Integumentary Flashcards

You may prefer our related Brainscape-certified flashcards:
1
Q

what are wounds

A

break or opening in the skin

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2
Q

factor sthat delay wound healing

A

think “ DID NOT HEAL”

drugs
infection
ischemia
diabetes
nutrition
oxygen 
toxins
hyperthermia/hypothermia
ETOH - ethanol
acidosis 
local anesthetics

and then meds and age
- immunosuppressants and corticosteroids

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3
Q

primary intetnion

A

method of wound healing

used when wound edges can be closed by stitches, staples skin glue or steri strips

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4
Q

secondary intention

A

method of wound healing

cannot be brought together by wound edges
- example: pressure ulcers

wound healing occurs through slow, gradula filling in lost tissue

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5
Q

tertiary intetnion

A

when a delay in closing the wound is NEEDED to allow for drainage and swelling to subside

surgical debridement, skin grafts and flaps are additional interventions used for certain wounds

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6
Q

wound management

A

successful wound management includes:
- removing or eliminating causative factors, preventing infection, using the appropriate type of dressing and ensuring systemic support

maintain adequate hydration and nutrition

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7
Q

what is atopic dermatitis

A

chronic, inhertied skin disorder

eczema

exacerbated by stress and certain foods

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8
Q

atopic dermatitis manifestation

A

acute:

  • dry skin
  • itching
  • erythema
  • macules
  • papules
  • pustules and/or vesicles

subacute and chronic stages:

  • scaly, dry skin
  • skin color changes
  • lichentification: thickened or leather like areas
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9
Q

atopic dermatitis diagnostic studies

A

med history
physical exam
skin biopsy
allergy skin testing

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10
Q

atopic dermatitis nursing management

A

lubriacation of dry skin
administration of corticosteroids, topical immunomodulators

stress reduction
phototherapy when inflammation is severe

antibiotics for secondary infections

soaking the hands in lukewarm water will debrider the crust and scales and soft the skin

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11
Q

what is seborrheic dermatitis

A

recurrent inflammatory reaction of the skin that causes flaky, white to yellowish scales to form on oily areas like:

  • scalp
  • external ear canal

aka: dandruff or “cradle cap” in infants

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12
Q

seborrheic dermatitis manifestation

A

observe non-pruritic, oily scales on scalap, forehead and eyebrows or behind ears

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13
Q

seborrheic dermatitis nursing management

A

cradle cap in infants:

  • massage baby’s scalp gently with your gfingers or soft brush
  • shampoo daily with mld baby shampoo
  • use hydrating creams or mineral oils

dandruff:

  • recommended OTC or medicated shampoos
  • prescription or lotions are available to severe cases
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14
Q

what is incontinence associated dermatitis

A

aka diaper rash

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15
Q

incotinence associated dermatitis nursing management

A

maintain perineal skin integrity

minimize episodes of incontinence
treat any underlying cause of loose stools

apply barrier cream.ointment

treat any underlying skin infections

prompt changing of wet, soiled clothing or incontinence products

using a fecal management system if appropriate
ensuring adequate hydration and nutrition

supporting diligent perineal hygiene with pH-based cleansers

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16
Q

what is contact dermatitis

A

inflammatory reaction of the skin to a chemical substance, whether natural or synthetic

ex: posison ivy

foods, solutions, allergens, plants are factors

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17
Q

contact dermatitis manifestations

A

irritant:

  • dry, inflamed
  • pruritic lesions wherever the irritant touched

allergic:
- “weeping blisters” as well as pruritic lesions

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18
Q

contact dermatitis management

A

topical application like anti-inflammatory and anti-pruritics
- topical corticosteroids are preferred for mild, localized cases of allergic dermatitis

try not to too frequently hand wash because itll become dry

systemic corticosteroids and antihistamines might be required if dermatitis is severe and widespread

exposure to heat or cold may cause or exacerbate symptoms

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19
Q

what is candidiasis

A

bacteria that causes candidiasis normally live in the body without any problems

its if they grow out of control that can lead to candida albicans

develops in the mouth or throat and is called thrush
in vagina = yeast infection

tends to appear in warm, moist areas and inhales steroids
- commonly seen in cancer and HIV/AIDs people

