Motor control Flashcards

1
Q

neglect is a result of damage to which hemisphere?

A

right
it affects the left side in perception/vision/sensartion etc

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2
Q

name the sequence. pathophysiology, .. , .. ?

A

pathophysiology, primary neuromuscular impairments, secondary musculoskeletal

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3
Q

name some motor cortex impairments - aftermath

A

motor weakness -paresis; muscle tone abnormal eg hyper or hypotonia; loss of selective muscle activation; abnormal synergies, coactivation

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4
Q

define hemiparesis

A

weakness or inability to move on one side of the body

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5
Q

define hemiplegia

A

caused by brain injury usually; a condition with varying degrees of weakness, stiffness, lack of control

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6
Q

describe hypertonicity- 2 terms

A

high muscle tone - spasticity, rigidity

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7
Q

is there more severe symptoms if damage to higher levels or lower levels?

A

lower levels

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8
Q

spasticity is

A

a hyperactive stretch reflex

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9
Q

whats hypertonicity

A
  1. spasticity- inability to recruit motor neurones
  2. abnormal RI between agonist and antagonist
  3. impaired coordination of muscle synergists
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10
Q

whats coactivation

A

activation of both agonist and antagonist

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11
Q

whats babinski reflex

A

occurs after the sole of the foot has been firmly stroked. The big toe then moves upward or toward the top surface of the foot. The other toes fan out.

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12
Q

increased muscle tone: a positive or negative symptom?

A

positive

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13
Q

loss of descending control over lower motor neurones: positive or negative symptom?

A

negative

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14
Q

paresis is a primary or secondary impairment?

A

primary

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15
Q

changes in the muscle : primary or secondary impairment?

A

secondary

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16
Q

define spasticity

A

a velocity-dependent increase in resistance of a muscle or muscle group to passive stretch

“a motor disorder characterized by a velocity-dependent increase in tonic stretch reflexes (muscle tone) with exaggerated tendon jerks, resulting from hyperexcitability of the stretch reflex, and is one component of the upper motor neuron syndrome”

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17
Q

whats individuation?

A

ability to selectively activate a muscle (or limited set of muscles) allowing isolated joint motion

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18
Q

describe 3 aspects that are affected with basal ganglia pathology

A

motor deficits, cognitive and behavioral disturbances

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19
Q

name 3 things that characterize parkinsons

A

bradykinesia (slow movements reduced in amplitude);
rigidity (increased muscle tone);
resting tremor

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20
Q

what can you observe with Parkinsons patients regarding gait and posture

A

flexed posture
impaired balance and slow gait

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21
Q

why does parkinsons occur? physiologically

A

degeneration of dopaminergic neurons in the substantia nigra pars compacta.

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22
Q

define
- bradykinesia
- akinesia
- hypokinesia

A
  • Bradykinesia: slowed movement time
  • Akinesia: reduced ability to initiate movement
  • Hypokinesia: movements decreased in amplitude
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23
Q

whats the difference between spasticity and rigidity?

A

rigidity is not dependent on velocity of stretch

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24
Q

we observe hypotonia : which part of brain is affected?

A

cerebellar impairments

25
we observe coordination issues: which part of the brain is affected?
cerebellum
26
we observe lack of fault correction: which part of the brain is affected?
cerebellum
27
describe lead pipe rigidity
constant resistance to movement thru entire ROM
28
describe cogwheel rigidity
alternating episodes of resistance and relaxation
29
explain difference between athetosis movements and chorea
athetosis: more in upper limbs, slow involuntary movements chorea: rapid jerky movements
30
postural control is divided into 2 categories. name these
postural stability and postural orientation
31
whats diaschisis?
shock, a sudden change of function in a part of brain connected to a distant damaged brain area
32
what also can occur after a lesion in brain besides diaschisis?
edema - cellular and vasogenic (disruption of BBB, accumulation of fluid)
33
what happens when there's uncontrolled release of glutamate after a brain lesion?
increase in calcium influx leading to programmed cell death -apoptosis
34
whats denervation
loss of nerve supply
35
name the 4 intercellular responses to injury
1. denervation supersensitivity 2.unmasking of silent synpases 3. neural reorganization after injured axon
36
which 5 things make for a recovery after a brain damage/lesion?
1. decrease of diaschisis 2. decrease of edema 3. reorganization of redundant pathways 4. cortical remapping treating ischemic penumbra
37
whats penumbra
reversibly damaged injured brain tissue
38
whats a cross modality plasticity
a striking adaptive feature of the brain, whereby the loss of one sensory modality induces cortical reorganization that leads to enhanced sensory performance in remaining modalities.
39
name 2 types of nonassociative implicit learning
habituation and sensitization
40
describe procedural learning
tasks and habits
41
describe problems with cognitive disorder
attention and concentration; memory; actions; plan; language; behaviour
42
someone who overestimates themselves, is detailed specific, has poor proprioception and neglect - has had a lesion in which hemisphere of the brain?
right hemisphere lesion
43
someone who has aphasia, social isolation, depression, underesttimates themselves and has a global approach - has had a lesion in which side of the brain?
left side
44
whats a medially located brain area?
limbic system
45
name some neuromuscular impairments
- paresis, muscle weakness - coactivation - individuation - abnormal synergies - spasticity- abnormal muscle tone
46
how to remember what hemiparesis is?
hemi (one side) - paresis (muscle weakness)
47
does cerebellar impairment lead to hypertonia or hypotonia?
hypotonia
48
whats ataxia?
lack of muscle coordination
49
slow movement is termed as?
bradykinesia
50
explain athetosis movement
slow involuntary twisting movements
51
is babinski reflex a positive or negative symptom?
positive bc its added
52
which parts are the action system?
motor cortex cerebellum basal ganglia (midbrain)
53
spasticity; abnormal muscle tone; paresis; coordination problems: result of what impairment?
motor cortex impairment
54
is spasticity part of upper or lower motor neurone syndrome?
upper
55
hypotonia; tremor; ataxia and no fault correction: result of what impairment?
cerebellar impairment
56
how do you subdivide hyperkinetic disorders? explain each
into chorea (rapid jerky movements) and athetosis movement (involuntary slow twisting movements)
57
overestimating themselves: more common in R or L hemisphere lesions?
more common in R lesions
58
describe left hemisphere lesion aftermath
depression reluctant aphasia social isolation underestimates themselves global approach
59
describe right hemisphere lesion aftermath
impulsive, fast spatial disorientation left side neglect euphoria detailed focused