MI Flashcards

1
Q

what is secondary prevention

A

healthcare designed to r=prevent the reoccurrence of CV events in patients diagnoses with CVD

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2
Q

FRS not available for ` aged?

A

80 +

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3
Q

why CVD risk increase after menopause

A

decrease estrogen ( keeps blood vessels flexible)

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4
Q

what happens during menopause

A

increase in LDL, BP increases, HDL declines, TG increase

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5
Q

adults with diabetes are how much more likely to die from MI

A

2-4

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6
Q

normal BMI

A

18.5- 24.9

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7
Q

symptoms of MI

A

pain or discomfort, vomiting, nausea, shortness of breath, back or joint pain

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8
Q

troponin peak

A

12 hours - test for myocardial damage

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9
Q

what other tests for myocardial damage

A

CK-MB test (10-24 hours) and lactate dehydrogenase test (72 hours)

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10
Q

PCTA

A

minimally invasive surgery to open up blood vessels

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11
Q

diet immediately after MI

A

NPO or clear liquids without caffeine ( want o max blood to heart not GI)

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12
Q

heart healthy eating plan

A

mediterranean diet

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13
Q

if high TG ( above 1.7)

A

avoid sweets, sugars, simple carbs, alcohol, reach heathy weight

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14
Q

atrial fibrillation

A

irregular heart beat ( risk factor for stroke)

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15
Q

treatment for atrial fibrillation

A

anti-coagulant

vit K consistent

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16
Q

warfarin

A

what have to be careful with? cooked greens ( bc very concentrated)

17
Q

natural products that affect clotting time ?

A

onion, garlic, avovado soy milk, green tea

18
Q

why does the heart become less effective in heart failure?

A

there has been injury to the heart and compensatory actions take over to maintain cardiac output ( norepphrine and RAAS) and so ventricular muscles undergo hypertrophy ( large heart ) not a good thin g

19
Q

abdominal aortic aneurysm

A

weakened or bulging area in aorta- cause unknown but factor that cause it are smoking, HBP and vascululitus ( infection )

20
Q

angina

A

chest pain caused by coronary arteries damaged or blocked ( less oxygen)

21
Q

CAD

A

coronary artery disease ( narrow of the small blood vessels that carry blood and O2 to the heart ( angina is a symptom of this)

22
Q

congestive heart disease

A

heart can’t pump enough

23
Q

syncopy

A

fainting, light headedness caused by brief dropping blood flow to the brain

24
Q

PVD

A

atherosclerotic disease in peripheral vessels

25
Q

risk factors for heart failure

A

HPT

IHT, diabetes, heavy alcohol, smoking ,

26
Q

symptoms of HF

A

leg swelling, decreased ejection fraction breathlessness, fatigue, confusion, lungs crackling, low BP, but fast HR ( compensatory)
Q waves, tachycardia

27
Q

ejection fraction measured by

A

ECHO

28
Q

most often which side of heart fails first

A

left ( decreased pressure going to the body) ( kidney detects this and activates RAAS- increase BP) and high pressure in the pulmonary capillaries ( pressure build up) so pulmonary edema

29
Q

right side fails decreased pressure going to the

A

lungs , and edema in the rest of the body ( from pressure build up)

30
Q

ascites

A

edema in the abdominal cavities

31
Q

what can be a sign that heart failure is getting wrose

A

sudden weight gain

32
Q

why does the heart enlarge?

A

RAAS is activated, increase HR, has to pump more often to supply the same amount of blood bc less efficient

33
Q

why elevated natriuretic peptides?

A

secreted why left ventricles are stretched ( bc normally want o lower BP )

34
Q

how to treat CHF

A

diuretics to increase urination and lower BP

35
Q

how would dietician assess fluid balance in a patient with CHF

A

lab values may be diluted, skin clammy, I &O from nurses, weight and BMI may be hard to assess due to fluid retention

36
Q

3 nutritional concerns for HF patients

A

sodium intake
fluid intake
nutritional adequacy

37
Q

sodium and water intake for HF managment

A

2000 mg/d ( if more= raise BP)

1-2 L/d

38
Q

10-15% HF patients develope

A
cardiac cachexia ( loss of lean body mass - soft and flabby heart) 
- high mortality