Module 5 Flashcards

1
Q

Name three disorders associated with ARAS dysregulation

A

a. Narcolepsy
b. Depression
c. PTSD
d. Schizophrenia
e. ADD/ADHD
f. Parkinson’s

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2
Q

What leg is inhibited with TH1 dysregulation?

A

a. Left quadriceps
b. Left soleus
c. Right shoulder extensors

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3
Q

What is the most common helminth infection in humans? What percentage of the developing world is
infected?

A

a. Ascaris lumbricoides

b. 10%

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4
Q

What constitutes the platyhelminths?

A

Flatworms

i. Cestodes
ii. Trematodes

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5
Q

What is an intermediate host?

A

Is an infected animal which is then consumed by the definitive host which results in infection of the
definitive host

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6
Q

What is contained in a proglottid?

A

. An independent reproductive and digestive system

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7
Q

List the most common general symptoms associated with helminth infection?

A

a. Diarrhoea
b. Abdominal pain
c. Nausea
d. Weakness
e. Weight loss in chronic cases of infection

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8
Q

What worms lay their eggs external to their hosts anus? Why?

A

a. Pinworms

b. Eggs require oxygen for viability

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9
Q

What is the best examination procedure for the detection of parasitic infection?

A

PCR – polymerase chain reaction

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10
Q

List the four main infections associated with irritation to the BPS?

A

a. Helicobacter Pylori
b. John Cunningham virus
c. Rota virus
d. Clostridium perfringens

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11
Q

What meridian point is associated with BPS infections?

A

GB14

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12
Q

What is the body’s primary response to parasite infection?

A

GIT purge in an attempt to dislodge infection

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13
Q

What antibodies are associated with a parasite infection?

A

. IgE

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14
Q

What is the immune systems response to a parasitic infection?

A

a. The primary immune response is TH2 based
b. There is an increased production of IgE antibodies that result in mast and basophil degranulation
causing a large local inflammatory response in attempt to destroy the parasite infection
c. Eosinophils are then recruited to the area to actively destroy the parasite

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15
Q

What do helminths do to the immune system to increase their survivability?

A

Upregulate T-regulatory cells to dampen any immune response

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16
Q

When is the only time we treat a pathogen?

A

WHEN, AND ONLY WHEN, IT TESTS IN THE CLEAR

17
Q

What is the main cause of Migraine?

A

Trigeminal central sensitization (trigeminovascular pathway)

18
Q

Problems in which areas of the body can aggravate Migraine and Central and Peripheral sensitization?

A

a. Trigeminal system (TMJ and associated areas of dysfunction, face, neck)
b. Oestrogen metabolism

19
Q

In what way, can one induce iatrogenic migraine?

A

Sustained opening of TMJ – ie a dental visit

20
Q

How can dentistry significantly aggravate Migraine and Central Sensitization?

A

Have the patient maintain an open mouth posture for an extended period

21
Q

How can a dentist mistakenly diagnose a tooth as needing a root canal? What can result if the dentist
performs a root canal or pulls the tooth of this patient?

A

a. Neuropathic pain
b. Tooth extraction is one of the largest insults to the CNS and an active nociceptive barrage can lead
to central trigeminal sensitization aggravating the existing neuropathic pain

22
Q

What is the effect of EMF on metalothionein? How can this or other influences to metalothionein be
diagnosed?

A

a. Downregulates MT

b. Can be seen with recurrent K27 switching or seen by copper:zinc ratio changes

23
Q

How can we assess to see if jewelry is creating an electric field?

A

a. Using a voltmeter

b. Checking each piece of jewellery individually to see if an electrical field is being produced

24
Q

What is the effect of prolonged jaw opening on the trigeminal nerve?

A

Overaction of the trigeminal system leading to excitotoxicity and glial cell activation

25
Q

How can we easily diagnose someone with neuritis?

A

Light / dark test is believed to be associated

26
Q

How do we diagnose a primary TMJ, primary foot or primary sacrum?

A

Becks testing

27
Q

What is the order of correction for these?

A

a. Primary TMJ – PCSD-5 or parachute test
b. Primary foot – PCSD-7 Peroneal nerve test
c. Primary sacrum

28
Q

hat is the traditional dental correction for the primary TMJ?

A

Plates / dental orthotic

29
Q

How can we diagnose a primary TMJ with AK?

A

. When an IM is inhibited with a practitioner applying posterior pressure on the mandible in the
presence of RNA

30
Q

What is the cause of the primary TMJ and how do we correct it?

A

a. TH1 dysregulation

b. Herbs/homeopathics

31
Q

What are the signs of sleep apnoea and when should we refer out for assessment?

A

Teeth grinding and abnormal tooth wear

32
Q

How is a sleep study useful in practice?

A

a. As a pre-and post-comparison for treatment modalities
b. Sleep apnoea is a key comorbidity factor for central sensitization
c. Sleep apnoea can alter HPA Axis function
d. Sleep Apnoea can lead to increased brain arousal through grinding leading to changes in the
mesencephalon and related structures

33
Q

Maintenance insomnia can result from which conditions?

A

a. HPA dysfunction
b. Insulin dysregulation
c. Parasite infection
d. Airway obstruction
e. Metalothionein depletion

34
Q

Which structures does the trigeminal nerve influence, originate from or supply?

A

a. The trigeminal system is derived from neuroectoderm
b. The Trigeminal nerve receives input from large and small diameter afferents from the anterior
aspect of the skull and the meninges. This includes sensory to face, nasal and orbital cavities as
well as teeth and the TMJ
c. The trigeminal nerve travels centrally to form nuclei from as low as C2/3 all the way to the
mesencephalon
d. It has a direct excitatory influence on the Locus Coeruleus
e. It has direct influence on cerebral vasculature