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20
Q

candidiasis manifestations

A

white plagues on the tongue, gums or buccal mucosa
- inner lining of the cheeks and back of lips where they touch teeth

ulcerations in oral mucosa

may also complain of itching

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21
Q

candidiasis management

A

oral anti-fungals will be prescribed
- either use swish and swallow after feeding or through topical application

those who use inhaled steroids should rinse their mouth after to avoid candidiasis

be sure to monitor whether infection has spread to groin or buttocks

can be prescribe prophylactic probiotic
- Florastor

eating products with live cultures of lactobacillus acedophillus like some yogurts can be beneficial

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22
Q

what is acne vulgaris

A

skin condition in which pores becomes clogged and inflamed

acne breakouts may be exacerbated by emotional stress

sebaceous glands secrete more sebum and pores become more plugged and dilated

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23
Q

acne vulgaris manifestation

A
whiteheards (close comedones)
blackheads (open comedone)
papules
pustules
nodules

red and excoriated skin

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24
Q

acne vulgaris management

A

good cleansing of the skin
nutrition
avoiding squeezing or picking at lesions
teach about resulting body image users

pharmacological intervention:

  • Vit A
  • benzoyl peroxide
  • antibiotics
  • isotretinoin: generic only- available as Accutane
  • photodynamic therapy
  • dermabrasion or chemical skin peels
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25
Q

what is impetigo

A

highly contagious, bacterial infections of the skin

infective agents:

  • Staph A
  • group A beta hemolytic streptococcus

MRSA is becoming a common cause

will invade broken skin are and is highly contagious for 7-10 days
most common in young children

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26
Q

impetigo manifestations

A

primary phase:
pustules or vesicles

secondary phase:
honey-colored crusts for strep
clear for staph
superficial erosion 
pustules that easily bleed and itche

skin does tend to heal without scarring unless there is a secondary infection

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27
Q

impetigo diagnostic studies

A

based on the appearance of skin lesion

maybe skin culture

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28
Q

impetigo nursing management

A

priority interventions should focus on preventing transmissions:

  • careful removal of lesions crusts or debris with warm soapy solution
  • cutting childrens fingernails

pharmacological interventions:

  • apply topical antibiotics for early small lesions
  • systemic antibiotics are treatment of choice

teach caregiver that infection is HIGHLY CONTAGIOUS

good handwashing is essential
- sharing towels and eating utensils should be AVOIDED

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29
Q

what is cellulitis

A

infection of the dermis and or subcutaneous tissue

causative agents:

  • streptococcal bacteria
  • Group A strep
  • streptococcus pyogenes
  • staph aureus
  • Haemophilus influenza
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30
Q

cellulitis manifestation

A

affected area:

  • red, edematous
  • tender
  • occasional discoloration

enlarged lymph nodes
fever
malaise
headache

streaking is frequently seen

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31
Q

cellulitis diagnostic studies

A

CBC
blood culture
possible skin culture

32
Q

cellulitis nursing management

A

administer prescribed antibiotics
apply warm compress
inciision and drainage may be needed
monitor size of area by marking and dating reddened area

monitor for secondary infection and any lateration in skin integrity

if cellulitis is not diagnosed and treated correctly, it can progress to gangrene

