Molar-Incisor Hypomineralisation Flashcards

1
Q

how is enamel formed?

A
  • ameloblasts
  • retreat from the ADJ
  • secrete matrix protein
  • initial calcification (mineralisation)
  • ameloblasts transition
  • secrete enzymes - remove organic component
  • full mineralisation to 99% = maturation stage
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2
Q

what is hypoplastic and hypo maturation? at what stages of enamel formation do these occur?

A

both are incomplete developments of enamel

hypoplastic
- during the enamel matrix laying down
- reduced thickness of enamel

hypomaturation
- in the calcification or maturation stage
- enamel is much softer
reduced calcification of enamel

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3
Q

how are enamel defects basically classified?

A

systemic
- environmental - chronological (MIH) or generalised (fluorosis)
- genetic - amelogenesis imperfecta - generalised defect

local
- trauma infection

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4
Q

how do you describe enamel defects - 3 main terms - give examples

A
  1. demarcated - MIH
    - clear boundary between defect
    - yellow, white or brown
  2. diffuse - fluorosis
    - no clear boundaries
    - lines/patchiness
  3. hypoplastic
    - loss of enamel
    - pits or grooves
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5
Q

define MIH

A

hypomineralisation of systemic origin

  • of 1+ permanent molars as well as any associated and affected incisors
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6
Q

5 key features of MIH

A

demarcated defects of 1st molar
- may be associated with demarcated incisor teeth
- second primary molars MAY also be hypomineralised

  • post-eruptive breakdown
  • sensitivities
  • more susceptible to caries - decay faster
  • more difficult to restore
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7
Q

the darker the lesion, the more….

A

porous
= enamel is more affected

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8
Q

possible aetiologies of MIH

A

environmental changes
chronic illness
low birth weight

chicken pox
antibiotics
dioxine exposure - pollutants

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9
Q

how is MIH treated/managed?

A
  1. start with the molars - prognosis
    - assess the devlopmental defects
    - porosity, caries risk
    - post-eruptive breakdown
    - pain/sensitivities
    - does it extend across the cusps - harder to restore
  2. options: extract or conserve
  3. monitor
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10
Q

what is the ideal orthodontic patient

A

class I occlusion
normal overbite
mild-mod crowding
all permanent teeth are present

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11
Q

when is it best to extract a first molar? a 6? what are the issues

A

lower arch
- pt to be 8-9
- L7’s have calcified dentine
- 7 can move into the space

upper arch
- timing is less important
- likely to get a space closure if extracted before 11/12

issues
- if you extract a lower 6
- upper 6 may overerupt
= you need to compensate and XLA opposite tooth
- don’t need to compensate with an U6

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12
Q

how is a MIH tooth conserved?
give the options, describe each with pros and cons

A

tooth mousse
- Recaldent - casein phosphopeptide
- changes ionic balance
- pushes Ca into the tooth
- may have milk proteins - ALLERGIES
- how to use: at home or dentist

fissure sealants
- enamel needs to be intact and hard
- no sensitivities
- if bitewings have no caries

amalgam
- little value
- not adhesive, marginal leakage, poor retentive in large, shallow cavity
GIC
- good for a temp
- adhesive, fluoride release, good insulator, poor wear resistance
composite
- good
- good wear resistance, supports tissue, technique sensitive and time consuming

SSC - stainless steel crown
- requires approximal and occlusal prep
- prone to wear, poor aesthetic, poorly adapted margins
onlay

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13
Q

if the incisors are also affected, how are these managed aesthetically?

A
  1. bleaching
    - will lighten all the tooth
  2. composite veneers
  3. porcelain veneers - CI for children
  4. resin infiltration
  5. micro abrasion
    - usually on subsurface defects
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