with physical therapist for early ambulation and exercise
86
Q

hip arthroplasty client and caregiver education

A

correctly use assistive device
implement strategies to prevent dislocation
- raised toilet seat

perform prescribed exercises at home
avoid sitting for more than one hour at a time

wear antiembolism stocking
adhere to anticoagulant therapy

fall prevention measures

87
Q

knee arthroplasty post op care

A

apply ice to knees
physical therapy will be started within 24 hours to promote bone function

continuous passive motion (CPM) in use
when not in use, a knee immobilizer might be used

88
Q

amputation indications

A
progressive periphera vascular disease
gangrene
trauma
congenital deformities
malignant tumors
89
Q

amputation management

A

phantom limb pain can be relieved with:

  • stump desensitization through kneading or massaging of the stump
  • TENs
  • distraction
  • beta adrenergic blocking agents for burning, dull pain
  • anticonvulsants for sharp and cramping pain

phantom limb pain may occur any time up to 3 months or longer post-amputation
- most common with above the knee amputation

use aseptic dressing change
compression wrapped in a figure eight fashion
casting to control edema

early rehabilitation
prostatic preparation is key to restoring mobility

below the knee amputation;
- client should lie SUPINE with the affected leg fully extended for 20-30 minutes, 3-4 times/day

above the knee amputation:
- client should lie PRONE with the affected leg fully extended for 20-30 min, 3-4 times/day

convey body image acceptance

    • not ALL clients are candidates for a prosthesis after amputation, it required a lot of upper body strength.
  • for those who struggle in these areas, should use a wheelchair
90
Q

rheumatoid arthritis pharmacological interventions

A

pharmacological interventions:

  • NSAIDs
  • hydroxychloroquine sulfate

immunosuppressant agents like:

  • azathioprine
  • cyclophosphamide
  • methotrexate
  • prednisone
  • sulfasalazine
  • lefluromide
  • anakinra

biological response modifiers:

  • etanercept
  • infliximab
  • adalimumba
91
Q

sulfasalazine side effects

A

crystalluria
- drink 8 glasses of water to produce 1200- 1500mL/day

photosensitivity
risk of sunburn

folic acid deficiency
- so take folic acid 1mg/day

agranulocytosis (leukopenia)
- report sore throat immediately

stevens johnson syndrome
- stop if getting rash

urine and skin will turn orange but normal
- will go away once discontinued