33
Q

what is tinea

A

ringworm

can be transmitted person to person, through animal contact or through contact with contaminated feces

can be associated with poor hygiene and friction from tight clothing

34
Q

type of tinea

A

capitis:
- hair, scalp

corporis:
- body

cruris:
- perineum

pedis “athletes foot”:
- feet or ankles

35
Q

capitis manifestation

A
scaly, circular patches
"blood dot"
alopecia
red area
pruritus
fever
36
Q

capitis treatment

A

antifungals, shampoos

37
Q

corporis sxs

A

erythematosus
scaling patches
round or oval shaped

38
Q

corporis treatment

A

local therapy with antifungal creams or powders

39
Q

cruris sxs

A

similar to corporis
pink papules
pruritus in genital fold

40
Q

cruris treatment

A

local therapy with antifungal creams or powders

aka jock itch

41
Q

pedis sxs

A

lesions
pruritus
maceration between toes
burning sensation

42
Q

pedis treatment

A

local thearpy with antifungal spray or lotion

possibly Burow’s solution

43
Q

tinea management

A

antifungals meds need to be taken for several week or months

encourage good hygiene products

do not share clothing, hair brushes, linens and towels

avoid overuse of OTC products, esp for their feet

44
Q

what is pediculosis capitis

A

head lice

live only humans and are transmitted by direct and indirect contact

other types of lice:

  • body lice
  • pubic area
  • eyebrows and eyelashes
45
Q

pediculosis capitis manifesation

A

nits - tiny silvery or grayish white specks
pruritius
rose-colored dermatitis

46
Q

pediculosis capitis management

A

topical medicated shampoo or body ointments
- an OTC cream rinse petroleum for infants and children or lindane shampoo

contact precautions

prevent transmission and spread by washing clothes in hot water

o not share clothes or hair brushes

47
Q

what are scabies

A

contagious skin infection of mites

causative agent = mites
- female mite s burrow into the epidermis and lay eggs

48
Q

scabies manifestation

A

pruritus
burrows - fine grayish brown threadlike lines

intense itching especially at night
papule-like eczema in infants

49
Q

scabies diagnostic studies

A

history
clinical symptoms
microscopic visualization of skin scrapings

50
Q

scabies management

A

pharmacological interventions:

  • topical scabicide medications or creams
  • treatment of choice = permethrin
  • anti-steroidal creams for itching
  • systemic anti-infectives used for secondary infections

wash cloths and bedding in hot water
- all persons in close contact with affected person will need treatment

51
Q

what is frostbite

A

occurs when ice crystals form in the tissue causing tissue damage

52
Q

frostbite manifestations

A

first degree:
blanching
decreased sensation
mottling

second degree:
blisters with possible bulla
- large blisters that are filled with clear fluid

third degree:
cyanosis
mottling then red with swelling
local necrosis
hemorrhagc vessels

fourth degree
complete necrosis
gangrene
loss of affected body part

53
Q

frostbite nursing management

A

cover the area immediately but do not apply massage

rewarm the affect part gradually by immersing in water heated to 100 to 108 (38 - 42 degrees celsius)

give analgesics and sedatives for severe pain during rewarming

srugery (escharotomy) may be required

54
Q

what is hypothermia

A

when body’s core temp is below 95 degrees celsius

55
Q

hypothermia manifesations

A

body’s core temp cools to injurious levels below 95

56
Q

hypothermia management

A

3 categories of rewarming

  1. for mild hypothermia
    - passive external rewarming
    - place client in a warm environemtn and covered with insulation
  2. active external rewarming
    - heat is applied to skin over the trunk of the body only
  3. active core rewarming
    - most effective way to rapidly increase core temp
    - warm humidified air
    - diathermy: ultrasound and low- frequency microwave radiation

extracorporal- most rapid means of rewarming

57
Q

what is hyperthermia

A

can occur with exposure to air temp over 85 farneheit (29 celsius) and humidity above 50%

58
Q

hyperthermia tyeps

A

heat cramps:
- involuntarily spams of the large muscles of the body
- least severe type of hyperthermia
more common in athletes

heat exhaution
- commonly affects firefighters, construction or factory workers

heat stroke

  • least common but most life-threatening
  • body’s cooling system fails
59
Q

hypothermia manifestations

A

heat cramps

  • painful muscle spams, may include abdominal cramps
  • face will be red, flushed and sweating
  • oral temp can be 98.6 - 100

heat exhaustion

  • cool, moist
  • pale and ahsen skin
  • headache
  • nausea
  • dizziness
  • oral temp: above 100
heat stroke
- red skin but without perspiration
- rapid but weak pulse 
- shallow breathing
body temp right to 106 farenheit
60
Q

sunburn management

A

broad spectrum protection, protect against UVA and UVB rays

SPF 30 or higher

water resistance

avoid exposure to direct sunlight between 10am and 3 pm

61
Q

factors influencing degree of sunburn

A

genetic makeup and skin type

season of year, altitude and time of day
whether burn took place through window or glass or cause by light reflected by snow or water

meds
underlying conditions

topcial products

62
Q

classification of burns

A
local response:
increased capillary permability
fluid leaks into interstitial space
edema surround burned area
pain
systemic response:
hypovolemic shock
resp distress
ARDs
anemia
aspiration pneumonia
decreased bowel sounds
possible ileus
AKI
decreaed level of consciousness
encephalopathy
seizures 
coma

it can also cause curlings ulcer

63
Q

curlings ulcer

A

stress ulcer that occur within 24 hours of injury

administer proton pump inhibitors to prevent these like: pantoprazole

others that might help prevent these ulcers:

  • early enteral feeding
  • H2 histamine blaockers
  • NSAIDs - should be used with caution
  • diuretics: furosemine, bumetianide = CONTRAINDICATED
64
Q

burns manifestations

A
1st degree - superficial:
localized pain
dry surface
blanches with pressure
redness and possible blisters
2nd degree - partial thickness:
open wound
very painful
denuded skin
blistered and moist
3rd degree - full thickness:
tough
leathery
dull and dry with variable pain
color may be brown, tan, black or red

4th degree - full thickness:
dull and dry
ligaments, tendons, and bone may be exposed

65
Q

burns managements

A

acute phase:
priority is stop the burning process

superficial burns:
- cleanse with solutions ordered and debride loose debris and ncrotic tissue

may receive prophylactive tetanus immunizations and mild analgesics

66
Q

parkland formula

A

4 mL x TBSA % x body weight in kg

50% given in first 8 hours

other 505 given in the next 16 hours

67
Q

risk factors for melanoma skin cancer

A
fair skin tone
blonde or red hair
blue or green eyes
people who work outdoors
age (>50, <30)
immunosuppressant therapy
drug therapy
history of burns
indoor tanning
fam history of skin cancer

high altitudes
live in sunny climates

68
Q

non melanoma skin cancer

A

basal cell carinoma

squamous cell caricnoma

69
Q

what is basal cell carcinoma

A

abnormal, uncontrolled growths or lesions from skin basal cell

cumulative ltraviolet exposure and intense or occasional UV exposure

70
Q

basal cell carcinoma manifestations

A

open sore
red patches
pink growth
shiny bumps or scars

71
Q

basal cell carcinoma treatment

A

surgery:

  • Mohs micrographic surgery
  • excisional surgery
  • cryosurgery
  • curettage and electrodesiccation

radiation

imiquimod - for superifical BCC
5 Fluoruracil
Vismodegib
- ALL 3 HAVE BLACK BOX WARNING

72
Q

basal cell carcinoma management

A

skin cancer prevention

73
Q

what is squamous cell carcinoma

A

uncontrolled growth of abnormal cells arising in squamous cells

caused by cumulative UV exposure over the course of a lifetime

can be disfiguring and may be deadly if allowed to grow

74
Q

swuamous cell carcinoma manifestations

A

scaly red patches
open sores
elevated growth with central depression
occurs on any area exposed to skin

75
Q

squamous cell carcinoma treatment

A

surgery: same ones as BCC

radiation
5 Fluorouracil